The DOI matches its Crossref record; 101 of the 101 references it cites were found in a registry or an index.
Oxidative Stress: A Common Factor in Testicular Dysfunction
Abstract
ABSTRACT:Oxidative stress results from the production of oxygen radicals in excess of the antioxidant capacity of the stressed tissue. Many conditions or events associated with male infertility are inducers of oxidative stress. X‐irradiation, for example, or exposure to environmental toxicants and the physical conditions of varicocele and cryptorchidism have been demonstrated to increase testicular oxidative stress, which leads to an increase in germ cell apoptosis and subsequent hypospermatogenesis. Such stress conditions can cause changes in the dynamics of testicular microvascular blood flow, endocrine signaling, and germ cell apoptosis. Testicular oxidative stress appears to be a common feature in much of what underlies male infertility, which suggests that there may be benefits to developing better antioxidant therapies for relevant cases of hypospermatogenesis.
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Cites
Apoptosis in the Pathogenesis and Treatment of Disease
Thompson, C. B. (1995) · Science · doi:10.1126/science.7878464 · Cited by 6,524
Hengartner, M. O. (2000) · Nature · doi:10.1038/35037710 · Cited by 5,779
The Bcl-2 Protein Family: Arbiters of Cell Survival
Adams, J. M., & Cory, S. (1998) · Science · doi:10.1126/science.281.5381.1322 · Cited by 5,332
Nagata, S. (1997) · Cell · doi:10.1016/s0092-8674(00)81874-7 · Cited by 4,938
(1980) · Annals of Internal Medicine · doi:10.7326/0003-4819-92-3-460_4 · Cited by 4,529
Glucocorticoid-induced thymocyte apoptosis is associated with endogenous endonuclease activation
Wyllie, A. H. (1980) · Nature · doi:10.1038/284555a0 · Cited by 4,504
editor-in-chief, A. J. W. (2012) · Elsevier eBooks · Cited by 2,713
Peroxynitrite: biochemistry, pathophysiology and development of therapeutics
Szabó, C., Ischiropoulos, H., & Radi, R. (2007) · Nature Reviews Drug Discovery · doi:10.1038/nrd2222 · Cited by 2,134
Regulation of Mammalian O 2 Homeostasis by Hypoxia-Inducible Factor 1
Semenza, G. L. (1999) · Annual Review of Cell and Developmental Biology · doi:10.1146/annurev.cellbio.15.1.551 · Cited by 1,914 · cited 1×
“Under normoxic conditions in other tissues, HIF-1α is rapidly degraded (Semenza 1999) and is not detected on western blots (Fig.”
BackgroundAlcohol, oxidative stress and free radical damage
Albano, E. (2006) · Proceedings of the Nutrition Society · doi:10.1079/pns2006496 · Cited by 1,215
Cited by
Sartorius, G. A., & Nieschlag, E. (2010) · Human Reproduction Update · doi:10.1093/humupd/dmp027 · Cited by 355
Paul, C., Teng, S., & Saunders, P. T. K. (2009) · Biology of Reproduction · doi:10.1095/biolreprod.108.071779 · Cited by 355
Martinez, C. S., Escobar, A. G., Uranga-Ocio, J. A., Peçanha, F. M., Vassallo, D. V., Exley, C., Miguel, M., & Wiggers, G. A. (2017) · Reproductive Toxicology · doi:10.1016/j.reprotox.2017.08.008 · Cited by 46
Metadata
- Authors
- Turner, T. T., & Lysiak, J. J.
- Venue
- Journal of Andrology
- Publisher
- Wiley
- Published
- 2008-09-10
- Volume
- 29
- Issue
- 5
- Pages
- 488-498
- ISSN
- 0196-3635 (ISSN-L)
- 1939-4640
- OpenAlex
- W2119924772
- Cited by
- 586
- References
- 101
- FWCI
- 22.41
- Type
- journal article
- Language
- en
- Open access
- closed
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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