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Activating mutations in FGFR3 and HRAS reveal a shared genetic origin for congenital disorders and testicular tumors

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Crystal Structure of Human RhoA in a Dominantly Active Form Complexed with a GTP Analogue

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Rac1 GTPase activates the WAVE regulatory complex through two distinct binding sites

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Germline KRAS mutations cause aberrant biochemical and physical properties leading to developmental disorders

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A RAS Renaissance: Emerging Targeted Therapies for KRAS-Mutated Non–Small Cell Lung Cancer

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RAS variant signalling

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Ras-independent activation of Ral by a Ca2+-dependent pathway

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RAS Mutations and Oncogenesis: Not all RAS Mutations are Created Equally

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