Cited by 113

The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

PCSK9 Modulates the Secretion But Not the Cellular Uptake of Lipoprotein(a) Ex Vivo

Villard, Elise F., Thedrez, Aurélie, Blankenstein, Jorg, Croyal, Mikaël, Tran, Thi-Thu-Trang, Poirier, Bruno, Le Bail, Jean-Christophe, Illiano, Stéphane, Nobécourt, Estelle, Krempf, Michel, Blom, Dirk J., Marais, A. David, Janiak, Philip, Muslin, Anthony J., Guillot, Etienne, Lambert, Gilles·2016·JACC: Basic to Translational Science

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Crossref · 10.1016/j.jacbts.2016.06.006

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Research works printed but not linked: 20

  1. Robinson, 2015, Efficacy and safety of alirocumab in reducing lipids and cardiovascular events, N Engl J Med, 372, 1489, 10.1056/NEJMoa1501031 · DOI · Paper page
  2. Ballantyne, 2015, Results of bococizumab, a monoclonal antibody against proprotein convertase subtilisin/kexin type 9, from a randomized, placebo-controlled, dose-ranging study in statin-treated subjects with hypercholesterolemia, Am J Cardiol, 115, 1212, 10.1016/j.amjcard.2015.02.006 · DOI
  3. Marais, 2015, PCSK9 inhibition in LDL cholesterol reduction: genetics and therapeutic implications of very low plasma lipoprotein levels, Pharmacol Ther, 145, 58, 10.1016/j.pharmthera.2014.07.004 · DOI
  4. Lambert, 2014, Normalization of low-density lipoprotein receptor expression in receptor defective homozygous familial hypercholesterolemia by inhibition of PCSK9 with alirocumab, J Am Coll Cardiol, 64, 2299, 10.1016/j.jacc.2014.07.995 · DOI
  5. Raal, 2014, Reduction in lipoprotein(a) with PCSK9 monoclonal antibody evolocumab (AMG 145): a pooled analysis of more than 1,300 patients in 4 phase II trials, J Am Coll Cardiol, 63, 1278
  6. Lamon-Fava, 2014, Lipoprotein(a) metabolism, Curr Opin Lipidol, 25, 189, 10.1097/MOL.0000000000000070 · DOI
  7. Alonso, 2014, Lipoprotein(a) levels in hypercholesterolemia: an important predictor of cardiovascular disease independent of the type of LDL receptor mutation, J Am Coll Cardiol, 63, 1982, 10.1016/j.jacc.2014.01.063 · DOI · Paper page
  8. Erqou, 2009, Lipoprotein(a) concentration and the risk of coronary heart disease, stroke, and nonvascular mortality, JAMA, 302, 412, 10.1001/jama.2009.1063 · DOI · Paper page
  9. Rader, 1994, The inverse association of plasma lipoprotein(a) concentrations with apolipoprotein(a) isoform size is not due to differences in Lp(a) catabolism but to differences in production rate, J Clin Invest, 93, 2758, 10.1172/JCI117292 · DOI
  10. Tsimikas, 2012, Lipoprotein(a) as a potential causal genetic risk factor of cardiovascular disease: a rationale for increased efforts to understand its pathophysiology and develop targeted therapies, J Am Coll Cardiol, 60, 716, 10.1016/j.jacc.2012.04.038 · DOI
  11. Romagnuolo, 2015, Lipoprotein(a) catabolism is regulated by proprotein convertase subtilisin/kexin type 9 through the low density lipoprotein receptor, J Biol Chem, 290, 11649, 10.1074/jbc.M114.611988 · DOI · Paper page
  12. Lambert, 2014, Elevated plasma PCSK9 level is equally detrimental for patients with nonfamilial hypercholesterolemia and heterozygous familial hypercholesterolemia, irrespective of low-density lipoprotein receptor defects, J Am Coll Cardiol, 63, 2365, 10.1016/j.jacc.2014.02.538 · DOI
  13. Croyal, 2015, Effects of extended-release nicotinic acid on apolipoprotein (a) kinetics in hypertriglyceridemic patients, Arterioscler Thromb Vasc Biol, 35, 2042, 10.1161/ATVBAHA.115.305835 · DOI
  14. Etxebarria, 2015, Functional characterization and classification of frequent low-density lipoprotein receptor variants, Hum Mutat, 36, 129, 10.1002/humu.22721 · DOI · Paper page
  15. Reblin, 1997, Cellular uptake of lipoprotein[a] by mouse embryonic fibroblasts via the LDL receptor and the LDL receptor-related protein, J Lipid Res, 38, 2103, 10.1016/S0022-2275(20)37140-6 · DOI · Paper page
  16. Niemeier, 1999, Identification of megalin/gp330 as a receptor for lipoprotein(a) in vitro, Arterioscler Thromb Vasc Biol, 19, 552, 10.1161/01.ATV.19.3.552 · DOI
  17. Stein, 2013, Effect of the proprotein convertase subtilisin/kexin 9 monoclonal antibody, AMG 145, in homozygous familial hypercholesterolemia, Circulation, 128, 2113, 10.1161/CIRCULATIONAHA.113.004678 · DOI · Paper page
  18. Rader, 1995, The low density lipoprotein receptor is not required for normal catabolism of Lp(a) in humans, J Clin Invest, 95, 1403, 10.1172/JCI117794 · DOI · Paper page
  19. Tavori, 2015, On the function and homeostasis of PCSK9: reciprocal interaction with LDLR and additional lipid effects, Atherosclerosis, 238, 264, 10.1016/j.atherosclerosis.2014.12.017 · DOI · Paper page
  20. Tavori, 2013, Loss of plasma proprotein convertase subtilisin/kexin 9 (PCSK9) after lipoprotein apheresis, Circ Res, 113, 1290, 10.1161/CIRCRESAHA.113.302655 · DOI

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Metadata

Authors
Villard, E. F., Thedrez, A., Blankenstein, J., Croyal, M., Tran, T.-T.-T., Poirier, B., Le Bail, J.-C., Illiano, S., Nobécourt, E., Krempf, M., Blom, D. J., Marais, A. D., Janiak, P., Muslin, A. J., Guillot, E., & Lambert, G.
Venue
JACC: Basic to Translational Science
Publisher
Elsevier BV
Published
2016-10
Volume
1
Issue
6
Pages
419-427
ISSN
2452-302X
Cited by
113
References
3
Language
en
Funders
  • Amgen
  • Pfizer

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