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Cited by 269

The DOI matches its Crossref record; 34 of the 34 references it cites were found in a registry or an index.

Contents, preface, executive summary, chapters 1 and 2

Valentin, J.·2005·Annals of the ICRP
DOI 10.1016/j.icrp.2005.11.002
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Abstract

ABSTRACT GUEST EDITORIAL PREFACE EXECUTIVE SUMMARY INTRODUCTION EPIDEMIOLOGICAL CONSIDERATION Introduction Inferences based on acute doses in the moderate-to-high dose range Estimation of risk at low doses and low dose rates Thresholds vs. the linear, no-threshold theory Conclusion: Implications for low-dose cancer risk LOW-DOSE RISK BIOLOGY Introduction Damage caused by radiation Damage Response Pathways Fidelity of DSB repair Impact of defects in DNA repair, checkpoint control and apoptosis Conclusions CELLULAR CONSEQUENCES OF RADIATION-INDUCED DAMAGE Radiation- induced Chromosome Aberrations Radiation-Induced Somatic Cell Mutations Adaptive Response, Genomic Instability and Bystander Effect Conclusions: Implications for Risk Assessment CARCINOGENIC EFFECTS OF IONIZING RADIATION Mechanisms of radiation-induced cancer Tissue Modifying Factors Radiation-induced Cancer in Animals Life Shortening Summary Conclusions: Implications for radiation-related cancer at low doses QUANTITATIVE UNCERTAINTY ANALYSIS Overview Sources of uncertainty Allowing for the uncertain possibility of a threshold Conclusions CONCLUSIONS REFERENCES

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Metadata

Authors
Valentin, J.
Venue
Annals of the ICRP
Publisher
SAGE Publications
Published
2005-12
Volume
35
Issue
4
Pages
1-39
ISSN
  • 0146-6453 (ISSN-L)
  • 1872-969X
DOI
10.1016/j.icrp.2005.11.002
OpenAlex
W1974343556
Cited by
269
References
34
Type
paratext
Language
en
Open access
closed

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CoChat. (2026). Citation check: Contents, preface, executive summary, chapters 1 and 2. https://app.cochat.ai/papers/10.1016/j.icrp.2005.11.002
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Valentin, J. (2005). Contents, preface, executive summary, chapters 1 and 2. Annals of the ICRP. https://doi.org/10.1016/j.icrp.2005.11.002

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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