Monoamine oxidase inactivation: from pathophysiology to therapeutics

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Amphetamine, 3,4-Methylenedioxymethamphetamine, Lysergic Acid Diethylamide, and Metabolites of the Catecholamine Neurotransmitters Are Agonists of a Rat Trace Amine Receptor

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Structure of human monoamine oxidase B, a drug target for the treatment of neurological disorders

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Major Depression and Activation of The Inflammatory Response System

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Some puzzling pharmacological effects of monoamine oxidase inhibitors.

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Homocysteine, folate, methylation, and monoamine metabolism in depression

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Lack of Barrels in the Somatosensory Cortex of Monoamine Oxidase A–Deficient Mice: Role of a Serotonin Excess during the Critical Period

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Major depressive disorder is accompanied with oxidative stress: short-term antidepressant treatment does not alter oxidative-antioxidative systems

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Mammalian central nervous system trace amines. Pharmacologic amphetamines, physiologic neuromodulators

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Increased monoamine oxidase b activity in plaque-associated astrocytes of Alzheimer brains revealed by quantitative enzyme radioautography

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Indicators of immune activation in major depression

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Localization of distinct monoamine oxidase a and monoamine oxidase b cell populations in human brainstem

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Increased Serum Levels of 8-Hydroxy-2′-Deoxyguanosine in Clinical Depression

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MAO-B Elevation in Mouse Brain Astrocytes Results in Parkinson's Pathology

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A detailed analysis of hydrogen peroxide-induced cell death in primary neuronal culture

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Increased stress response and β–phenylethylamine in MAOB–deficient mice

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In vivo Inhibition of Liver and Brain Monoamine Oxidase by 1-Isonicotinyl-2-Isopropyl Hydrazine.

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The Trace Amine 1 receptor knockout mouse: an animal model with relevance to schizophrenia

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Substrate- and inhibitor-related characteristics of human platelet monoamine oxidase

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The Metabolism of Tyramine by Monoamine Oxidase A/B Causes Oxidative Damage to Mitochondrial DNA

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Human monoamine oxidase A and B genes map to xp11.23 and are deleted in a patient with norrie disease

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Structure of Rat Monoamine Oxidase A and Its Specific Recognitions for Substrates and Inhibitors

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Antioxidant Enzyme Activities and Malondialdehyde Levels in Patients with Obsessive-Compulsive Disorder

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Tissue Distribution of Human Monoamine Oxidase A and B mRNA

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The new generation of monoamine oxidase inhibitors

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Rasagiline: Neurodegeneration, neuroprotection, and mitochondrial permeability transition

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Substrate and Inhibitor Specificities for Human Monoamine Oxidase A and B Are Influenced by a Single Amino Acid

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Species differences in the deamination of dopamine and other substrates for monoamine oxidase in brain

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Glucocorticoid and Androgen Activation of Monoamine Oxidase A Is Regulated Differently by R1 and Sp1

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Selegiline for Alzheimer's disease

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Monoamine oxidase A and repressor R1 are involved in apoptotic signaling pathway

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Selective enhancement of emotional, but not motor, learning in monoamine oxidase A-deficient mice

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A Spontaneous Point Mutation Produces Monoamine Oxidase A/B Knock-out Mice with Greatly Elevated Monoamines and Anxiety-like Behavior*♦

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Antioxidant Enzyme and Malondialdehyde Levels in Patients with Panic Disorder

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Novel monoamine oxidase A knock out mice with human-like spontaneous mutation

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Use of dopamine-beta-hydroxylase-deficient mice to determine the role of norepinephrine in the mechanism of action of antidepressant drugs.

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The relationship between potency of oxidative stress and severity of depression

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Course and treatment of atypical depression.

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β-Phenylethylamine Alters Monoamine Transporter Function via Trace Amine-Associated Receptor 1: Implication for Modulatory Roles of Trace Amines in Brain

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Use of Dopamine-β-hydroxylase-Deficient Mice to Determine the Role of Norepinephrine in the Mechanism of Action of Antidepressant Drugs

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L-Deprenil, A Selective Monoamine Oxidase Type B Inhibitor, in the Treatment of Depression: A Double Blind Evaluation

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Ketanserin and tetrabenazine abolish aggression in mice lacking monoamine oxidase A

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R1, a Novel Repressor of the Human Monoamine Oxidase A

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Forebrain-specific Expression of Monoamine Oxidase A Reduces Neurotransmitter Levels, Restores the Brain Structure, and Rescues Aggressive Behavior in Monoamine Oxidase A-deficient Mice

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MAO A knockout attenuates adrenocortical response to various kinds of stress

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Cerebral cortical blood flow maps are reorganized in MAOB-deficient mice

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