The DOI matches its Crossref record; 9 of the 9 references it cites were found in a registry or an index.
Apoptosis: Basic Mechanisms and Implications for Cardiovascular Disease
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Citations
Cites
Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
Oltval, Z. N., Milliman, C. L., & Korsmeyer, S. J. (1993) · Cell · doi:10.1016/0092-8674(93)90509-o · Cited by 6,003
Tumor suppressor p53 is a direct transcriptional activator of the human bax gene
Toshiyuki, M., & Reed, J. C. (1995) · Cell · doi:10.1016/0092-8674(95)90412-3 · Cited by 4,063
p53 is required for radiation-induced apoptosis in mouse thymocytes
Lowe, S. W., Schmitt, E. M., Smith, S. W., Osborne, B. A., & Jacks, T. (1993) · Nature · doi:10.1038/362847a0 · Cited by 2,864
Martin, S. J., Reutelingsperger, C. P., McGahon, A. J., Rader, J. A., van Schie, R. C., LaFace, D. M., & Green, D. R. (1995) · The Journal of experimental medicine · doi:10.1084/jem.182.5.1545 · Cited by 2,824
Liu, Z.-G., Hsu, H., Goeddel, D. V., & Karin, M. (1996) · Cell · doi:10.1016/s0092-8674(00)81375-6 · Cited by 1,937
Genetic control of programmed cell death in the nematode C. elegans
Ellis, H. (1986) · Cell · doi:10.1016/0092-8674(86)90004-8 · Cited by 1,828
Apoptosis in the Failing Human Heart
Olivetti, G., Abbi, R., Quaini, F., Kajstura, J., Cheng, W., Nitahara, J. A., Quaini, E., Di Loreto, C., Beltrami, C. A., Krajewski, S., et al. (1997) · New England Journal of Medicine · doi:10.1056/nejm199704173361603 · Cited by 1,673
TNF-Dependent Recruitment of the Protein Kinase RIP to the TNF Receptor-1 Signaling Complex
Hsu, H., Huang, J., Shu, H.-B., Baichwal, V., & Goeddel, D. V. (1996) · Immunity · doi:10.1016/s1074-7613(00)80252-6 · Cited by 1,182
Apoptosis in human acute myocardial infarction.
Saraste, A., Pulkki, K., Kallajoki, M., Henriksen, K., Parvinen, M., & Voipio-Pulkki, L.-M. (1997) · Circulation · doi:10.1161/01.cir.95.2.320 · Cited by 825 · cited 1×
“In human postmortem studies of myocardial infarction, apoptotic cardiomyocytes appeared to be predominantly localized in the hypoperfused border zone between the central infarct area and noncompromised myocardial tissue.(218,219) Interestingly, myocytes in the peri-infarct region were shown to upregulate the apoptotic…”
Background
Cited by
Diabetic Cardiomyopathy: Evidence, Mechanisms, and Therapeutic Implications
Fang, Z. Y., Prins, J. B., & Marwick, T. H. (2004) · Endocrine Reviews · doi:10.1210/er.2003-0012 · Cited by 912
Bernardo, B. C., Weeks, K. L., Pretorius, L., & McMullen, J. R. (2010) · Pharmacology & Therapeutics · doi:10.1016/j.pharmthera.2010.04.005 · Cited by 894
Kim, I., Kim, H. G., So, J.-N., Kim, J. H., Kwak, H. J., & Koh, G. Y. (2000) · Circulation Research · doi:10.1161/01.res.86.1.24 · Cited by 649
Myocyte Death, Growth, and Regeneration in Cardiac Hypertrophy and Failure
Nadal-Ginard, B., Kajstura, J., Leri, A., & Anversa, P. (2003) · Circulation Research · doi:10.1161/01.res.0000053618.86362.df · Cited by 503
Nitric Oxide Inhibits Caspase-3 by S-Nitrosationin Vivo
Rössig, L., Fichtlscherer, B., Breitschopf, K., Haendeler, J., Zeiher, A. M., Mülsch, A., & Dimmeler, S. (1999) · Journal of Biological Chemistry · doi:10.1074/jbc.274.11.6823 · Cited by 432
Metadata
- Authors
- Haunstetter, A., & Izumo, S.
- Venue
- Circulation Research
- Published
- 1998-06-15
- Volume
- 82
- Issue
- 11
- Pages
- 1111-1129
- ISSN
- 0009-7330 (ISSN-L)
- 1524-4571
- OpenAlex
- W2158326731
- Cited by
- 807
- References
- 9
- FWCI
- 21.27
- Type
- journal article
- Language
- en
- Open access
- closed
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- Haunstetter, A., & Izumo, S. (1998). Apoptosis: Basic Mechanisms and Implications for Cardiovascular Disease. Circulation Research. https://doi.org/10.1161/01.res.82.11.1111
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.