The DOI matches its Crossref record; 8 of the 8 references it cites were found in a registry or an index.
Molecular events associated with reactive oxygen species and cell cycle progression in mammalian cells
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Cites
[1] Role of free radicals and catalytic metal ions in human disease: An overview
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Oxidative DNA damage: mechanisms, mutation, and disease
Cooke, M. S., Evans, M. D., Dizdaroglu, M., & Lunec, J. (2003) · The FASEB Journal · doi:10.1096/fj.02-0752rev · Cited by 3,245
New functional activities for the p21 family of CDK inhibitors.
LaBaer, J., Garrett, M. D., Stevenson, L. F., Slingerland, J. M., Sandhu, C., Chou, H. S., Fattaey, A., & Harlow, E. (1997) · Genes & Development · doi:10.1101/gad.11.7.847 · Cited by 1,402
How Proteolysis Drives the Cell Cycle
King, R. W., Deshaies, R. J., Peters, J.-M., & Kirschner, M. W. (1996) · Science · doi:10.1126/science.274.5293.1652 · Cited by 1,273
Protein kinase C signaling and oxidative stress
Gopalakrishna, R., & Jaken, S. (2000) · Free Radical Biology and Medicine · doi:10.1016/s0891-5849(00)00221-5 · Cited by 701 · cited 5×
“Various studies have established that oxygen free radicals can cause the phosphorylation and activation of numerous signaling proteins (Finkel and Holbrook, 2000), including Receptor Tyrosine Kinases (RTK) (Gamou and Shimizu, 1995; González-Rubio et al., 1996), PKC (Gopalakrishna and Jaken, 2000), PLCg1 (Schieven et…”
BackgroundInfluentialComparative resistance of the 20S and 26S proteasome to oxidative stress
REINHECKEL, T., SITTE, N., ULLRICH, O., KUCKELKORN, U., DAVIES, K. J. A., & GRUNE, T. (1998) · Biochemical Journal · doi:10.1042/bj3350637 · Cited by 447
Inhibition of p21‐mediated ROS accumulation can rescue p21‐induced senescence
Macip, S., Igarashi, M., Fang, L., Chen, A., Pan, Z., Lee, S. W., & Aaronson, S. A. (2002) · The EMBO Journal · doi:10.1093/emboj/21.9.2180 · Cited by 374
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Cited by
Targeting cancer cells by ROS-mediated mechanisms: a radical therapeutic approach?
Trachootham, D., Alexandre, J., & Huang, P. (2009) · Nature Reviews Drug Discovery · doi:10.1038/nrd2803 · Cited by 5,452
Gupta, S. C., Hevia, D., Patchva, S., Park, B., Koh, W., & Aggarwal, B. B. (2012) · Antioxidants & Redox Signaling · doi:10.1089/ars.2011.4414 · Cited by 732
Reactive Oxygen Species-Related Nanoparticle Toxicity in the Biomedical Field
Yu, Z., Li, Q., Wang, J., Yu, Y., Wang, Y., Zhou, Q., & Li, P. (2020) · Nanoscale Research Letters · doi:10.1186/s11671-020-03344-7 · Cited by 709
You, C., Han, C., Wang, X., Zheng, Y., Li, Q., Hu, X., & Sun, H. (2012) · Molecular Biology Reports · doi:10.1007/s11033-012-1792-8 · Cited by 447
Zarbock, A., Schmidt, C., Van Aken, H., Wempe, C., Martens, S., Zahn, P. K., Wolf, B., Goebel, U., Schwer, C. I., Rosenberger, P., et al. (2015) · JAMA · doi:10.1001/jama.2015.4189 · Cited by 422
Metadata
- Authors
- Boonstra, J., & Post, J. A.
- Venue
- Gene
- Publisher
- Elsevier BV
- Published
- 2004-08
- Volume
- 337
- Pages
- 1-13
- ISSN
- 0378-1119 (ISSN-L)
- OpenAlex
- W2002746961
- Cited by
- 737
- References
- 8
- FWCI
- 8.64
- Type
- journal article
- Language
- en
- Open access
- green
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Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
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- CoChat. (2026). Citation check: Molecular events associated with reactive oxygen species and cell cycle progression in mammalian cells. https://app.cochat.ai/papers/10.1016/j.gene.2004.04.032
- The paper
- Boonstra, J., & Post, J. A. (2004). Molecular events associated with reactive oxygen species and cell cycle progression in mammalian cells. Gene. https://doi.org/10.1016/j.gene.2004.04.032
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
