The DOI matches its Crossref record; 9 of the 9 references it cites were found in a registry or an index.
Perinatal environment and its influences on metabolic programming of offspring
Open accessAbstract
The intrauterine environment supports the development and health of offspring. Perturbations to this environment can have detrimental effects on the fetus that have persistent pathological consequences through adolescence and adulthood. The developmental origins of health and disease concept, also known as the “Barker Hypothesis”, has been put forth to describe the increased incidence of chronic disease such as cardiovascular disease and diabetes in humans and animals exposed to a less than ideal intrauterine environment. Maternal infection, poor or excess nutrition, and stressful events can negatively influence development of different cell types, tissues and organ systems ultimately predisposing the organism to pathological conditions. Although there are a variety of conditions associated to exposure to altered intrauterine environments, the focus of this review will be on the consequences of stress and high fat diet during the pre- and perinatal periods and associated outcomes related to obesity and other metabolic conditions. We further discuss possible neuroendocrine and epigenetic mechanisms responsible for metabolic programming of offspring.
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Cites
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Cited by
Physiological adaptations to weight loss and factors favouring weight regain
Greenway, F. L. (2015) · International Journal of Obesity · doi:10.1038/ijo.2015.59 · Cited by 431
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Behavioral effects of chronic adolescent stress are sustained and sexually dimorphic
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Metadata
- Authors
- Tamashiro, K. L. K., & Moran, T. H.
- Venue
- Physiology & Behavior
- Publisher
- Elsevier BV
- Published
- 2010-07
- Volume
- 100
- Issue
- 5
- Pages
- 560-566
- ISSN
- 0031-9384 (ISSN-L)
- OpenAlex
- W2041778509
- Cited by
- 195
- References
- 9
- FWCI
- 17.38
- Type
- journal article
- Language
- en
- Open access
- green
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
