Cited by 48

The DOI matches its Crossref record; 12 of the 12 references it cites were found in a registry or an index.

Inflammatory factors and amyloid β-induced microglial polarization promote inflammatory crosstalk with astrocytes

Xie, Lushuang, Zhang, Ning, Zhang, Qun, Li, Chenyu, Sandhu, Aaron F., III, George Williams, Lin, Sirui, Lv, PeiRan, Liu, Yi, Wu, Qiaofeng, Yu, Shuguang·2020·Aging

Overview

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Integrity signals

Registry

Crossref · 10.18632/aging.103663

Cites

12 of 12 references verified0 citation errors · 0 not checkable

Statistics

Verification

Claims the paper makes

The claims and statistics of this paper haven’t been checked yet.

Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.

A few free checks a day · papers already verified open instantly · results are public

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

Self-check

Percentages, sums and reported differences re-computed from the paper’s own numbers.

The self-check needs the paper’s full text.

Citations

Cites

Flagged first, then by citations

Research works printed but not linked: 22

  1. [1]Reemst K, Noctor SC, Lucassen PJ, Hol EM. The indispensable roles of microglia and astrocytes during brain development. Front Hum Neurosci. 2016; 10: 566. 10.3389/fnhum.2016.00566 27877121 PMC5099170 · DOI · Paper page
  2. [2]Pascual O, Ben Achour S, Rostaing P, Triller A, Bessis A. Microglia activation triggers astrocyte-mediated modulation of excitatory neurotransmission. Proc Natl Acad Sci USA. 2012; 109: E197– 205. 10.1073/pnas.1111098109 22167804 PMC3268269 · DOI · Paper page
  3. [3]Jha MK, Jo M, Kim JH, Suk K. Microglia-astrocyte crosstalk: an intimate molecular conversation. Neuroscientist. 2019; 25: 227– 40. 10.1177/1073858418783959 29931997 · DOI · Paper page
  4. [4]Tang Y, Le W. Differential roles of M1 and M2 microglia in neurodegenerative diseases. Mol Neurobiol. 2016; 53: 1181– 94. 10.1007/s12035-014-9070-5 25598354 · DOI · Paper page
  5. [6]Shi X, Cai X, Di W, Li J, Xu X, Zhang A, Qi W, Zhou Z, Fang Y. MFG-E8 selectively inhibited Aβ-induced microglial M1 polarization via NF-κB and PI3K-Akt pathways. Mol Neurobiol. 2017; 54: 7777– 88. 10.1007/s12035-016-0255-y 27844286 · DOI
  6. [7]Xie L, Wu Q, Tang Y, Zhuang Z, Zhao N, Huang B, Yu S. [Study on electroacupuncture promoting the polarization of M2 phenotype microglia cells in hippocampus of AD model rats]. Zhonghua Zhongyiyao Zazhi. 2018; 33: 1816– 20. https://kns.cnki.net/kcms/detail/detail.aspx?FileName=BXYY201805028&DbName=CJFQ2018
  7. [8]Lian H, Litvinchuk A, Chiang AC, Aithmitti N, Jankowsky JL, Zheng H. Astrocyte-microglia cross talk through complement activation modulates amyloid pathology in mouse models of Alzheimer’s disease. J Neurosci. 2016; 36: 577– 89. 10.1523/JNEUROSCI.2117-15.2016 26758846 PMC4710776 · DOI · Paper page
  8. [9]Litvinchuk A, Wan YW, Swartzlander DB, Chen F, Cole A, Propson NE, Wang Q, Zhang B, Liu Z, Zheng H. Complement C3aR inactivation attenuates tau pathology and reverses an immune network deregulated in tauopathy models and Alzheimer’s disease. Neuron. 2018; 100: 1337– 53.e5. 10.1016/j.neuron.2018.10.031 30415998 PMC6309202 · DOI · Paper page
  9. [10]Qin C, Liu Q, Hu ZW, Zhou LQ, Shang K, Bosco DB, Wu LJ, Tian DS, Wang W. Microglial TLR4-dependent autophagy induces ischemic white matter damage via STAT1/6 pathway. Theranostics. 2018; 8: 5434– 51. 10.7150/thno.27882 30555556 PMC6276098 · DOI
  10. [11]Shi H, Wang XL, Quan HF, Yan L, Pei XY, Wang R, Peng XD. Effects of betaine on LPS-stimulated activation of microglial M1/M2 phenotypes by suppressing TLR4/NF-κB pathways in N9 cells. Molecules. 2019; 24: 367. 10.3390/molecules24020367 30669620 PMC6359206 · DOI
  11. [13]Chen KY, Wang LC. Stimulation of IL-1β and IL-6 through NF-κB and sonic hedgehog-dependent pathways in mouse astrocytes by excretory/secretory products of fifth-stage larval angiostrongylus cantonensis. Parasit Vectors. 2017; 10: 445. 10.1186/s13071-017-2385-0 28950910 PMC5615811 · DOI
  12. [14]Neal M, Luo J, Harischandra DS, Gordon R, Sarkar S, Jin H, Anantharam V, Désaubry L, Kanthasamy A, Kanthasamy A. Prokineticin-2 promotes chemotaxis and alternative A2 reactivity of astrocytes. Glia. 2018; 66: 2137– 57. 10.1002/glia.23467 30277602 PMC6240381 · DOI
  13. [15]Cunha C, Gomes C, Vaz AR, Brites D. Exploring new inflammatory biomarkers and pathways during LPS-induced M1 polarization. Mediators Inflamm. 2016; 2016: 6986175. 10.1155/2016/6986175 28096568 PMC5209629 · DOI · Paper page
  14. [17]Fakhoury M. Microglia and astrocytes in Alzheimer’s disease: implications for therapy. Curr Neuropharmacol. 2018; 16: 508– 18. 10.2174/1570159X15666170720095240 28730967 PMC5997862 · DOI
  15. [18]Shah A, Silverstein PS, Singh DP, Kumar A. Involvement of metabotropic glutamate receptor 5, AKT/PI3K signaling and NF-κB pathway in methamphetamine-mediated increase in IL-6 and IL-8 expression in astrocytes. J Neuroinflammation. 2012; 9: 52. 10.1186/1742-2094-9-52 22420994 PMC3338363 · DOI
  16. [19]Fang M, Yuan Y, Rangarajan P, Lu J, Wu Y, Wang H, Wu C, Ling EA. Scutellarin regulates microglia-mediated TNC1 astrocytic reaction and astrogliosis in cerebral ischemia in the adult rats. BMC Neurosci. 2015; 16: 84. 10.1186/s12868-015-0219-6 26608466 PMC4660684 · DOI
  17. [20]Yao A, Liu F, Chen K, Tang L, Liu L, Zhang K, Yu C, Bian G, Guo H, Zheng J, Cheng P, Ju G, Wang J. Programmed death 1 deficiency induces the polarization of macrophages/microglia to the M1 phenotype after spinal cord injury in mice. Neurotherapeutics. 2014; 11: 636– 50. 10.1007/s13311-013-0254-x 24853068 PMC4121443 · DOI
  18. [22]Francos-Quijorna I, Amo-Aparicio J, Martinez-Muriana A, López-Vales R. IL-4 drives microglia and macrophages toward a phenotype conducive for tissue repair and functional recovery after spinal cord injury. Glia. 2016; 64: 2079– 92. 10.1002/glia.23041 27470986 · DOI
  19. [27]Origlia N, Criscuolo C, Arancio O, Yan SS, Domenici L. RAGE inhibition in microglia prevents ischemia-dependent synaptic dysfunction in an amyloid-enriched environment. J Neurosci. 2014; 34: 8749– 60. 10.1523/JNEUROSCI.0141-14.2014 24966375 PMC4069353 · DOI
  20. [31]Latta CH, Sudduth TL, Weekman EM, Brothers HM, Abner EL, Popa GJ, Mendenhall MD, Gonzalez-Oregon F, Braun K, Wilcock DM. Determining the role of IL-4 induced neuroinflammation in microglial activity and amyloid-β using BV2 microglial cells and APP/PS1 transgenic mice. J Neuroinflammation. 2015; 12: 41. 10.1186/s12974-015-0243-6 25885682 PMC4350455 · DOI
  21. [32]Mussbacher M, Salzmann M, Brostjan C, Hoesel B, Schoergenhofer C, Datler H, Hohensinner P, Basílio J, Petzelbauer P, Assinger A, Schmid JA. Cell type-specific roles of NF-κB linking inflammation and thrombosis. Front Immunol. 2019; 10: 85. 10.3389/fimmu.2019.00085 30778349 PMC6369217 · DOI · Paper page
  22. [34]McCormick SM, Heller NM. Commentary: IL-4 and IL-13 receptors and signaling. Cytokine. 2015; 75: 38– 50. 10.1016/j.cyto.2015.05.023 26187331 PMC4546937 · DOI

Cited by

Sorted by citations

Metadata

Authors
Xie, L., Zhang, N., Zhang, Q., Li, C., Sandhu, A. F., III, G. W., Lin, S., Lv, P., Liu, Y., Wu, Q., & Yu, S.
Venue
Aging
Published
2020-11-09
ISSN
1945-4589
Cited by
48
References
12
Language
en

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review