Cited by 1,618Nitric Oxide and Endothelin EffectsAntioxidant Activity and Oxidative StressRedox biology and oxidative stress

The DOI matches its Crossref record; 137 of the 137 references it cites were found in a registry or an index.

Oxidative Stress and Vascular Disease

Madamanchi, Nageswara R., Vendrov, Aleksandr, Runge, Marschall S.·2005·Arteriosclerosis, Thrombosis, and Vascular Biology

DOI 10.1161/01.atv.0000150649.39934.13

Open this paper in CoChat — free account

Abstract

Growing evidence indicates that chronic and acute overproduction of reactive oxygen species (ROS) under pathophysiologic conditions is integral in the development of cardiovascular diseases (CVD). These ROS can be released from nicotinamide adenine dinucleotide (phosphate) oxidase, xanthine oxidase, lipoxygenase, mitochondria, or the uncoupling of nitric oxide synthase in vascular cells. ROS mediate various signaling pathways that underlie vascular inflammation in atherogenesis: from the initiation of fatty streak development through lesion progress to ultimate plaque rupture. Various animal models of oxidative stress support the notion that ROS have a causal role in atherosclerosis and other cardiovascular diseases. Human investigations also support the oxidative stress hypothesis of atherosclerosis. Oxidative stress is the unifying mechanism for many CVD risk factors, which additionally supports its central role in CVD. Despite the demonstrated role of antioxidants in cellular and animal studies, the ineffectiveness of antioxidants in reducing cardiovascular death and morbidity in clinical trials has led many investigators to question the importance of oxidative stress in human atherosclerosis. Others have argued that the prime factor for the mixed outcomes from using antioxidants to prevent CVD may be the lack of specific and sensitive biomarkers by which to assess the oxidative stress phenotypes underlying CVD. A better understanding of the complexity of cellular redox reactions, development of a new class of antioxidants targeted to specific subcellular locales, and the phenotype-genotype linkage analysis for oxidative stress will likely be avenues for future research in this area as we move toward the broader use of pharmacological and regenerative therapies in the treatment and prevention of CVD.

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

Integrity signals

Registry

Crossref · 10.1161/01.atv.0000150649.39934.13

Cites

137 of 137 references verified0 citation errors · 0 not checkable

Statistics

Claims the paper makes

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

The self-check needs the paper’s full text.

Atherosclerosis--an inflammatory disease
Ross, R. (1999) · New England Journal of Medicine · doi:10.1056/nejm199901143400207 · Cited by 21,725
Seventh Report of the Joint National Committee on Prevention, Detection, Evaluation, and Treatment of High Blood Pressure
Chobanian, A. V., Bakris, G. L., Black, H. R., Cushman, W. C., Green, L. A., Izzo, J. L., Jones, D. W., Materson, B. J., Oparil, S., Wright, J. T., et al. (2003) · Hypertension · doi:10.1161/01.hyp.0000107251.49515.c2 · Cited by 13,271
The pathogenesis of atherosclerosis: a perspective for the 1990s
Ross, R. (1993) · Nature · doi:10.1038/362801a0 · Cited by 10,618
Free Radicals in the Physiological Control of Cell Function
Dröge, W. (2002) · Physiological Reviews · doi:10.1152/physrev.00018.2001 · Cited by 9,855
Normalizing mitochondrial superoxide production blocks three pathways of hyperglycaemic damage
Nishikawa, T., Edelstein, D., Du, X. L., Yamagishi, S.-I., Matsumura, T., Kaneda, Y., Yorek, M. A., Beebe, D., Oates, P. J., Hammes, H.-P., et al. (2000) · Nature · doi:10.1038/35008121 · Cited by 4,277
Role of Oxidative Stress in Development of Complications in Diabetes
Baynes, J. (1991) · Diabetes · doi:10.2337/diab.40.4.405 · Cited by 3,791
Untitled work
Fridovich (1978) · doi:10.1126/science.210504 · Cited by 3,329
Mitochondrial diseases in man and mouse
Wallace, D. C. (1999) · Science · doi:10.1126/science.283.5407.1482 · Cited by 3,060
NAD(P)H Oxidase: Role in Cardiovascular Biology and Disease
Griendling, K. K., Sorescu, D., & Ushio-Fukai, M. (2000) · Circulation Research · doi:10.1161/01.res.86.5.494 · Cited by 2,886
Requirement for Generation of H 2 O 2 for Platelet-Derived Growth Factor Signal Transduction
Sundaresan, M., Yu, Z.-X., Ferrans, V. J., Irani, K., & Finkel, T. (1995) · Science · doi:10.1126/science.270.5234.296 · Cited by 2,520

Show all 135 references

Research works printed but not linked: 33

10.1093/ije/18.3_Supplement_1.S20 · DOI
10.1161/atvb.16.7.831 · DOI
10.1161/res.80.6.810 · DOI
1996, Am J Physiol, 271, H1626
10.1161/res.86.5.494 · DOI
10.1161/circ.99.15.2027 · DOI
10.1161/atvb.21.5.739 · DOI
10.1161/atvb.22.1.21 · DOI
10.1161/circ.101.12.1372 · DOI
10.1161/circ.103.10.1446 · DOI
10.1161/circ.103.9.1282 · DOI
10.1161/res.86.9.960 · DOI
10.1161/hyp.38.6.1395 · DOI
10.1161/circ.105.3.393 · DOI
10.1161/hyp.37.6.1450 · DOI
10.1161/res.89.5.408 · DOI
10.1161/hyp.37.2.334 · DOI
10.1161/circ.101.14.1653 · DOI
10.1161/circ.98.8.794 · DOI
10.1161/hyp.32.3.488 · DOI
10.1161/res.85.5.437 · DOI
10.1161/res.85.4.357 · DOI
1994, Am J Physiol, 267, H1804
10.1161/res.87.3.241 · DOI
10.1161/str.31.8.1889 · DOI
10.1161/circ.99.2.224 · DOI
10.1161/circ.103.12.1618 · DOI
10.1161/res.88.2.e14 · DOI
10.1161/circ.101.11.1261 · DOI
10.1161/atvb.21.9.1451 · DOI
10.1161/circ.88.4.8403327 · DOI
10.1161/circ.105.7.849 · DOI
10.1161/res.87.12.1074 · DOI

Sorted by citations

Items 501–600 of 1,000

Impaired Endothelial Repair Capacity of Early Endothelial Progenitor Cells in Hypertensive Patients With Primary Hyperaldosteronemia
Chen, L. F., Ding, M.-L., Wu, F., He, W., Li, J., Zhang, X.-Y., Xie, W., Duan, S., Xia, W., & Tao, J. (2015) · Hypertension · doi:10.1161/hypertensionaha.115.06597 · Cited by 42
“Increasing evidence indicates that acute and chronic oxidative stress in cardiovascular system is an essential molecular mechanism underlying the development of atherosclerotic vascular disease.(26) Previous studies reported that aldosterone treatment in vitro induced ROS production of early EPCs.”
Background
Redox regulation of transient receptor potential channels in the endothelium
Pires, P. W., & Earley, S. (2016) · Microcirculation · doi:10.1111/micc.12329 · Cited by 39
Renin-angiotensin system acting on reactive oxygen species in paraventricular nucleus induces sympathetic activation via AT1R/PKCγ/Rac1 pathway in salt-induced hypertension
Su, Q., Huo, C.-J., Li, H., Liu, K., Li, X. O., Yang, Q. Z., Song, X., Chen, W., Cui, W., Zhu, G., et al. (2017) · Scientific Reports · doi:10.1038/srep43107 · Cited by 42
Potential Cardioprotective Effects of Sumac Capsule in Patients With Hyperlipidemia: A Triple-Blind Randomized, Placebo-Controlled Crossover Trial
Asgary, S., Salehizadeh, L., Keshvari, M., Taheri, M., Spence, N. D., Farvid, M. S., Rafieian‐Kopaei, M., & Sarrafzadegan, N. (2018) · Journal of the American College of Nutrition · doi:10.1080/07315724.2017.1394237 · Cited by 42
Metabolomics in early detection and prognosis of acute coronary syndrome
Amiri, M. P., Khoshkam, M., Salek, R. M., Madadi, R., Ganji, G. F., & Ramazani, A. (2019) · Clinica Chimica Acta · doi:10.1016/j.cca.2019.03.1632 · Cited by 46
Role of Glutaredoxin-1 and Glutathionylation in Cardiovascular Diseases
Burns, M., Rizvi, S. H. M., Tsukahara, Y., Pimentel, D. R., Luptak, I., Hamburg, N. M., Matsui, R., & Bachschmid, M. M. (2020) · International Journal of Molecular Sciences · doi:10.3390/ijms21186803 · Cited by 36
Plasma levels of nitric oxide and stroke outcome
Taffi, R., Nanetti, L., Mazzanti, L., Bartolini, M., Vignini, A., Raffaelli, F., Pasqualetti, P., Vernieri, F., Provinciali, L., & Silvestrini, M. (2007) · Journal of Neurology · doi:10.1007/s00415-007-0700-y · Cited by 42
Evaluation of oxidative stress, enzymatic and non-enzymatic antioxidants and metabolic thyroid hormone status in patients with diabetes mellitus
Pasupathi, P., Varatharaju, C., & Kumar, U. S. (2009) · Diabetes & Metabolic Syndrome Clinical Research & Reviews · doi:10.1016/j.dsx.2009.07.004 · Cited by 43
Antioxidant and anti-atherogenic activities of three Piper species on atherogenic diet fed hamsters
Agbor, G. A., Vinson, J. A., Sortino, J., & Johnson, R. B. (2010) · Experimental and Toxicologic Pathology · doi:10.1016/j.etp.2010.10.003 · Cited by 42
“Some of the oxidative systems that contribute to LDL-C oxidation in vivo include NADPH oxidases, xanthine oxidase, myeloperoxidase, uncoupled nitric oxide synthase, lipoxygenases, and the mitochondrial electron transport chain (Madamanchi et al., 2005; Mueller et al., 2005).”
Background
Prognostic implication of macrocytosis on adverse outcomes after coronary intervention
Myojo, M., Iwata, H., Kohro, T., Sato, H., Kiyosue, A., Ando, J., Sawaki, D., Takahashi, M., Fujita, H., Hirata, Y., et al. (2011) · Atherosclerosis · doi:10.1016/j.atherosclerosis.2011.11.044 · Cited by 42
“However, oxidaive stress resulting from the overproduction of reactive oxygen pecies (ROS) has been considered to contribute to the develpment and progression of cardiovascular diseases [2]; besides, etabolism and homeostasis of the erythrocyte membrane have”
Background
Lipid Lowering and Antioxidant Effect of Miglitol in Triton Treated Hyperlipidemic and High Fat Diet Induced Obese Rats
Shrivastava, A., Chaturvedi, U., Singh, S. V., Saxena, J. K., & Bhatia, G. (2013) · Lipids · doi:10.1007/s11745-012-3753-3 · Cited by 41
“Hyperlipidemia and oxidative stress have been implicated as the important causative factors for the development of cardiovascular diseases [1, 7, 41], such as atherosclerosis and its complications, acute myocardial infarction, hypertension and coronary heart disease [42, 43].”
Background
Mechanisms of Vascular Calcification: The Pivotal Role of Pyruvate Dehydrogenase Kinase 4
Leem, J., & Lee, I. K. (2016) · Endocrinology and Metabolism · doi:10.3803/enm.2016.31.1.52 · Cited by 40
Niazirin from Moringa oleifera Lam. attenuates high glucose-induced oxidative stress through PKCζ/Nox4 pathway
Wang, F., Bao, Y., Shen, X., Zengin, G., Lyu, Y., Xiao, J., & Weng, Z. (2019) · Phytomedicine · doi:10.1016/j.phymed.2019.153066 · Cited by 41
Cosmetic and Dermatological Properties of Selected Ayurvedic Plant Extracts
Zagórska-Dziok, M., Ziemlewska, A., Bujak, T., Nizioł‐Łukaszewska, Z., & Hordyjewicz‐Baran, Z. (2021) · Molecules · doi:10.3390/molecules26030614 · Cited by 46
Identification of ACOX2 as a shared genetic risk factor for preeclampsia and cardiovascular disease
Johansson, Å., Curran, J. E., Johnson, M. P., Freed, K. A., Fenstad, M. H., Bjørge, L., Eide, I. P., Carless, M. A., Rainwater, D. L., Göring, H. H. H., et al. (2011) · European Journal of Human Genetics · doi:10.1038/ejhg.2011.19 · Cited by 41
Shear stress influences spatial variations in vascular Mn-SOD expression: implication for LDL nitration
Ai, L., Rouhanizadeh, M., Wu, J. C., Takabe, W., Yu, H., Alavi, M., Li, R., Chu, Y., Miller, J. D., Heistad, D. D., et al. (2008) · American Journal of Physiology-Cell Physiology · doi:10.1152/ajpcell.00518.2007 · Cited by 42
“5% to 2% of electrons leak out to form O2 , which is dismutased by Mn-SOD (39).”
Background
Hemeoxygenase-1 expression in response to arecoline-induced oxidative stress in human umbilical vein endothelial cells
Hung, T.-C., Huang, L., Su, S.-J., Hsieh, B.-S., Cheng, H.-L., Hu, Y.-C., Chen, Y., Hwang, C., & Chang, K. (2010) · International Journal of Cardiology · doi:10.1016/j.ijcard.2010.05.015 · Cited by 40
“This “two-faced” character of ROS is clearly substantiated [26,27].”
Background
Micronutrient Malnutrition, Obesity, and Chronic Disease in Countries Undergoing the Nutrition Transition: Potential Links and Program/Policy Implications
Eckhardt, C. L., & Eckhardt, C. L. (2006) · AgEcon Search (University of Minnesota, USA) · doi:10.22004/ag.econ.55889 · Cited by 41
“However, clinical supplementation trials of antioxidant vitamins, such as vitamin E and vitamin C, have not resulted in consistent evidence of reduced CVD morbidity or mortality (Stocker and Keaney 2004; Madamanchi, Vendrov, and Runge 2005; Sachidanandam, Fagan, and Ergul 2005; Gaziano 2004).”
A thioredoxin-mimetic peptide exerts potent anti-inflammatory, antioxidant, and atheroprotective effects in ApoE2.Ki mice fed high fat diet
Canesi, F., Mateo, V., Couchie, D., Karabina, S., Nègre‐Salvayre, A., Rouis, M., & Hadri, K. E. (2018) · Cardiovascular Research · doi:10.1093/cvr/cvy183 · Cited by 40
Evaluating the link between Paraoxonase-1 levels and Alzheimer's disease development
Cervellati, C., Valacchi, G., Tisato, V., Zuliani, G., & Marsillach, J. (2019) · Minerva Medica · doi:10.23736/s0026-4806.18.05875-5 · Cited by 40
The therapeutic potential of BRD4 in cardiovascular disease
Lin, S., & Du, L. (2020) · Hypertension Research · doi:10.1038/s41440-020-0459-4 · Cited by 40
The Potential Role of Creatine in Vascular Health
Clarke, H. E., Hickner, R. C., & Ormsbee, M. J. (2021) · Nutrients · doi:10.3390/nu13030857 · Cited by 44
Thioredoxin-1/peroxiredoxin-1 as sensors of oxidative stress mediated by NADPH oxidase activity in atherosclerosis
Madrigal‐Matute, J., Fernandez-Garcia, C.-E., Blanco‐Colio, L. M., Burillo, E., Fortuño, A., Martínez-Pinna, R., Llamas‐Granda, P., Beloqui, Ó., Egido, J., Zalba, G., et al. (2015) · Free Radical Biology and Medicine · doi:10.1016/j.freeradbiomed.2015.06.001 · Cited by 41
Cajaninstilbene acid (CSA) exerts cytoprotective effects against oxidative stress through the Nrf2-dependent antioxidant pathway
Lü, L., Luo, M., Fu, Y., Zu, Y., Wang, W., Gu, C., Zhao, C., Li, C., & Efferth, T. (2013) · Toxicology Letters · doi:10.1016/j.toxlet.2013.03.008 · Cited by 40
Selective cerebral vascular dysfunction in Mn-SOD-deficient mice
Faraci, F., Modrick, M., Lynch, C., Didion, L., Fegan, P. E., & Didion, S. (2006) · Journal of Applied Physiology · doi:10.1152/japplphysiol.00939.2005
“Increasing evidence suggests that oxidative stress has diverse effects on vascular structure and function (8,9, 15 ,17).”
Endothelial Akt activation by hyperoxia: Role in cell survival
Ahmad, A. M., Ahmad, S., Chang, L., Schaack, J. B., & White, C. W. (2005) · Free Radical Biology and Medicine · doi:10.1016/j.freeradbiomed.2005.10.045 · Cited by 42
Effects of lysophosphatidylcholine on monolayer cell permeability of human coronary artery endothelial cells
Yan, S., Chai, H., Wang, H., Yang, H., Nan, B., Yao, Q., & Chen, C. (2005) · Surgery · doi:10.1016/j.surg.2005.06.027 · Cited by 40
The Endogenous Estradiol Metabolite 2-Methoxyestradiol Reduces Atherosclerotic Lesion Formation in Female Apolipoprotein E-Deficient Mice
Fagman, J. B., Bergström, G. M. L., Krettek, A., Sjöberg, S., Borén, J., & Tivesten, Å. (2007) · Endocrinology · doi:10.1210/en.2007-0259 · Cited by 40
FoxOs: Unifying Links Between Oxidative Stress and Skeletal Homeostasis
Kousteni, S. (2011) · Current Osteoporosis Reports · doi:10.1007/s11914-011-0054-3 · Cited by 42
Air Pollution Upregulates Endothelial Cell Procoagulant Activity via Ultrafine Particle-Induced Oxidant Signaling and Tissue Factor Expression
Snow, S. J., Cheng, W., Wolberg, A. S., & Carraway, M. S. (2014) · Toxicological Sciences · doi:10.1093/toxsci/kfu071 · Cited by 41
“ROS are implicated in the pathogenesis and progression of several CV diseases including atherosclerosis, hypertension, and diabetes (Madamanchi et al., 2005).”
Background
Correction: Impaired Transcriptional Activity of Nrf2 in Age-Related Myocardial Oxidative Stress Is Reversible by Moderate Exercise Training
Gounder, S. S., Kannan, S., Devadoss, D., Miller, C. J., Whitehead, K. J., Odelberg, S. J., Firpo, M. A., Paine, R., Hoidal, J. R., Abel, E. D., et al. (2012) · PLoS ONE · doi:10.1371/annotation/8690bb36-3c5d-48a6-b3be-39a2b50896e1 · Cited by 40
Chlorpyrifos Induced Cardiotoxicity in Rats and the Protective Role of Quercetin and Catechin
Baş, H., & Kalender, Y. (2011) · DergiPark (Istanbul University) · Cited by 39
Oligoproline-derived nanocarrier for dual stimuli-responsive gene delivery
Gupta, M. K., Lee, S. H., Crowder, S. W., Wang, X., Hofmeister, L. H., Nelson, C. E., Bellan, L. M., Duvall, C. L., & Sung, H. (2015) · Journal of Materials Chemistry B · doi:10.1039/c5tb00988j · Cited by 39
Potentially Reduced Exposure Cigarettes Accelerate Atherosclerosis: Evidence for the Role of Nicotine
Catanzaro, D. F., Zhou, Y. Y., Chen, R., Yu, F., Catanzaro, S., Lorenzo, M. S. D., Subbaramaiah, K., Zhou, X. K., Praticò, D., Dannenberg, A. J., et al. (2007) · Cardiovascular Toxicology · doi:10.1007/s12012-007-0027-z · Cited by 39
“NAD(P)H oxidases [21], although other sources of reactive oxygen species have also been implicated [22].”
Background
Cytochrome P450 1B1 Contributes to the Development of Atherosclerosis and Hypertension in Apolipoprotein E–Deficient Mice
Song, C. Y., Ghafoor, K., Ghafoor, H. U., Khan, N. S., Thirunavukkarasu, S., Jennings, B. L., Estes, A. M., Zaidi, S., Bridges, D., Tso, P., et al. (2015) · Hypertension · doi:10.1161/hypertensionaha.115.06427 · Cited by 39
SIRT3 inhibits cardiac hypertrophy by regulating PARP-1 activity
Feng, X., Wang, Y., Chen, W., Xu, S., Li, L., Geng, Y., Shen, A., Gao, H., Zhang, L., & Liu, S. (2020) · Aging · doi:10.18632/aging.102862 · Cited by 39
Antioxidant, molecular docking and computational investigation of new flavonoids
Tajammal, A., Siddiqa, A., Irfan, A., Azam, M., Hafeez, H., Munawar, M. A., & Basra, M. A. R. (2021) · Journal of Molecular Structure · doi:10.1016/j.molstruc.2021.132189 · Cited by 41
Cellular impact of combinations of endosulfan, atrazine, and chlorpyrifos on human primary hepatocytes and HepaRG cells after short and chronic exposures
Nawaz, A., Razpotnik, A., Rouimi, P., de Sousa, G., Cravedi, J., & Rahmani, R. (2013) · Cell Biology and Toxicology · doi:10.1007/s10565-013-9266-x · Cited by 39
Homocysteine from endothelial cells promotes LDL nitration and scavenger receptor uptake
Griffiths, H. R., Aldred, S., Dale, C., Nakano, E., Kitas, G. D., Grant, M. M., Nugent, D., Taiwo, F. A., Li, L., & Powers, H. J. (2005) · Free Radical Biology and Medicine · doi:10.1016/j.freeradbiomed.2005.08.039 · Cited by 39
Whey protein hydrolysate and branched-chain amino acids downregulate inflammation-related genes in vascular endothelial cells
Silva, M. S. D., Bigo, C., Barbier, O., & Rudkowska, I. (2017) · Nutrition Research · doi:10.1016/j.nutres.2017.01.005 · Cited by 39
“Furthermore, antioxidant enzymes such as superoxide dismutases (SOD) can neutralize reactive oxygen species, which have been linked to endothelial dysfunction [13].”
Background
Red wine intake but not other alcoholic beverages increases total antioxidant capacity and improves pro-inflammatory profile after an oral fat diet in healthy volunteers
Torres, A. H., Cachofeiro, V., Millán, J., Lahera, V., Nieto, M. L., Martín, R., Bello, E., & Alvarez-Sala, L. A. (2015) · Revista Clínica Española · doi:10.1016/j.rce.2015.07.002 · Cited by 38
Brazilin Ameliorates High Glucose-Induced Vascular Inflammation via Inhibiting ROS and CAMs Production in Human Umbilical Vein Endothelial Cells
Jayakumar, T., Chang, C.-C., Lin, S.-L., Huang, Y., Hu, C.-M., Elizebeth, A. R., Lin, S., & Choy, C. (2014) · BioMed Research International · doi:10.1155/2014/403703 · Cited by 40
“ROS can modify endothelial function by a variety of mechanisms, such as peroxidation of membrane lipids, activation of NF-κB, and decrease of the availability of nitric oxide (NO) [6].”
Background
Novel links among peroxiredoxins, endothelial dysfunction, and severity of atherosclerosis in type 2 diabetic patients with peripheral atherosclerotic disease
Eter, E. A. E., Al‐Masri, A. A., Habib, S., Zamil, H. A., Hersi, A., Hussein, F. A., & Omran, M. A. (2013) · Cell Stress and Chaperones · doi:10.1007/s12192-013-0442-y · Cited by 40
Induction of brain natriuretic peptide and monocyte chemotactic protein-1 gene expression by oxidized low-density lipoprotein: relevance to ischemic heart failure
Chandrakala, A. N., Sukul, D., Selvarajan, K., Sai‐Sudhakar, C., Sun, B., & Parthasarathy, S. (2011) · American Journal of Physiology-Cell Physiology · doi:10.1152/ajpcell.00116.2011 · Cited by 38
“SEVERAL PLASMA BIOCHEMICAL markers of oxidative stress have been noted to be increased in heart failure (HF) patients (3, 23, 30, 34).”
Background
Imbalance between Endothelial Damage and Repair: A Gateway to Cardiovascular Disease in Systemic Lupus Erythematosus
Mak, A., & Kow, N. Y. (2014) · BioMed Research International · doi:10.1155/2014/178721 · Cited by 41
“Oxidative stress Oxidative stress increases with higher disease activity of SLE [83].”
Background
Inhibitory effects of Kaempferia parviflora extract on monocyte adhesion and cellular reactive oxygen species production in human umbilical vein endothelial cells
Horigome, S., Yoshida, I., Ito, S., Inohana, S., Fushimi, K., Nagai, T., Yamaguchi, A., Fujita, K., Satoyama, T., Katsuda, S.-I., et al. (2015) · European Journal of Nutrition · doi:10.1007/s00394-015-1141-5 · Cited by 38
“This also leads to the formation of ox-LDL, which activates NF-κB by binding to ox-LDL receptor-1 (LOX-1) [36] and is taken up by macrophages via scavenger receptors, resulting in the formation of activated foam cells [32, 37].”
Background
Electrochemical reverse engineering: A systems-level tool to probe the redox-based molecular communication of biology
Li, J., Liu, Y., Kim, E., March, J. C., Bentley, W. E., & Payne, G. F. (2016) · Free Radical Biology and Medicine · doi:10.1016/j.freeradbiomed.2016.12.029 · Cited by 38
The role of P2Y12 receptor in ischemic stroke of atherosclerotic origin
Gao, Y., Yu, C. J., Pi, S., Mao, L., & Hu, B. (2018) · Cellular and Molecular Life Sciences · doi:10.1007/s00018-018-2937-2 · Cited by 38
Computationally Designed Sesamol Derivatives Proposed as Potent Antioxidants
Castro-González, L. M., Alvarez-Idaboy, J. R., & Galano, A. (2020) · ACS Omega · doi:10.1021/acsomega.0c00898 · Cited by 41
Effects of menstrual cycle and menopause on internal carotid artery shear-mediated dilation in women
Iwamoto, E., Sakamoto, R., Tsuchida, W., Yamazaki, K., Kamoda, T., Neki, T., Katayose, M., & Casey, D. P. (2020) · American Journal of Physiology-Heart and Circulatory Physiology · doi:10.1152/ajpheart.00810.2020 · Cited by 38
Sirtuin 3 (SIRT3) Pathways in Age-Related Cardiovascular and Neurodegenerative Diseases
Silaghi, C. N., Farcas, M. F., & Craciun, A. M. (2021) · Biomedicines · doi:10.3390/biomedicines9111574 · Cited by 37
“Excessive mitochondrial ROS (mtROS) leads to mitochondrial DNA damage, respiratory chain dysfunction, endothelial inflammation, atherogenesis and HT [92,93].”
Background
Changes in Expression of the Antioxidant Enzyme SOD3 Occur Upon Differentiation of Human Bone Marrow-Derived Mesenchymal Stem Cells In Vitro
Nightingale, H., Kemp, K. C., Gray, E., Hares, K. M., Mallam, E., Scolding, N. J., & Wilkins, A. (2011) · Stem Cells and Development · doi:10.1089/scd.2011.0516 · Cited by 38
Pseudoxanthoma Elasticum: Oxidative Stress and Antioxidant Diet in a Mouse Model (Abcc6−/−)
Li, Q., Jiang, Q., & Uitto, J. (2007) · Journal of Investigative Dermatology · doi:10.1038/sj.jid.5701145 · Cited by 38
“Oxidative stress is encountered in a number of degenerative disease processes, as exemplified by atherosclerosis (Lankin et al., 2005; Madamanchi et al., 2005).”
Background
Vitamin D3 up-regulating protein 1 (VDUP1) antisense DNA regulates tumorigenicity and melanogenesis of murine melanoma cells via regulating the expression of fas ligand and reactive oxygen species
Song, H., Cho, D., Jeon, J. H., Han, S. H., Hur, D. Y., Kim, Y. S., & Choi, I. (2003) · Immunology Letters · doi:10.1016/s0165-2478(03)00024-5 · Cited by 38
Cardiovascular and psychiatric characteristics associated with oxidative stress markers among adolescents with bipolar disorder
Hatch, J., Andreazza, A., Olowoyeye, O., Rezin, G. T., Moody, A., & Goldstein, B. I. (2015) · Journal of Psychosomatic Research · doi:10.1016/j.jpsychores.2015.04.005 · Cited by 39
“Traditional CVD risk factors, including HDL, BMI, LDL, glucose, as well as high diastolic and systolic blood pressure, have all been shown to be associated with oxidative stress [19,38].”
Background
Mechanism by Which Inflammation and Oxidative Stress Induce Mineralocorticoid Receptor Gene Expression in Aging Vascular Smooth Muscle Cells
Ibarrola, J., Lu, Q. R., Zennaro, M., & Jaffe, I. Z. (2022) · Hypertension · doi:10.1161/hypertensionaha.122.19213 · Cited by 37
Common variants of four bilirubin metabolism genes and their association with serum bilirubin and coronary artery disease in Chinese Han population
Lin, R., Wang, Y., Wáng, Y., Fu, W., Zhang, D., Zheng, H., Yu, T. Y., Wang, Y., Shen, M., Lei, R., et al. (2009) · Pharmacogenetics and Genomics · doi:10.1097/fpc.0b013e328328f818 · Cited by 37
Reactive Oxygen Species Related Noncoding RNAs as Regulators of Cardiovascular Diseases
Dong, Y., Xu, W., Liu, C., Liu, P., Li, P., & Wang, K. (2019) · International Journal of Biological Sciences · doi:10.7150/ijbs.30464 · Cited by 38
“Furthermore, the redox imbalance induced by altered ROS generally influences signaling pathways that contribute to cardiomyocytes’ apoptosis, ER stress, and autophagy, which ultimately contribute to a wide range of cardiovascular diseases, such as myocardial infarction, stroke, atherosclerosis and heart failure [14, 1…”
Background
Urinary Biomarkers of Inflammation and Oxidative Stress Are Elevated in Obese Children and Correlate with a Marker of Endothelial Dysfunction
Selvaraju, V., Ayine, P., Fadamiro, M., Babu, J. R., Brown, M. D., & Geetha, T. (2019) · Oxidative Medicine and Cellular Longevity · doi:10.1155/2019/9604740 · Cited by 37
“Oxidative stress also plays a significant role in inducing endothelial dysfunction [20], which could lead to atherogenesis [21].”
Histone Methylation and Oxidative Stress in Cardiovascular Diseases
Yi, X., Zhu, Q., Wu, X., Tan, T.-T., & Jiang, X.-J. (2022) · Oxidative Medicine and Cellular Longevity · doi:10.1155/2022/6023710 · Cited by 42
“Under normal conditions, nearly 98% of electrons transferred in the ETC are coupled with the generation of ATP, while the rest of the electrons leak out and account for O2 - production [9, 10].”
The Relationship between Cancer and Paraoxonase 1
Medina‐Díaz, I. M., Ponce-Ruiz, N., Rojas‐García, A. E., Zambrano‐Zaragoza, J. F., Bernal-Hernández, Y. Y., González-Arias, C. A., Barrón-Vivanco, B. S., & Herrera‐Moreno, J. F. (2022) · Antioxidants · doi:10.3390/antiox11040697 · Cited by 42
Retinal and Choroidal Thinning—A Predictor of Coronary Artery Occlusion?
Matulevičiūtė, I., Sidaraitė, A., Tatarūnas, V., Veikutienė, A., Dobilienė, O., & Žaliūnienė, D. (2022) · Diagnostics · doi:10.3390/diagnostics12082016 · Cited by 39
“Waghamare and others carried out a study where choroidal thickness was measured and compared in healthy controls and patients with one of the main CVD factors— hypertension.”
Background
Blood flow modulation of vascular dynamics
Lee, J., Packard, R. R. S., & Hsiai, T. K. (2015) · Current Opinion in Lipidology · doi:10.1097/mol.0000000000000218 · Cited by 37
“Haemodynamic force, such as wall shear stress on the endothelial cells, modulates inflammatory and metabolic effects in the vascular system [1].”
Background
Fishing for fire: strategies for biological targeting and criteria for material design in anti‐inflammatory therapies
d’Arcy, R., & Tirelli, N. (2014) · Polymers for Advanced Technologies · doi:10.1002/pat.3264 · Cited by 38
Hepatic steatosis and vascular disease.
Santoliquido, A., Campli, C. D., Miele, L., Gabrieli, M. L., Forgione, A., Zocco, M. A., Lupascu, A., Giorgio, A. D., Flore, R. A., Pola, P., et al. (2005) · PubMed · Cited by 37
Phytomedicinal activity of Terminalia arjuna against carbon tetrachloride induced cardiac oxidative stress
Manna, P., Sinha, M., & Sil, P. C. (2007) · Pathophysiology · doi:10.1016/j.pathophys.2007.05.002 · Cited by 37
The role of oxidative stress in the pathogenesis of Alzheimer's disease
Filipčík, P., Cente, M., Ferencĭk, M., Hulín, I., & Novak, M. (2006) · Bratislavské lekárske listy/Bratislava medical journal · doi:10.1007/bf03656857 · Cited by 37
A sensitive chemiluminescence detection approach for determination of 2,4-dinitrophenylhydrazine derivatized aldehydes using online UV irradiation – luminol CL reaction. Application to the HPLC analysis of aldehydes in oil samples
El‐Maghrabey, M., Suzuki, H., Kishikawa, N., & Kuroda, N. (2021) · Talanta · doi:10.1016/j.talanta.2021.122522 · Cited by 35
Nitric oxide and the cardiovascular system: cell activation, vascular reactivity and genetic variant.
Dias, R. G., Negrão, C. E., & Krieger, M. H. (2011) · PubMed · Cited by 36
Anti-inflammatory agents as modulators of the inflammation in adipose tissue: A systematic review
Nascimento, S. S. D. C., de Queiroz, J. L. C., de Medeiros, A. F., Nunes, A. C. D. F., Piuvezam, G., Maciel, B. L. L., Passos, T. S., & Morais, A. H. D. A. (2022) · PLoS ONE · doi:10.1371/journal.pone.0273942 · Cited by 37
4-hydroxynonenal contributes to macrophage foam cell formation through increased expression of class A scavenger receptor at the level of translation
Yun, M. R., Im, D., Lee, S. J., Woo, J. W., Hong, K. W., Bae, S. S., & Kim, C. D. (2008) · Free Radical Biology and Medicine · doi:10.1016/j.freeradbiomed.2008.04.014 · Cited by 36
Estrogen and cerebrovascular regulation in menopause
Raz, L. (2014) · Molecular and Cellular Endocrinology · doi:10.1016/j.mce.2014.01.015 · Cited by 35
Information processing through a bio-based redox capacitor: Signatures for redox-cycling
Liu, Y., Kim, E., White, I. M., Bentley, W. E., & Payne, G. F. (2014) · Bioelectrochemistry · doi:10.1016/j.bioelechem.2014.03.012 · Cited by 35
Impaired Vascular Redox Signaling in the Vascular Complications of Obesity and Diabetes Mellitus
Akoumianakis, I., & Antoniades, C. A. (2017) · Antioxidants and Redox Signaling · doi:10.1089/ars.2017.7421 · Cited by 37
“Furthermore, ROS are able to oxidise low density 128 lipoproteins (LDL), which are then internalised by macrophages and deposited in the 129 subendothelial space at sites of dysfunctional endothelium, this being a key step for the 130 initiation of atherogenesis (121,155).”
The protective impact of betaine on the tissue structure and renal function in isoproterenol‐induced myocardial infarction in rat
Ghartavol, M. M., Aziz, S. G., Babaei, G., Farjah, G. H., & Ansari, M. H. K. (2019) · Molecular Genetics & Genomic Medicine · doi:10.1002/mgg3.579 · Cited by 35
The Protective Mechanism of Cannabidiol in Cardiac Injury: A Systematic Review of Non-Clinical Studies
Shayesteh, M. R. H., Haghi‐Aminjan, H., Mousavi, M. J., Momtaz, S., & Abdollahi, M. R. (2019) · Current Pharmaceutical Design · doi:10.2174/2210327909666190710103103 · Cited by 35
Use of metabolomics to elucidate the metabolic perturbation associated with hypertension in a black South African male cohort: the SABPA study
van Deventer, C. A., Lindeque, J. Z., van Rensburg, P. J., Malan, L., van der Westhuizen, F. H., & Louw, R. (2014) · Journal of the American Society of Hypertension · doi:10.1016/j.jash.2014.11.007 · Cited by 37
Protective effects of ligustrazine on TNF-α-induced endothelial dysfunction
Wu, H., Hao, J., Wang, S., Jin, B., & Chen, X.-B. (2011) · European Journal of Pharmacology · doi:10.1016/j.ejphar.2011.10.046 · Cited by 35
Increased superoxide anion production is associated with early atherosclerosis and cardiovascular dysfunctions in a rabbit model
Collin, B., Busseuil, D., Zeller, M., Perrin‐Sarrado, C., Barthez, O., Duvillard, L., Vergely, C., Bardou, M., Dumas, M., Cottin, Y., et al. (2006) · Molecular and Cellular Biochemistry · doi:10.1007/s11010-006-9263-y · Cited by 35
“Thus, the mitochondrial dysfunction can be involved in cardiovascular diseases via their involvement in oxidative stress [51, 52].”
Background
Low fat adiponectin expression is associated with oxidative stress in nondiabetic humans with chronic kidney disease—impact on plasma adiponectin concentration
Barazzoni, R., Bernardi, A., Biasia, F., Semolic, A., Bosutti, A., Mucci, M., Dore, F., Zanetti, M., & Guarnieri, G. (2007) · American Journal of Physiology-Regulatory, Integrative and Comparative Physiology · doi:10.1152/ajpregu.00745.2006 · Cited by 35
“Oxidative stress can contribute to insulin resistance and cardiovascular risk in chronic disease states, including obesity and diabetes (9, 11, 25), due to imbalance between oxidant generation and antioxidant defense systems.”
Background
Paraoxonase-1 Inhibits Oxidized Low-Density Lipoprotein-Induced Metabolic Alterations and Apoptosis in Endothelial Cells: A Nondirected Metabolomic Study
García‐Heredia, A., Marsillach, J., Rull, A., Triguero, I., Fort-Gallifa, I., Mackness, B., Mackness, M., Shih, D. M., Joven, J., & Camps, J. (2013) · Mediators of Inflammation · doi:10.1155/2013/156053 · Cited by 35
Developmental toxicity and molecular responses of marine medaka (Oryzias melastigma) embryos to ciguatoxin P-CTX-1 exposure
Yan, M., Leung, P. T. Y., Ip, J. C., Cheng, J., Wu, J., Gu, J., & Lam, P. K. S. (2017) · Aquatic Toxicology · doi:10.1016/j.aquatox.2017.02.006 · Cited by 35
“In addition to its role on inflammatory responses, TNF- ̨ is reported to stimulate ROS production (Madamanchi et al., 2005; Mariappan et al., 2007).”
Ginsenoside Rb1 Reverses H2O2-induced Senescence in Human Umbilical Endothelial Cells
Liu, D., Chen, Y., Liu, Y., Hao, B., Zhou, B., Wu, L., Wang, M., Chen, L., Wu, W.-K., & Qian, X. (2011) · Journal of Cardiovascular Pharmacology · doi:10.1097/fjc.0b013e31823c1d34 · Cited by 34
Synergism Between Platelet-Activating Factor-Like Phospholipids and Peroxisome Proliferator-Activated Receptor γ Agonists Generated During Low Density Lipoprotein Oxidation That Induces Lipid Body Formation in Leukocytes
de Assis, E. F., Silva, A. R., Caiado, L. F. C., Marathe, G. K., Zimmerman, G. A., Prescott, S. M., McIntyre, T. M., Bozza, P. T., & Castro-Faria-Neto, H. C. (2003) · The Journal of Immunology · doi:10.4049/jimmunol.171.4.2090 · Cited by 34
Effect of Advanced Glycation End Products on Oxidative Stress and Senescence of Trabecular Meshwork Cells
Park, C. H., & Kim, J. W. (2012) · Korean Journal of Ophthalmology · doi:10.3341/kjo.2012.26.2.123 · Cited by 33
“Our results are consistent with previous studies that found that AGE enhanced peroxynitrite formation mediated by NO and superoxide in retinal neurons [58,61,62].”
BackgroundResult
Ginsenoside Rd Attenuates DNA Damage by Increasing Expression of DNA Glycosylase Endonuclease VIII-like Proteins after Focal Cerebral Ischemia
Yang, L.-X., Zhang, X., & Zhao, G. (2016) · Chinese Medical Journal · doi:10.4103/0366-6999.187851 · Cited by 33
“[2] Oxidative DNA damage can trigger dysfunction and death of brain neurons and eventually lead to poor outcomes.[3] Maintaining genome integrity of neurons after ischemia is considered to be an important strategy to improve the prognosis of ischemic strokes.”
Background
Optimisation of ultrasound‐assisted extraction of rosehip (Rosa canina L.) with response surface methodology
İlbay, Z., Şahin, S., & Kırbaşlar, Ş. İ. (2013) · Journal of the Science of Food and Agriculture · doi:10.1002/jsfa.6104 · Cited by 33
Oxidative Stress through Activation of NAD(P)H Oxidase in Hypertensive Mice with Spontaneous Intracranial Hemorrhage
Wakisaka, Y., Miller, J. D., Chu, Y., Baumbach, G. L., Wilson, S., Faraci, F. M., Sigmund, C. D., & Heistad, D. D. (2008) · Journal of Cerebral Blood Flow & Metabolism · doi:10.1038/jcbfm.2008.7 · Cited by 34
Overexpression of mitochondrial antioxidant manganese superoxide dismutase (MnSOD) provides protection against AZT- or 3TC-induced endothelial dysfunction
Glover, M. C., Hebert, V. Y., Nichols, K., Xue, S. Y., Thibeaux, T. M., Zavecz, J. H., & Dugas, T. R. (2014) · Antiviral Research · doi:10.1016/j.antiviral.2014.09.010 · Cited by 33
Rapeseed oil fortified with micronutrients reduces atherosclerosis risk factors in rats fed a high-fat diet
Xu, J., Zhou, X., Deng, Q., Huang, Q., Yang, J., & Huang, F. (2011) · Lipids in Health and Disease · doi:10.1186/1476-511x-10-96 · Cited by 33
“In fact, oxidative stress is the unifying mechanism for many CVD risk factors [26].”
Background
Mild endothelial dysfunction in Sirt3 knockout mice fed a high-cholesterol diet: protective role of a novel C/EBP-β-dependent feedback regulation of SOD2
Winnik, S. H., Gaul, D. S., Siciliani, G., Lohmann, C., Pasterk, L., Calatayud, N., Weber, J., Eriksson, U., Auwerx, J. H., van Tits, L. J., et al. (2016) · Basic Research in Cardiology · doi:10.1007/s00395-016-0552-7 · Cited by 33
“Excess endothelial reactive oxygen species (ROS), subsequent mitochondrial DNA damage and progressive respiratory chain dysfunction are essential in the development of endothelial dysfunction, an early event in and an independent predictor of cardiovascular disease [17, 28, 29, 33, 36, 40, 41, 48].”
Background
Persistent Oxidative Stress after Myocardial Infarction Treated by Percutaneous Coronary Intervention
Nikolić-Heitzler, V., Rabuzin, F., Tatzber, F., Vrkić, N., Bulj, N., Borović, S., Wonisch, W., Sunko, B. M., & Žarković, N. (2006) · The Tohoku Journal of Experimental Medicine · doi:10.1620/tjem.210.247 · Cited by 33
Lipid droplets formation in human endothelial cells in response to polyunsaturated fatty acids and 1‐methyl‐nicotinamide (MNA); confocal Raman imaging and fluorescence microscopy studies
Majzner, K., Chłopicki, S., & Barańśka, M. (2015) · Journal of Biophotonics · doi:10.1002/jbio.201500134 · Cited by 32
Ezetimibe Treatment Lowers Indicators of Oxidative Stress in Hypercholesterolemic Subjects with High Oxidative Stress
Kostapanos, M. S., Spyrou, A., Tellis, C. C., Gazi, I. F., Tselepis, A. D., Elisaf, M., & Liberopoulos, E. N. (2011) · Lipids · doi:10.1007/s11745-011-3539-z · Cited by 32
“Hyperlipidemia and increased oxidative stress often coexist and their combined unfavorable effects may result in increased atherogenicity [13].”
Background
Thioredoxin in coronary culprit lesions: Possible relationship to oxidative stress and intraplaque hemorrhage
Nishihira, K., Yamashita, A., Imamura, T., Hatakeyama, K., Sato, Y. Z., Nakamura, H., Yodoi, J., Ogawa, H., Kitamura, K., & Asada, Y. (2008) · Atherosclerosis · doi:10.1016/j.atherosclerosis.2008.03.005 · Cited by 32
Assessment of genotoxicity of catecholics using impedimetric DNA-biosensor
Ensafi, A. A., Amini, M., & Rezaei, B. (2013) · Biosensors and Bioelectronics · doi:10.1016/j.bios.2013.09.030 · Cited by 32
Effects of Phyllanthus reticulatus on lipid profile and oxidative stress in hypercholesterolemic albino rats
Maruthappan, V., & Shree, K. S. (2010) · Indian Journal of Pharmacology · doi:10.4103/0253-7613.71923 · Cited by 32
“Oxidative stress is an important event in the development and maintenance of atherosclerosis.[29] Protein carbonyl is the most widely used biomarker for oxidative damage to proteins by multiple forms reactive oxygen species (ROS).”
Trends and performance of oxidative stress research from 1991 to 2010
Wen, H., & Huang, Y. (2011) · Scientometrics · doi:10.1007/s11192-011-0535-2 · Cited by 36
“…Pollution Control, College of Environmental Sciences and Engineering, Peking University, Beijing 100871, China e-mail: yhuang@pku.edu.cn The core definition of oxidative stress is the imbalance between oxidants and antioxidants in favor of the oxidants, potentially leading to damage (John 2000).”
Background
Oxidative Stress in Cardiovascular Inflammation: Its Involvement in Autoimmune Responses
Profumo, E., Buttari, B., & Riganò, R. (2011) · International Journal of Inflammation · doi:10.4061/2011/295705 · Cited by 33
“Excessive oxidative stress and chronic inflammation are both major characteristics of atherosclerosis [18, 19].”
Background
Apolipoprotein E −/− Mice Lacking Hemopexin Develop Increased Atherosclerosis via Mechanisms That Include Oxidative Stress and Altered Macrophage Function
Mehta, N. U., Grijalva, V. R., Hama, S. Y., Wagner, A. C., Navab, M., Fogelman, A. M., & Reddy, S. T. (2016) · Arteriosclerosis Thrombosis and Vascular Biology · doi:10.1161/atvbaha.115.306991 · Cited by 32

Authors: Madamanchi, N. R., Vendrov, A., & Runge, M. S.
Affiliations: University of North Carolina at Chapel Hill (US)
ORCID: Madamanchi, Nageswara R.
Vendrov, Aleksandr
Runge, Marschall S.
Venue: Arteriosclerosis, Thrombosis, and Vascular Biology
Publisher: Ovid Technologies (Wolters Kluwer Health)
Published: 2005-01
Volume: 25
Issue: 1
Pages: 29-38
ISSN: 1079-5642 (ISSN-L)
1524-4636
DOI: 10.1161/01.atv.0000150649.39934.13
OpenAlex: W2109698138
Cited by: 1,618
References: 135
Citations by year: 2026: 47 · 2025: 46 · 2024: 42 · 2023: 53 · 2022: 61 · 2021: 62 · 2020: 73 · 2019: 73 · 2018: 57 · 2017: 75 · 2016: 78 · 2015: 105 · 2014: 111 · 2013: 104 · 2012: 95
FWCI: 7.02
Type: journal article
Language: en
Open access: closed
Locations: Arteriosclerosis Thrombosis and Vascular Biology (publishedVersion)
PubMed (publishedVersion)
Topics: Health Sciences › Medicine › Physiology (medical) › Nitric Oxide and Endothelin Effects
Health Sciences › Medicine › Biochemistry (medical) › Antioxidant Activity and Oxidative Stress
Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Redox biology and oxidative stress
Keywords: oxidative stress, reactive oxygen species, atherosclerosis, vascular disease, cardiovascular disease, NADPH oxidase, xanthine oxidase, lipoxygenase, nitric oxide synthase uncoupling, vascular inflammation, atherogenesis, antioxidants, redox signaling, biomarkers, cardiovascular risk factors
MeSH terms: Animals, Humans, Vascular Diseases, Oxidative Stress

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review

Oxidative Stress and Vascular Disease

Abstract

Overview

Integrity signals

Verification

Claims the paper makes

Claims other papers make about this one

Self-check

Citations

Cites

Cited by

Related

Metadata

Record

About these checks