The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.
Idax and Rinf facilitate expression of Tet enzymes to promote neural and suppress trophectodermal programs during differentiation of embryonic stem cells
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“TET2 is also unique among TET enzymes in that it is not covalently linked to the zinc-finger CXXC domain protein CXXC4 or CXXC512–14”
From RNA m5C oxidation by TET2 regulates chromatin state and leukaemogenesis (2024) · cites this paper as [13]
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Cites
TET1 and hydroxymethylcytosine in transcription and DNA methylation fidelity
Williams, K., Christensen, J., Pedersen, M. T., Johansen, J. V., Cloos, P. A. C., Rappsilber, J., & Helin, K. (2011) · Nature · doi:10.1038/nature10066 · Cited by 1,021
TETonic shift: biological roles of TET proteins in DNA demethylation and transcription
Pastor, W. A., Aravind, L., & Rao, A. (2013) · Nature Reviews Molecular Cell Biology · doi:10.1038/nrm3589 · Cited by 897
Loss of Tet Enzymes Compromises Proper Differentiation of Embryonic Stem Cells
Dawlaty, M. M., Breiling, A., Le, T., Barrasa, M. I., Raddatz, G., Gao, Q., Powell, B. E., Cheng, A. W., Faull, K. F., Lyko, F., et al. (2014) · Developmental Cell · doi:10.1016/j.devcel.2014.03.003 · Cited by 338
Self-citation
Research works printed but not linked: 25
- Chrysanthou, 2018, A critical role of TET1/2 proteins in cell-cycle progression of trophoblast stem cells, Stem Cell Rep., 10, 1355, 10.1016/j.stemcr.2018.02.014 · DOI
- Chrysanthou, 2022, The DNA dioxygenase Tet1 regulates H3K27 modification and embryonic stem cell biology independent of its catalytic activity, Nucleic Acids Res., 10.1093/nar/gkac089 · DOI
- Dawlaty, 2011, Tet1 is dispensable for maintaining pluripotency and its loss is compatible with embryonic and postnatal development, Cell Stem Cell, 9, 166, 10.1016/j.stem.2011.07.010 · DOI · Paper page
- Dawlaty, 2013, Combined deficiency of Tet1 and Tet2 causes epigenetic abnormalities but is compatible with postnatal development, Dev. Cell, 24, 310, 10.1016/j.devcel.2012.12.015 · DOI
- Dixon, 2021, QSER1 protects DNA methylation valleys from de novo methylation, Science, 372, 10.1126/science.abd0875 · DOI
- Dobin, 2013, STAR: ultrafast universal RNA-seq aligner, Bioinformatics, 29, 15, 10.1093/bioinformatics/bts635 · DOI · Paper page
- Jaenisch, 2008, Stem cells, the molecular circuitry of pluripotency and nuclear reprogramming, Cell, 132, 567, 10.1016/j.cell.2008.01.015 · DOI
- Kim, 2016, CXXC5 plays a role as a transcription activator for myelin genes on oligodendrocyte differentiation, Glia, 64, 350, 10.1002/glia.22932 · DOI
- Kim, 2014, CXXC5 is a transcriptional activator of Flk-1 and mediates bone morphogenic protein-induced endothelial cell differentiation and vessel formation, FASEB J., 28, 615, 10.1096/fj.13-236216 · DOI
- Kim, 2010, A novel Wilms tumor 1 (WT1) target gene negatively regulates the WNT signaling pathway, J. Biol. Chem., 285, 14585, 10.1074/jbc.M109.094334 · DOI
- Knappskog, 2011, RINF (CXXC5) is overexpressed in solid tumors and is an unfavorable prognostic factor in breast cancer, Ann. Oncol., 22, 2208, 10.1093/annonc/mdq737 · DOI
- Ko, 2013, Modulation of TET2 expression and 5-methylcytosine oxidation by the CXXC domain protein IDAX, Nature, 497, 122, 10.1038/nature12052 · DOI · Paper page
- Koh, 2011, Tet1 and Tet2 regulate 5-hydroxymethylcytosine production and cell lineage specification in mouse embryonic stem cells, Cell Stem Cell, 8, 200, 10.1016/j.stem.2011.01.008 · DOI · Paper page
- Lee, 2015, The Dishevelled-binding protein CXXC5 negatively regulates cutaneous wound healing, J. Exp. Med., 212, 1061, 10.1084/jem.20141601 · DOI
- Li, 2014, CXXC5 regulates differentiation of C2C12 myoblasts into myocytes, J. Muscle Res. Cell Motil., 35, 259, 10.1007/s10974-014-9400-2 · DOI
- Liao, 2014, featureCounts: an efficient general purpose program for assigning sequence reads to genomic features, Bioinformatics, 30, 923, 10.1093/bioinformatics/btt656 · DOI · Paper page
- Liu, N., Wang, M., Deng, W., Schmidt, C.S., Qin, W., Leonhardt, H., and Spada, F. (2013). Intrinsic and extrinsic connections of Tet3 dioxygenase with CXXC zinc finger modules. PLoS One 8, e62755. · DOI
- Ma, 2017, Epigenetic regulator CXXC5 recruits DNA demethylase Tet2 to regulate TLR7/9-elicited IFN response in pDCs, J. Exp. Med., 214, 1471, 10.1084/jem.20161149 · DOI · Paper page
- Pendino, 2009, Functional involvement of RINF, retinoid-inducible nuclear factor (CXXC5), in normal and tumoral human myelopoiesis, Blood, 113, 3172, 10.1182/blood-2008-07-170035 · DOI
- Rasmussen, 2019, TET2 binding to enhancers facilitates transcription factor recruitment in hematopoietic cells, Genome Res., 29, 564, 10.1101/gr.239277.118 · DOI · Paper page
- Ravichandran, M., Lei, R., Tang, Q., Zhao, Y., Lee, J., Ma, L., Chrysanthou, S., Lorton, B.M., Cvekl, A., Shechter, D., et al. (2019). Rinf Regulates Pluripotency Network Genes and Tet Enzymes in Embryonic Stem Cells. Cell Rep 28, 1993-2003 e1995. · DOI · Paper page
- Robinson, 2010, edgeR: a Bioconductor package for differential expression analysis of digital gene expression data, Bioinformatics, 26, 139, 10.1093/bioinformatics/btp616 · DOI · Paper page
- Tahiliani, 2009, Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1, Science, 324, 930, 10.1126/science.1170116 · DOI · Paper page
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- Wu, 2011, Dual functions of Tet1 in transcriptional regulation in mouse embryonic stem cells, Nature, 473, 389, 10.1038/nature09934 · DOI · Paper page
Cited by
RNA m5C oxidation by TET2 regulates chromatin state and leukaemogenesis
Zou, Z., Dou, X., Li, Y., Zhang, Z., Wang, J., Gao, B., Xiao, Y., Wang, Y., Zhao, L., Sun, C., et al. (2024) · Nature · doi:10.1038/s41586-024-07969-x · Cited by 103
Metadata
- Authors
- Abou-Jaoude, A., Huang, C.-Y., Flores, J. C., Ravichandran, M., Lei, R., Chrysanthou, S., & Dawlaty, M. M.
- Venue
- Stem Cell Research
- Publisher
- Elsevier BV
- Published
- 2022-05
- Volume
- 61
- Pages
- 102770
- ISSN
- 1873-5061
- PMC
- PMC10810145
- Cited by
- 4
- References
- 3
- Language
- en
- Licence
- Funders
- National Institutes of Health (R01GM122839, R01HL148852)
- New York State Department of Health (C32589GG)
- New York State Stem Cell Science
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