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Cited by 677

The DOI matches its Crossref record; 21 of the 21 references it cites were found in a registry or an index.

Resolution of Lung Inflammation by CD44

Teder, Priit, Vandivier, R. William, Jiang, Dianhua, Liang, Jiurong, Cohn, Lauren, Puré, Ellen, Henson, Peter M., Noble, Paul W.·2002·Science
DOI 10.1126/science.1069659
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Abstract

Successful repair after tissue injury and inflammation requires resolution of the inflammatory response and removal of extracellular matrix breakdown products. We have examined whether the cell-surface adhesion molecule and hyaluronan receptor CD44 plays a role in resolving lung inflammation. CD44-deficient mice succumb to unremitting inflammation following noninfectious lung injury, characterized by impaired clearance of apoptotic neutrophils, persistent accumulation of hyaluronan fragments at the site of tissue injury, and impaired activation of transforming growth factor–β 1 . This phenotype was partially reversed by reconstitution with CD44 + cells, thus demonstrating a critical role for this receptor in resolving lung inflammation.

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Crossref · 10.1126/science.1069659

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Metadata

Authors
Teder, P., Vandivier, R. W., Jiang, D., Liang, J., Cohn, L., Puré, E., Henson, P. M., & Noble, P. W.
Venue
Science
Publisher
American Association for the Advancement of Science (AAAS)
Published
2002-04-05
Volume
296
Issue
5565
Pages
155-158
ISSN
  • 0036-8075 (ISSN-L)
  • 1095-9203
DOI
10.1126/science.1069659
OpenAlex
W1964179595
Cited by
677
References
21
FWCI
14.62
Type
journal article
Language
en
Open access
closed

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.