The DOI matches its Crossref record; 55 of the 55 references it cites were found in a registry or an index.
Mitochondrial ROS promote mitochondrial dysfunction and inflammation in ischemic acute kidney injury by disrupting TFAM-mediated mtDNA maintenance
Open accessAbstract
mtROS can promote renal injury by suppressing TFAM-mediated mtDNA maintenance, resulting in decreased mitochondrial energy metabolism and increased cytokine release. TFAM defects may be a promising target for renal repair after IRI-AKI.
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Cites
Mitochondrial Reactive Oxygen Species (ROS) and ROS-Induced ROS Release
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Oxidized mitochondrial DNA activates the NLRP3 inflammasome during apoptosis
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Self-citationChronic kidney disease after acute kidney injury: a systematic review and meta-analysis
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Mitochondrial DNA stress primes the antiviral innate immune response
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(2009) · New England Journal of Medicine · doi:10.1056/nejmoa0902413 · Cited by 1,473
Mitochondrial energetics in the kidney
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BAK/BAX macropores facilitate mitochondrial herniation and mtDNA efflux during apoptosis
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Apocynin Is Not an Inhibitor of Vascular NADPH Oxidases but an Antioxidant
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Oxidative stress and mitochondrial impairment: Key drivers in neurodegenerative disorders
Wen, P., Sun, Z., Gou, F., Wang, J., Fan, Q., Zhao, D., & Yang, L. (2025) · Ageing Research Reviews · doi:10.1016/j.arr.2025.102667 · Cited by 253
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Dumitru, C. N., Mariana, L., Budacu, C. C., Mitea, G., Radu, M. D., Dumitru, A. O., Lupoae, A., Tatu, A., & Topor, G. (2025) · Dentistry Journal · doi:10.3390/dj13050222 · Cited by 17
Polydopamine-coated bioactive glass for immunomodulation and odontogenesis in pulpitis
Li, J., Wu, J., Zhu, L., Mao, S., Wang, S., Jia, P., & Dong, Y. (2024) · Materials Today Bio · doi:10.1016/j.mtbio.2024.101130 · Cited by 12
Li, Y., Kuang, H., Fan, G., & Yang, X. (2025) · Inflammation · doi:10.1007/s10753-025-02308-2 · Cited by 4
Metadata
- Authors
- Zhao, M., Wang, Y., Li, L., Liu, S., Wang, C., Yuan, Y., Yang, G., Chen, Y., Cheng, J., Lu, Y., & Liu, J.
- Venue
- Theranostics
- Publisher
- Ivyspring International Publisher
- Published
- 2021
- Volume
- 11
- Issue
- 4
- Pages
- 1845-1863
- ISSN
- 1838-7640 (ISSN-L)
- OpenAlex
- W3113188483
- Cited by
- 820
- References
- 55
- FWCI
- 19.57
- Type
- journal article
- Language
- en
- Open access
- gold
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- CoChat. (2026). Citation check: Mitochondrial ROS promote mitochondrial dysfunction and inflammation in ischemic acute kidney injury by disrupting TFAM-mediated mtDNA maintenance. https://app.cochat.ai/papers/10.7150/thno.50905
- The paper
- Zhao, M., Wang, Y., Li, L., Liu, S., Wang, C., Yuan, Y., Yang, G., Chen, Y., Cheng, J., Lu, Y., & Liu, J. (2021). Mitochondrial ROS promote mitochondrial dysfunction and inflammation in ischemic acute kidney injury by disrupting TFAM-mediated mtDNA maintenance. Theranostics. https://doi.org/10.7150/thno.50905
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
