The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.
Tendon and ligament: Development, repair and disease
Abstract
Tendons and ligaments (T/L) are very similar fibrous tissues that respectively connect muscle to bone and bone to bone. They are comprised of fibroblasts that produce large amounts of extra‐cellular matrix, resulting in a dense and hypocellular structure. The complex molecular organization of T/L, together with high water content, are responsible for their viscoelastic properties, hence insuring their mechanical function. We will first review recent work on tendon embryology and discuss ligament formation, which has been less documented. We will next summarize our current knowledge of T/L molecular architecture, alterations of which are a major cause for disease. We will finally focus on T/L repair after injury and on genetic diseases responsible for T/L defects. Birth Defects Research (Part C) 75:226–236, 2005. © 2005 Wiley‐Liss, Inc.
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Cites
Collagen fibrillogenesis in vitro: interaction of types I and V collagen regulates fibril diameter
Birk, D. E., Fitch, J. M., Babiarz, J. P., Doane, K. J., & Linsenmayer, T. F. (1990) · Journal of Cell Science · doi:10.1242/jcs.95.4.649 · Cited by 596
Colige, A., Sieron, A. L., Li, S.-W., Schwarze, U., Petty, E., Wertelecki, W., Wilcox, W., Krakow, D., Cohn, D. H., Reardon, W., et al. (1999) · The American Journal of Human Genetics · doi:10.1086/302504 · Cited by 370
Tenascin-X deficiency is associated with Ehlers-Danlos syndrome
Burch, G. H., Gong, Y., Liu, W., Dettman, R. W., Curry, C. J., Smith, L., Miller, W. L., & Bristow, J. (1997) · Nature Genetics · doi:10.1038/ng0997-104 · Cited by 366
Haploinsufficiency of TNXB Is Associated with Hypermobility Type of Ehlers-Danlos Syndrome
Zweers, M. C., Bristow, J., Steijlen, P. M., Dean, W. B., Hamel, B. C., Otero, M., Kucharekova, M., Boezeman, J. B., & Schalkwijk, J. (2003) · The American Journal of Human Genetics · doi:10.1086/376564 · Cited by 242
The Ehlers-Danlos syndrome: on beyond collagens
Mao, J.-R., & Bristow, J. (2001) · Journal of Clinical Investigation · doi:10.1172/jci12881 · Cited by 233
Classical Ehlers-Danlos Syndrome Caused by a Mutation in Type I Collagen
Nuytinck, L., Freund, M., Lagae, L., Pierard, G. E., Hermanns-Le, T., & De Paepe, A. (2000) · The American Journal of Human Genetics · doi:10.1086/302859 · Cited by 169
Cited by
Extracellular Matrix Assembly: A Multiscale Deconstruction
Mouw, J. K., Ou, G., & Weaver, V. M. (2014) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm3902 · Cited by 1,534
Pathogenesis of tendinopathies: inflammation or degeneration?
Abate, M., Gravare-Silbernagel, K., Siljeholm, C., Di Iorio, A., De Amicis, D., Salini, V., Werner, S., & Paganelli, R. (2009) · Arthritis Research & Therapy · doi:10.1186/ar2723 · Cited by 571
Tendon and ligament regeneration and repair: Clinical relevance and developmental paradigm
Yang, G., Rothrauff, B. B., & Tuan, R. S. (2013) · Birth Defects Research Part C: Embryo Today: Reviews · doi:10.1002/bdrc.21041 · Cited by 469
Pryce, B. A., Watson, S. S., Murchison, N. D., Staverosky, J. A., Dünker, N., & Schweitzer, R. (2009) · Development · doi:10.1242/dev.027342 · Cited by 420
Conversion of Mechanical Force into TGF-β-Mediated Biochemical Signals
Maeda, T., Sakabe, T., Sunaga, A., Sakai, K., Rivera, A. L., Keene, D. R., Sasaki, T., Stavnezer, E., Iannotti, J., Schweitzer, R., et al. (2011) · Current Biology · doi:10.1016/j.cub.2011.04.007 · Cited by 366
Metadata
- Authors
- Tozer, S., & Duprez, D.
- Venue
- Birth Defects Research Part C: Embryo Today: Reviews
- Publisher
- Wiley
- Published
- 2005-09
- Volume
- 75
- Issue
- 3
- Pages
- 226-236
- ISSN
- 1542-975X (ISSN-L)
- 1542-9768
- OpenAlex
- W2045545457
- Cited by
- 187
- References
- 6
- FWCI
- 3.81
- Type
- journal article
- Language
- en
- Open access
- closed
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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