The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.
Vascular System Defects and Impaired Cell Chemokinesis as a Result of Gα 13 Deficiency
Abstract
Heterotrimeric GTP-binding proteins (G proteins) participate in cellular signaling and regulate a variety of physiological processes. Disruption of the gene encoding the G protein subunit α 13 (Gα 13 ) in mice impaired the ability of endothelial cells to develop into an organized vascular system, resulting in intrauterine death. In addition, Gα 13 (−/−) embryonic fibroblasts showed greatly impaired migratory responses to thrombin. These results demonstrate that Gα 13 participates in the regulation of cell movement in response to specific ligands, as well as in developmental angiogenesis.
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Cites
Angiogenesis in cancer, vascular, rheumatoid and other disease
Folkman, J. (1995) · Nature Medicine · doi:10.1038/nm0195-27 · Cited by 7,627
Failure of blood-island formation and vasculogenesis in Flk-1-deficient mice
Shalaby, F., Rossant, J., Yamaguchi, T. P., Gertsenstein, M., Wu, X.-F., Breitman, M. L., & Schuh, A. C. (1995) · Nature · doi:10.1038/376062a0 · Cited by 3,931
Cited by
Regulation, signaling, and physiological functions of G-Proteins
Syrovatkina, V., Alegre, K. O., Dey, R., & Huang, X.-Y. (2016) · Journal of Molecular Biology · doi:10.1016/j.jmb.2016.08.002 · Cited by 498
Trani, M., & Dejana, E. (2015) · Current Opinion in Hematology · doi:10.1097/moh.0000000000000137 · Cited by 52
Kim, Y. M., Lim, S.-C., Han, C. Y., Kay, H. Y., Cho, I. J., Ki, S. H., Lee, M. Y., Kwon, H. M., Lee, C. H., & Kim, S. G. (2011) · Arteriosclerosis, Thrombosis, and Vascular Biology · doi:10.1161/atvbaha.110.218552 · Cited by 17
Metadata
- Authors
- Offermanns, S., Mancino, V., Revel, J.-P., & Simon, M. I.
- Venue
- Science
- Published
- 1997-01-24
- Volume
- 275
- Issue
- 5299
- Pages
- 533-536
- ISSN
- 0036-8075 (ISSN-L)
- 1095-9203
- OpenAlex
- W2032187164
- Cited by
- 338
- References
- 2
- FWCI
- 7.88
- Type
- journal article
- Language
- en
- Open access
- closed
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