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Cited by 276

The DOI matches its Crossref record; 81 of the 81 references it cites were found in a registry or an index.

SOCS1 and SOCS3 in the control of CNS immunity

Open access
Baker, Brandi J., Akhtar, Lisa Nowoslawski, Benveniste, Etty N.·2009·Trends in Immunology

Abstract

In the decade following their initial discovery, the suppressor of cytokine signaling (SOCS) proteins have been studied for their potential use as immunomodulators in disease. SOCS proteins, especially SOCS1 and SOCS3, are expressed by immune cells and cells of the central nervous system (CNS) and have the potential to impact immune processes within the CNS, including inflammatory cytokine and chemokine production, activation of microglia, macrophages and astrocytes, immune cell infiltration and autoimmunity. We describe CNS-relevant in vitro and in vivo studies that have examined the function of SOCS1 or SOCS3 under various neuroinflammatory or neuropathological conditions, including exposure of CNS cells to inflammatory cytokines or bacterial infection, demyelinating insults, stroke, spinal cord injury, multiple sclerosis and glioblastoma multiforme.

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Crossref · 10.1016/j.it.2009.07.001

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Metadata

Authors
Baker, B. J., Akhtar, L. N., & Benveniste, E. N.
Venue
Trends in Immunology
Publisher
Elsevier BV
Published
2009-08
Volume
30
Issue
8
Pages
392-400
ISSN
1471-4906 (ISSN-L)
OpenAlex
W2003790210
Cited by
276
References
81
FWCI
0.91
Type
journal article
Language
en
Open access
green

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CoChat. (2026). Citation check: SOCS1 and SOCS3 in the control of CNS immunity. https://app.cochat.ai/papers/10.1016/j.it.2009.07.001
The paper
Baker, B. J., Akhtar, L. N., & Benveniste, E. N. (2009). SOCS1 and SOCS3 in the control of CNS immunity. Trends in Immunology. https://doi.org/10.1016/j.it.2009.07.001

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.