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Cited by 261

The DOI matches its Crossref record; 128 of the 128 references it cites were found in a registry or an index.

Manipulating Angiogenesis: From Basic Science to the Bedside

Open access
Pepper, Michael S.·1997·Arteriosclerosis, Thrombosis, and Vascular Biology

Abstract

Abstract Considerable progress has been made recently in understanding the molecular mechanisms of angiogenesis, which like most other biological processes is the result of subtle and often complex interactions between molecules that have regulatory (eg, cytokines and their receptors) and effector (eg, extracellular matrix, integrins, and proteases) functions. The title of this review was chosen to reflect a recent trend in which knowledge acquired through a molecular/cell biological approach is being rapidly transferred to the clinical setting. As a result, by manipulating angiogenesis either positively or negatively, considerable therapeutic benefit can now be envisaged in physiological and pathological settings in which neovascularization is a prominent component.

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Crossref · 10.1161/01.atv.17.4.605

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Metadata

Authors
Pepper, M. S.
Venue
Arteriosclerosis, Thrombosis, and Vascular Biology
Published
1997-04
Volume
17
Issue
4
Pages
605-619
ISSN
  • 1079-5642 (ISSN-L)
  • 1524-4636
OpenAlex
W2042397185
Cited by
261
References
128
FWCI
6.97
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Manipulating Angiogenesis: From Basic Science to the Bedside. https://app.cochat.ai/papers/10.1161/01.atv.17.4.605
The paper
Pepper, M. S. (1997). Manipulating Angiogenesis: From Basic Science to the Bedside. Arteriosclerosis, Thrombosis, and Vascular Biology. https://doi.org/10.1161/01.atv.17.4.605

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.