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Cited by 626

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Transforming growth factor beta (TGF‐β) isoforms in wound healing and fibrosis

Open access
Lichtman, Michael K., Otero‐Vinas, Marta, Falanga, Vincent·2016·Wound Repair and Regeneration

Abstract

Scar formation, with persistent alteration of the normal tissue structure, is an undesirable and significant result of both wound healing and fibrosing disorders. There are few strategies to prevent or to treat scarring. The transforming growth factor beta (TGF‐β) superfamily is an important mediator of tissue repair. Each TGF‐β isoform may exert a different effect on wound healing, which may be context‐dependent. In particular, TGF‐β1 may mediate fibrosis in adults’ wounds, while TGF‐β3 may promote scarless healing in the fetus and reduced scarring in adults. Thus, TGF‐β3 may offer a scar‐reducing therapy for acute and chronic wounds and fibrosing disorders.

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Registry

Crossref · 10.1111/wrr.12398

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Metadata

Authors
Lichtman, M. K., Otero‐Vinas, M., & Falanga, V.
Venue
Wound Repair and Regeneration
Publisher
Wiley
Published
2016-03
Volume
24
Issue
2
Pages
215-222
ISSN
  • 1067-1927 (ISSN-L)
  • 1524-475X
OpenAlex
W2199255753
Cited by
626
References
2
FWCI
11.58
Type
journal article
Language
en
Open access
green

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CoChat. (2026). Citation check: Transforming growth factor beta (TGF‐β) isoforms in wound healing and fibrosis. https://app.cochat.ai/papers/10.1111/wrr.12398
The paper
Lichtman, M. K., Otero‐Vinas, M., & Falanga, V. (2016). Transforming growth factor beta (TGF‐β) isoforms in wound healing and fibrosis. Wound Repair and Regeneration. https://doi.org/10.1111/wrr.12398

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.