Finch, Caleb E.

Complement activation in very early Alzheimer disease

Zanjani, H., Finch, C. E., Kemper, C., Atkinson, J., McKeel, D., Morris, J. C., & Price, J. L. (2005) · Alzheimer Disease & Associated Disorders

Alzheimer's disease research and treatmentsComplement system in diseases

Cited by 115

Inflammation and Infection Do Not Promote Arterial Aging and Cardiovascular Disease Risk Factors among Lean Horticulturalists

Gurven, M., Kaplan, H., Winking, J., Eid Rodriguez, D., Vasunilashorn, S., Kim, J. K., Finch, C., & Crimmins, E. (2009) · PLoS ONE

Indigenous Health and EducationCardiovascular Health and Disease Prevention

Cited by 107

Blood lipids, infection, and inflammatory markers in the Tsimane of Bolivia

Vasunilashorn, S., Crimmins, E. M., Kim, J. K., Winking, J., Gurven, M., Kaplan, H., & Finch, C. E. (2010) · American Journal of Human Biology

Indigenous Health and EducationLipoproteins and Cardiovascular Health

Cited by 95

Macrosialin increases during normal brain aging are attenuated by caloric restriction

Wong, A. M., Patel, N. V., Patel, N. K., Wei, M., Morgan, T. E., de Beer, M. C., de Villiers, W. J. S., & Finch, C. E. (2005) · Neuroscience Letters

Neuroinflammation and Neurodegeneration MechanismsGenetics, Aging, and Longevity in Model Organisms

Cited by 85

The Oxygen Paradox, the French Paradox, and age-related diseases

Davies, J. M. S., Cillard, J., Friguet, B., Cadenas, E., Cadet, J., Cayce, R., Fishmann, A., Liao, D., Bulteau, A.-L., Derbré, F., et al. (2017) · GeroScience

Antioxidant Activity and Oxidative StressPhytochemicals and Antioxidant Activities

Cited by 82

Commentary: is Alzheimer's disease uniquely human?

Finch, C. E., & Austad, S. N. (2015) · Neurobiology of Aging

Alzheimer's disease research and treatmentsDementia and Cognitive Impairment Research

Cited by 72

Apolipoprotein J (clusterin) activates rodent microglia in vivo and in vitro

Xie, Z., Harris‐White, M. E., Wals, P. A., Frautschy, S. A., Finch, C. E., & Morgan, T. E. (2005) · Journal of Neurochemistry

Neuroinflammation and Neurodegeneration MechanismsClusterin in disease pathology

Cited by 53

Life-long dietary restriction attenuates age-related increases in hippocampal glial fibrillary acidic protein mRNA

Major, D. E., Kesslak, J. P., Cotman, C. W., Finch, C. E., & Day, J. R. (1997) · Neurobiology of Aging

Genetics, Aging, and Longevity in Model OrganismsNeuroinflammation and Neurodegeneration Mechanisms

Cited by 52

Food restriction delays the age-related increase in GFAP mRNA in rat hypothalamus

Nichols, N. R., Finch, C. E., & Nelson, J. F. (1995) · Neurobiology of Aging

Genetics, Aging, and Longevity in Model OrganismsNeuroinflammation and Neurodegeneration Mechanisms

Cited by 46

Comment on “The plateau of human mortality: Demography of longevity pioneers”

Beltrán-Sánchez, H., Austad, S. N., & Finch, C. E. (2018) · Science

Insurance, Mortality, Demography, Risk ManagementGenetics, Aging, and Longevity in Model Organisms

Cited by 42

Exposure to Nanoscale Particulate Matter from Gestation to Adulthood Impairs Metabolic Homeostasis in Mice

Woodward, N. C., Crow, A. L., Zhang, Y., Epstein, S., Hartiala, J., Johnson, R., Kocalis, H., Saffari, A., Sankaranarayanan, I., Akbari, O., et al. (2019) · Scientific Reports

Air Quality and Health ImpactsAdipokines, Inflammation, and Metabolic Diseases

Cited by 32

Cell resilience in species life spans: a link to inflammation?

Finch, C. E., Morgan, T. E., Longo, V. D., & De Magalhaes, J. P. (2010) · Aging Cell

Genetics, Aging, and Longevity in Model OrganismsTelomeres, Telomerase, and Senescence

Cited by 29