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Cited by 297

The DOI matches its Crossref record; 8 of the 8 references it cites were found in a registry or an index.

Critical functions of priming and lysosomal damage for NLRP3 activation

Open access
Hornung, Veit, Latz, Eicke·2010·European Journal of Immunology

Abstract

Inflammasomes are cytosolic multi‐protein complexes that form in response to infectious or injurious challenges. Inflammasomes control the activity of caspase‐1, which is essential for the maturation and release of IL‐1β family cytokines. The NLRP1, IPAF and AIM2 inflammasomes recognize specific substances, while the NLRP3 inflammasome responds to many structurally and chemically diverse triggers. Here, we discuss the critical roles of priming and lysosomal damage in NLRP3 inflammasome activation.

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Crossref · 10.1002/eji.200940185

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Metadata

Authors
Hornung, V., & Latz, E.
Venue
European Journal of Immunology
Publisher
Wiley
Published
2010-03
Volume
40
Issue
3
Pages
620-623
ISSN
  • 0014-2980 (ISSN-L)
  • 1521-4141
OpenAlex
W2063894714
Cited by
297
References
8
FWCI
7.56
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Critical functions of priming and lysosomal damage for NLRP3 activation. https://app.cochat.ai/papers/10.1002/eji.200940185
The paper
Hornung, V., & Latz, E. (2010). Critical functions of priming and lysosomal damage for NLRP3 activation. European Journal of Immunology. https://doi.org/10.1002/eji.200940185

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.