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Chemokine Gene Silencing in Decidual Stromal Cells Limits T Cell Access to the Maternal-Fetal Interface
Open accessAbstract
Keeping Baby Safe: Because half the genes from a developing fetus are inherited from the father, from the mother's perspective, a fetus is “foreign.” How, then, does the maternal immune system tolerate the fetus? Nancy et al. (p. 1317 ) found that careful regulation of cellular recruitment signals allowed for fetal tolerance in mice. Although high numbers of T cells localized to the myometrium layer of the uterine wall in pregnant mice, very few T cells were found in the decidua, the uterine tissue that encapsulates the fetus and placenta. Thus, in pregnancy, regulation of immune cell localization may allow for organ-specific immune tolerance.
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Cites
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Immune mechanisms at the maternal-fetal interface: perspectives and challenges
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Metadata
- Authors
- Nancy, P., Tagliani, E., Tay, C.-S., Asp, P., Levy, D. E., & Erlebacher, A.
- Venue
- Science
- Published
- 2012-06-08
- Volume
- 336
- Issue
- 6086
- Pages
- 1317-1321
- ISSN
- 0036-8075 (ISSN-L)
- 1095-9203
- OpenAlex
- W2137938346
- Cited by
- 429
- References
- 1
- FWCI
- 14.17
- Type
- journal article
- Language
- en
- Open access
- green
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- The paper
- Nancy, P., Tagliani, E., Tay, C.-S., Asp, P., Levy, D. E., & Erlebacher, A. (2012). Chemokine Gene Silencing in Decidual Stromal Cells Limits T Cell Access to the Maternal-Fetal Interface. Science. https://doi.org/10.1126/science.1220030
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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