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Cited by 679

The DOI matches its Crossref record; 4 of the 4 references it cites were found in a registry or an index.

Molecular Mimicry, Bystander Activation, or Viral Persistence: Infections and Autoimmune Disease

Open access
Fujinami, Robert S., von Herrath, Matthias G., Christen, Urs, Whitton, J. Lindsay·2006·Clinical Microbiology Reviews

Abstract

SUMMARY: Virus infections and autoimmune disease have long been linked. These infections often precede the occurrence of inflammation in the target organ. Several mechanisms often used to explain the association of autoimmunity and virus infection are molecular mimicry, bystander activation (with or without epitope spreading), and viral persistance. These mechanisms have been used separately or in various combinations to account for the immunopathology observed at the site of infection and/or sites of autoimmune disease, such as the brain, heart, and pancreas. These mechanisms are discussed in the context of multiple sclerosis, myocarditis, and diabetes, three immune-medicated diseases often linked with virus infections.

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Crossref · 10.1128/cmr.19.1.80-94.2006

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Metadata

Authors
Fujinami, R. S., von Herrath, M. G., Christen, U., & Whitton, J. L.
Venue
Clinical Microbiology Reviews
Published
2006-01
Volume
19
Issue
1
Pages
80-94
ISSN
  • 0893-8512 (ISSN-L)
  • 1098-6618
OpenAlex
W2097728608
Cited by
679
References
4
FWCI
11.06
Type
journal article
Language
en
Open access
green

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This page
CoChat. (2026). Citation check: Molecular Mimicry, Bystander Activation, or Viral Persistence: Infections and Autoimmune Disease. https://app.cochat.ai/papers/10.1128/cmr.19.1.80-94.2006
The paper
Fujinami, R. S., von Herrath, M. G., Christen, U., & Whitton, J. L. (2006). Molecular Mimicry, Bystander Activation, or Viral Persistence: Infections and Autoimmune Disease. Clinical Microbiology Reviews. https://doi.org/10.1128/cmr.19.1.80-94.2006

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.