Exogenous Induction of Cerebral ß-Amyloidogenesis Is Governed by Agent and Host

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Items 101–200 of 970

1. Propagation of Protein Aggregation in Neurodegenerative Diseases
   Jaime Vaquer‐Alicea, Marc I. Diamond · 2019 · Annual Review of Biochemistry
2. Cell-to-cell transmission of non-prion protein aggregates
   Seung‐Jae Lee, Paula Desplats, Christina J. Sigurdson, Igor F. Tsigelny, Eliezer Masliah · 2010 · Nature Reviews Neurology
3. A novel human disease with abnormal prion protein sensitive to protease
   Pierluigi L. Gambetti, Zhiqian Dong, Jue Yuan, Xiangzhu Xiao, Mengjie Zheng, Amer Alshekhlee, Rudy J. Castellani, Mark L. Cohen, Marcelo A. Barria, Dennisse Gonzalez-Romero et al. · 2008 · Annals of Neurology
4. Exosomes: vesicular carriers for intercellular communication in neurodegenerative disorders
   Anja Schneider, Mikael Simons · 2012 · Cell and Tissue Research
5. A critical appraisal of the pathogenic protein spread hypothesis of neurodegeneration
   Dominic M. Walsh, Dennis J. Selkoe · 2016 · Nature reviews. Neuroscience
6. Molecular pathology of neurodegenerative diseases by cryo-EM of amyloids
   Sjors H. W. Scheres, Benjamin Falcon, Michel Goedert · 2023 · Nature
7. Neuropathology and biochemistry of Aβ and its aggregates in Alzheimer’s disease
   Thal, Dietmar Rudolf, Walter, Jochen, Saido, Takaomi C., Fändrich, Marcus · 2015 · Acta Neuropathologica
8. Immunotherapeutic Approaches for Alzheimer’s Disease
   Wisniewski, Thomas, Goñi, Fernando · 2015 · Neuron
9. Prion-like disorders: blurring the divide between transmissibility and infectivity
   Mimi Cushman-Nick, Brian S. Johnson, Oliver D. King, Aaron D. Gitler, James Shorter · 2010 · Journal of Cell Science
10. Are synucleinopathies prion-like disorders?
    Élodie Angot, Jennifer A. Steiner, Christian Hansen, Jiayi Li, Patrik Brundin · 2010 · The Lancet Neurology
11. Induction of Tau Pathology by Intracerebral Infusion of Amyloid-β-Containing Brain Extract and by Amyloid-β Deposition in APP × Tau Transgenic Mice
    Bolmont, Tristan, Clavaguera, Florence, Meyer-Luehmann, Melanie, Herzig, Martin C., Radde, Rebecca, Staufenbiel, Matthias, Lewis, Jada, Hutton, Mike, Tolnay, Markus, Jucker, Mathias · 2007 · The American Journal of Pathology
12. Serial propagation of distinct strains of Aβ prions from Alzheimer’s disease patients
    Joel C. Watts, Carlo Condello, Jan Stöhr, Abby Oehler, Joanne Lee, Stephen J. DeArmond, Lars Lannfelt, Martin Ingelsson, Kurt Giles, Stanley B. Prusiner · 2014 · Proceedings of the National Academy of Sciences
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    Eisele, Yvonne S., Bolmont, Tristan, Heikenwalder, Mathias, Langer, Franziska, Jacobson, Laura H., Yan, Zheng-Xin, Roth, Klaus, Aguzzi, Adriano, Staufenbiel, Matthias, Walker, Lary C. et al. · 2009 · Proceedings of the National Academy of Sciences
14. The Pericyte: A Forgotten Cell Type with Important Implications for A lzheimer's Disease?
    Ethan A. Winkler, Abhay P. Sagare, Berislav V. Zloković · 2014 · Brain Pathology
15. Variably protease‐sensitive prionopathy: A new sporadic disease of the prion protein
    Wen‐Quan Zou, Gianfranco Puoti, Xiangzhu Xiao, Jue Yuan, Liuting Qing, Ignazio Calì, Miyuki Shimoji, Jan Langeveld, Rudy J. Castellani, Silvio Notari et al. · 2010 · Annals of Neurology
16. Antisense oligonucleotides: translation from mouse models to human neurodegenerative diseases
    Schoch, Kathleen M., Miller, Timothy M. · 2017 · Neuron
17. Genetic Analysis of Pathways to Parkinson Disease
    John Hardy · 2010 · Neuron
18. Prion strain discrimination using luminescent conjugated polymers
    Christina J. Sigurdson, K. Peter R. Nilsson, Simone Hornemann, Giuseppe Manco, Magdalini Polymenidou, Petra Schwarz, Mario Leclerc, Per Hammarström, Kurt Wüthrich A, Adriano Aguzzi · 2007 · Nature Methods
19. Molecular nexopathies: a new paradigm of neurodegenerative disease
    Jason D. Warren, Jonathan Daniel Rohrer, Jonathan M. Schott, Nick C. Fox, John Hardy, Martin N. Rossor · 2013 · Trends in Neurosciences
20. A mechanistic hypothesis for the impairment of synaptic plasticity by soluble Aβ oligomers from Alzheimer’s brain
    Shaomin Li, Dennis J. Selkoe · 2020 · Journal of Neurochemistry
21. Preparation and characterization of toxic Aβ aggregates for structural and functional studies in Alzheimer's disease research
    Asad Jan, Dean M. Hartley, Hilal Ahmed Lashuel · 2010 · Nature Protocols
22. Structural polymorphism of Alzheimer Aβ and other amyloid fibrils
    Marcus Fändrich, Jessica Meinhardt, Nikolaus Grigorieff · 2009 · Prion
23. Endocytic vesicle rupture is a conserved mechanism of cellular invasion by amyloid proteins
    William P. Flavin, Luc Bousset, Zachary C. Green, Yaping Chu, Stratos Skarpathiotis, Michael J. Chaney, Jeffrey H. Kordower, Ronald Melki, Edward M. Campbell · 2017 · Acta Neuropathologica
24. Conformation Determines the Seeding Potencies of Native and Recombinant Tau Aggregates
    Benjamin Falcon, Annalisa Cavallini, Rachel C. Angers, Sarah J. Glover, Tracey K. Murray, Luanda Barnham, Samuel Jackson, Michael J. O'Neill, Adrian M. Isaacs, Michael L. Hutton et al. · 2014 · Journal of Biological Chemistry
25. Misfolded protein aggregates: Mechanisms, structures and potential for disease transmission
    Moreno-Gonzalez, Ines, Soto, Claudio · 2011 · Seminars in Cell & Developmental Biology
26. Neurovascular Dysfunction and Faulty Amyloid  -Peptide Clearance in Alzheimer Disease
    Sagare, A. P., Bell, R. D., Zlokovic, B. V. · 2012 · Cold Spring Harbor Perspectives in Medicine
27. Amyloid-Beta (Aβ) Plaques Promote Seeding and Spreading of Alpha-Synuclein and Tau in a Mouse Model of Lewy Body Disorders with Aβ Pathology
    Fares Bassil, Hannah J. Brown, Shankar Pattabhiraman, Joe E. Iwasyk, Chantal M. Maghames, Emily S. Meymand, Timothy O. Cox, Dawn M. Riddle, Bin Zhang, John Q. Trojanowski et al. · 2019 · Neuron
28. Characterizing the Appearance and Growth of Amyloid Plaques in APP/PS1 Mice
    Yan, Ping, Bero, Adam W., Cirrito, John R., Xiao, Qingli, Hu, Xiaoyan, Wang, Yan, Gonzales, Ernesto, Holtzman, David M., Lee, Jin-Moo · 2009 · The Journal of Neuroscience
29. Neutral Sphingomyelinase-2 Deficiency Ameliorates Alzheimer's Disease Pathology and Improves Cognition in the 5XFAD Mouse
    Michael B. Dinkins, John Enasko, Caterina M. Hernandez, Guanghu Wang, Jina Kong, Inas Helwa, Yutao Liu, Alvin V. Terry, Erhard Bieberich · 2016 · Journal of Neuroscience
30. Spreading of Neurodegenerative Pathology via Neuron-to-Neuron Transmission of  -Amyloid
    Nath, S., Agholme, L., Kurudenkandy, F. R., Granseth, B., Marcusson, J., Hallbeck, M. · 2012 · Journal of Neuroscience
31. Physical and structural basis for polymorphism in amyloid fibrils
    Robert Tycko · 2014 · Protein Science
32. Soluble Aβ Seeds Are Potent Inducers of Cerebral β-Amyloid Deposition
    Langer, Franziska, Eisele, Yvonne S., Fritschi, Sarah K., Staufenbiel, Matthias, Walker, Lary C., Jucker, Mathias · 2011 · The Journal of Neuroscience
33. Mammalian prions and their wider relevance in neurodegenerative diseases
    John Collinge · 2016 · Nature
34. Microglia contributes to plaque growth by cell death due to uptake of amyloid β in the brain of Alzheimer's disease mouse model: Aβ Plaque Formation and Microglial Activation
    Baik, Sung Hoon, Kang, Seokjo, Son, Sung Min, Mook-Jung, Inhee · 2016 · Glia
35. Design and construction of diverse mammalian prion strains
    David W. Colby, Kurt Giles, Giuseppe Legname, Holger Wille, Ilia V. Baskakov, Stephen J. DeArmond, Stanley B. Prusiner · 2009 · Proceedings of the National Academy of Sciences
36. Blood-derived amyloid-β protein induces Alzheimer’s disease pathologies
    Xian‐Le Bu, Y Xiang, W-S Jin, J Wang, L-L Shen, Z-L Huang, Kai Zhang, Y-H Liu, Fan Zeng, Liu H et al. · 2017 · Molecular Psychiatry
37. Amyloid polymorphisms constitute distinct clouds of conformational variants in different etiological subtypes of Alzheimer’s disease
    Jay Rasmussen, Jasmin Mahler, Natalie Beschörner, Stephan A. Kaeser, Lisa M. Häsler, Frank Baumann, Sofie Nyström, Erik Portelius, Kaj Blennow, Tammaryn Lashley et al. · 2017 · Proceedings of the National Academy of Sciences
38. Alzheimer's-Type Amyloidosis in Transgenic Mice Impairs Survival of Newborn Neurons Derived from Adult Hippocampal Neurogenesis
    Laure Verret, Joanna L. Jankowsky, G. M. Xu, David Borchelt, Claire Rampon · 2007 · Journal of Neuroscience
39. Reversal of cognitive decline: A novel therapeutic program
    Dale E. Bredesen · 2014 · Aging
40. Extracellular phosphorylation of the amyloid β‐peptide promotes formation of toxic aggregates during the pathogenesis of Alzheimer's disease
    Sathish Kumar, Nasrollah Rezaei‐Ghaleh, Dick Terwel, Dietmar Rudolf Thal, Mélisande Richard, Michael Hoch, Jessica M. Mc Donald, Ullrich Wüllner, Konstantin Glebov, Michael Thomas Heneka et al. · 2011 · The EMBO Journal
41. High-speed atomic force microscopy reveals structural dynamics of amyloid β 1–42 aggregates
    Takahiro Watanabe‐Nakayama, Kenjiro Ono, Masahiro Itami, Ryoichi Takahashi, David B. Teplow, Masahito Yamada · 2016 · Proceedings of the National Academy of Sciences
42. Intracerebral injection of preformed synthetic tau fibrils initiates widespread tauopathy and neuronal loss in the brains of tau transgenic mice
    Peeraer, Eve, Bottelbergs, Astrid, Van Kolen, Kristof, Stancu, Ilie-Cosmin, Vasconcelos, Bruno, Mahieu, Michel, Duytschaever, Hilde, Ver Donck, Luc, Torremans, An, Sluydts, Ellen et al. · 2015 · Neurobiology of Disease
43. Parkinson’s disease and multiple system atrophy have distinct α-synuclein seed characteristics
    Tritia R. Yamasaki, Brandon B. Holmes, Jennifer L. Furman, Dhruva D. Dhavale, Bryant W. Su, Eun-Suk Song, Nigel J. Cairns, Paul T. Kotzbauer, Marc I. Diamond · 2018 · Journal of Biological Chemistry
44. Transmissible Proteins: Expanding the Prion Heresy
    Claudio A. Soto · 2012 · Cell
45. Cross‐seeding effects of amyloid β‐protein and α‐synuclein
    Kenjiro Ono, Ryoichi Takahashi, Tokuhei Ikeda, Masahito Yamada · 2012 · Journal of Neurochemistry
46. Prion-like Properties of Tau Protein: The Importance of Extracellular Tau as a Therapeutic Target
    Holmes, Brandon B., Diamond, Marc I. · 2014 · Journal of Biological Chemistry
47. Rapidly progressive Alzheimer’s disease features distinct structures of amyloid-β
    Mark L. Cohen, Chae Kim, Tracy Haldiman, Mohamed ElHag, Prachi Mehndiratta, Pichet Termsarasab, Frances M. Lissemore, Michelle Shea, Yvonne Cohen, Wei Chen et al. · 2015 · Brain
48. Human-to-mouse prion-like propagation of mutant huntingtin protein
    Iksoo Jeon, Francesca Cicchetti, Giulia Cisbani, Suji Lee, Endan Li, Jiwoo Bae, Nayeon Lee, Ling Li, Wooseok Im, Manho Kim et al. · 2016 · Acta Neuropathologica
49. Molecular Cross Talk between Misfolded Proteins in Animal Models of Alzheimer's and Prion Diseases
    Rodrigo Morales, Lisbell D. Estrada, Rodrigo Díaz‐Espinoza, Diego Morales‐Scheihing, María Clara Jara, Joaquı́n Castilla, Claudio A. Soto · 2010 · Journal of Neuroscience
50. De novo induction of amyloid-β deposition in vivo
    Rodrigo Morales, Claudia A. Duran-Aniotz, Joaquı́n Castilla, Lisbell D. Estrada, Claudio A. Soto · 2011 · Molecular Psychiatry
51. Imaging Distinct Conformational States of Amyloid-β Fibrils in Alzheimer’s Disease Using Novel Luminescent Probes
    K. Peter R. Nilsson, Andreas Åslund, Ina Berg, Sofie Nyström, Peter Konradsson, Anna Herland, Olle Inganäs, Frantz Stabo-Eeg, Mikaël Lindgren, Gunilla Torstensdotter Westermark et al. · 2007 · ACS Chemical Biology
52. Molecular structure of a prevalent amyloid-β fibril polymorph from Alzheimer's disease brain tissue
    Ujjayini Ghosh, Kent R. Thurber, Wai‐Ming Yau, Robert Tycko · 2021 · Proceedings of the National Academy of Sciences
53. Progression of cerebral amyloid angiopathy: a pathophysiological framework
    Emma A. Koemans, Jasmeer P. Chhatwal, Susanne J. van Veluw, Ellis S. van Etten, Matthias J.P. van Osch, Marianne A.A. van Walderveen, Hamid Reza Sohrabi, Mariel G. Kozberg, Zahra Shirzadi, Gisela Marie Terwindt et al. · 2023 · The Lancet Neurology
54. Protein aggregation and neurodegeneration in prototypical neurodegenerative diseases: Examples of amyloidopathies, tauopathies and synucleinopathies
    Mathieu Bourdenx, Nikolaos Stavros Koulakiotis, Despina Sanoudou, Erwan Bézard, Benjamin Dehay, Anthony Tsarbopoulos · 2015 · Progress in Neurobiology
55. Heterotypic seeding of Tau fibrillization by pre-aggregated Abeta provides potent seeds for prion-like seeding and propagation of Tau-pathology in vivo
    Vasconcelos, Bruno, Stancu, Ilie-Cosmin, Buist, Arjan, Bird, Matthew, Wang, Peng, Vanoosthuyse, Alexandre, Van Kolen, Kristof, Verheyen, An, Kienlen-Campard, Pascal, Octave, Jean-Noël et al. · 2016 · Acta Neuropathologica
56. Accumulation of prion protein in the brain that is not associated with transmissible disease
    Pedro Piccardo, Jean C. Manson, Declan King, Bernardino Francesco Ghetti, Rona M. Barron · 2007 · Proceedings of the National Academy of Sciences
57. TDP-43 extracted from frontotemporal lobar degeneration subject brains displays distinct aggregate assemblies and neurotoxic effects reflecting disease progression rates
    Florent Laferrière, Zuzanna Maniecka, Manuela Pérez‐Berlanga, Marián Hruška-Plocháň, Larissa A. B. Gilhespy, Eva‐Maria Hock, Ulrich Wagner, Tariq Afroz, Paul J. Boersema, Gery Barmettler et al. · 2018 · Nature Neuroscience
58. Prion-like spread of protein aggregates in neurodegeneration
    Polymenidou, Magdalini, Cleveland, Don W. · 2012 · Journal of Experimental Medicine
59. Protein aggregate spreading in neurodegenerative diseases: Problems and perspectives
    Seung‐Jae Lee, Hee-Sun Lim, Eliezer Masliah, He-Jin Lee · 2011 · Neuroscience Research
60. Biochemical and biophysical features of both oligomer/fibril and cell membrane in amyloid cytotoxicity
    Massimo Stefani · 2010 · FEBS Journal
61. Phosphorylation of amyloid beta (Aβ) peptides – A trigger for formation of toxic aggregates in Alzheimer's disease
    Sathish Kumar, Jochen Walter · 2011 · Aging
62. Beyond the prion principle
    Adriano Aguzzi · 2009 · Nature
63. Neuronal heparan sulfates promote amyloid pathology by modulating brain amyloid-β clearance and aggregation in Alzheimer’s disease
    Liu, Chia-Chen, Zhao, Na, Yamaguchi, Yu, Cirrito, John R., Kanekiyo, Takahisa, Holtzman, David M., Bu, Guojun · 2016 · Science Translational Medicine
64. Investigating the Spreading and Toxicity of Prion-like Proteins Using the Metazoan Model Organism C. elegans
    Carmen I. Nussbaum-Krammer, Mário F. Neto, Renée M. Brielmann, Jesper Søndergaard Pedersen, Richard I. Morimoto · 2015 · Journal of Visualized Experiments
65. Zn(II)‐ and Cu(II)‐induced non‐fibrillar aggregates of amyloid‐β (1–42) peptide are transformed to amyloid fibrils, both spontaneously and under the influence of metal chelators
    Tõugu, Vello, Karafin, Ann, Zovo, Kairit, Chung, Roger S., Howells, Claire, West, Adrian K., Palumaa, Peep · 2009 · Journal of Neurochemistry
66. Mechanisms of Pathogenic Tau and Aβ Protein Spreading in Alzheimer’s Disease
    Paolo d’Errico, Melanie Meyer‐Luehmann · 2020 · Frontiers in Aging Neuroscience
67. Structural features and cytotoxicity of amyloid oligomers: Implications in Alzheimer's disease and other diseases with amyloid deposits
    Massimo Stefani · 2012 · Progress in Neurobiology
68. Distinct synthetic Aβ prion strains producing different amyloid deposits in bigenic mice
    Jan Stöhr, Carlo Condello, Joel C. Watts, Lillian Bloch, Abby Oehler, Mimi Nick, Stephen J. DeArmond, Kurt Giles, William F. DeGrado, Stanley B. Prusiner · 2014 · Proceedings of the National Academy of Sciences
69. Defining Alzheimer as a common age-related neurodegenerative process not inevitably leading to dementia
    Ferrer, Isidro · 2012 · Progress in Neurobiology
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    A. Jan, O. Adolfsson, I. Allaman, A. Buccarello, P. Magistretti, Andrea Pfeifer, A. Muhs, H. Lashuel · 2010 · Journal of Biological Chemistry
71. In Situ Measurements of the Formation and Morphology of Intracellular β-Amyloid Fibrils by Super-Resolution Fluorescence Imaging
    Gabriele S. Kaminski Schierle, Sebastian van de Linde, Miklós Erdélyi, Elin K. Esbjörner, Teresa Klein, Eric J. Rees, Carlos W. Bertoncini, Christopher Martin Dobson, Markus Sauer, Clemens F. Kaminski · 2011 · Journal of the American Chemical Society
72. Intercellular (Mis)communication in Neurodegenerative Disease
    Gwenn A. Garden, Albert R. La Spada · 2012 · Neuron
73. Prion hypothesis: the end of the controversy?
    Claudio A. Soto · 2010 · Trends in Biochemical Sciences
74. Aβ42 Mutants with Different Aggregation Profiles Induce Distinct Pathologies in Drosophila
    Koichi Iijima, Hsueh‐Cheng Chiang, Stephen Hearn, Inessa Hakker, Anthony Gatt, Christopher Shenton, Linda Granger, Amy Leung, Kanae Iijima‐Ando, Yi Zhong · 2008 · PLoS ONE
75. Distinct α-Synuclein strains and implications for heterogeneity among α-Synucleinopathies
    Chao Peng, Ronald J. Gathagan, Virginia M.‐Y. Lee · 2017 · Neurobiology of Disease
76. Origins and Effects of Extracellular α-synuclein: Implications in Parkinson’s Disease
    Seung‐Jae Lee · 2007 · Journal of Molecular Neuroscience
77. A deadly spread: cellular mechanisms of α-synuclein transfer
    Jennifer A. Steiner, Élodie Angot, Patrik Brundin · 2011 · Cell Death and Differentiation
78. Amyloid-β immunisation for Alzheimer's disease
    Wisniewski, Thomas, Konietzko, Uwe · 2008 · The Lancet Neurology
79. Passive immunotherapies targeting Aβ and tau in Alzheimer's disease
    Steven Samuel Plotkin, Neil R. Cashman · 2020 · Neurobiology of Disease
80. Spreading of amyloid-β peptides via neuritic cell-to-cell transfer is dependent on insufficient cellular clearance
    Jakob Domert, Sahana Bhima Rao, Lotta Agholme, Ann‐Christin Brorsson, Jan O. Marcusson, Martin Hallbeck, Sangeeta Nath · 2014 · Neurobiology of Disease
81. Can Infections Cause Alzheimer's Disease?
    Francis Mawanda, Robert B. Wallace · 2013 · Epidemiologic Reviews
82. The cell biology of prion-like spread of protein aggregates: mechanisms and implication in neurodegeneration
    Maddalena Costanzo, Chiara Zurzolo · 2013 · Biochemical Journal
83. Somatic Mosaicism in the Human Genome
    Donald N. Freed, Eric L. Stevens, Jonathan Pevsner · 2014 · Genes
84. Disinfection: is it time to reconsider Spaulding?
    Gerald E. McDonnell, P. Burke · 2011 · Journal of Hospital Infection
85. Structural heterogeneity and intersubject variability of Aβ in familial and sporadic Alzheimer’s disease
    Carlo Condello, Thomas Lemmin, Jan Stöhr, Mimi Nick, Yibing Wu, Alison M. Maxwell, Joel C. Watts, Christoffer D. Caro, Abby Oehler, Christopher Dirk Keene et al. · 2018 · Proceedings of the National Academy of Sciences
86. The prion hypothesis: from biological anomaly to basic regulatory mechanism
    Tuite, Mick F., Serio, Tricia R. · 2010 · Nature Reviews Molecular Cell Biology
87. Role of Different Alpha-Synuclein Strains in Synucleinopathies, Similarities with other Neurodegenerative Diseases
    Ronald Melki · 2015 · Journal of Parkinson s Disease
88. Cerebral amyloid angiopathy in the aetiology and immunotherapy of Alzheimer disease
    Roy O. Weller, Stephen Preston, Malavika Subash, Roxana Octavia Carare · 2009 · Alzheimer s Research & Therapy
89. Recent advances in the histo‐molecular pathology of human prion disease
    Simone Baiardi, Marcello Rossi, Sabina Capellari, Piero Parchi · 2018 · Brain Pathology
90. Tau strains shape disease
    Jaime Vaquer‐Alicea, Marc I. Diamond, Łukasz A. Joachimiak · 2021 · Acta Neuropathologica
91. Formation and Propagation of Tau Oligomeric Seeds
    Julia E. Gerson, Rakez Kayed · 2013 · Frontiers in Neurology
92. Seeded strain‐like transmission of β‐amyloid morphotypes in APP transgenic mice
    Götz Heilbronner, Yvonne S. Eisele, Franziska Langer, Stephan A. Kaeser, Renata Novotny, Amudha Nagarathinam, Andreas Åslund, Per Hammarström, K. Peter R. Nilsson, Mathias Jucker · 2013 · EMBO Reports
93. Intercellular Spread of Protein Aggregates in Neurodegenerative Disease
    Davis, Albert A., Leyns, Cheryl E.G., Holtzman, David M. · 2018 · Annual Review of Cell and Developmental Biology
94. Atherosclerosis and Alzheimer - diseases with a common cause? Inflammation, oxysterols, vasculature
    Richard Lathe, Alexandra A. Tsitrina, Yuri V. Kotelevtsev · 2014 · BMC Geriatrics
95. Gut inflammation triggers C/EBPβ/δ‐secretase‐dependent gut‐to‐brain propagation of Aβ and Tau fibrils in Alzheimer’s disease
    Chun Chen, Yunzhe Zhou, Hualong Wang, Ashfaqul M. Alam, Seong Su Kang, Eun Hee Ahn, Xia Liu, Jianping Jia, Keqiang Ye · 2021 · The EMBO Journal
96. Canine Cognitive Dysfunction and Alzheimer’s Disease – Two Facets of the Same Disease?
    Sonja Prpar Mihevc, Gregor Majdič · 2019 · Frontiers in Neuroscience
97. Aβ and human amylin share a common toxicity pathway via mitochondrial dysfunction
    Yun‐An Lim, Virginie F. Rhein, Ginette Baysang, Fides Meier, Anne Poljak, Mark J. Raftery, Michael Guilhaus, Lars Matthias Ittner, Anne Eckert, Jürgen Götz · 2010 · PROTEOMICS
98. Cholesterol in Alzheimers Disease: Unresolved Questions
    Massimo Stefani, Gianfranco Liguri · 2009 · Current Alzheimer Research
99. Aβ and tau prion-like activities decline with longevity in the Alzheimer’s disease human brain
    Atsushi Aoyagi, Carlo Condello, Jan Stöhr, Weizhou Yue, Brianna M. Rivera, Joanne C. Lee, Amanda L. Woerman, Glenda Margaret Halliday, Sjoerd G. van Duinen, Martin Ingelsson et al. · 2019 · Science Translational Medicine
100. CB2 Cannabinoid Receptor As Potential Target against Alzheimer's Disease
     Ester Aso, Isidró Ferrer · 2016 · Frontiers in Neuroscience