The DOI matches its Crossref record; 63 of the 63 references it cites were found in a registry or an index.
Wnt signaling in myogenesis
Open accessAbstract
The formation of skeletal muscle is a tightly regulated process that is critically modulated by Wnt signaling. Myogenesis is dependent on the precise and dynamic integration of multiple Wnt signals allowing for self-renewal and progression of muscle precursors in the myogenic lineage. Dis-regulation of Wnt signaling can lead to severe developmental defects and perturbation of muscle homeostasis. Recent work has revealed novel roles of the non-canonical planar-cell-polarity (PCP) and AKT/mTOR pathways in mediating the effects of Wnt on skeletal muscle. In this review, we discuss the role of Wnt signaling in myogenesis and in regulating the homeostasis of adult muscle.
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Cites
Wnt/β-catenin signaling and disease
Clevers, H., & Nusse, R. (2012) · Cell · doi:10.1016/j.cell.2012.05.012 · Cited by 5,554
Wnt proteins are lipid-modified and can act as stem cell growth factors
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Transcriptional regulation by calcium, calcineurin, and NFAT
Hogan, P. G., Chen, L., Nardone, J., & Rao, A. (2003) · Genes & Development · doi:10.1101/gad.1102703 · Cited by 1,991
Myogenic satellite cells: physiology to molecular biology
Hawke, T. J., & Garry, D. J. (2001) · Journal of Applied Physiology · doi:10.1152/jappl.2001.91.2.534 · Cited by 1,644
Increased Wnt signaling during aging alters muscle stem cell fate and increases fibrosis
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Asymmetric self-renewal and commitment of satellite stem cells in muscle
Kuang, S., Kuroda, K., Le Grand, F., & Rudnicki, M. A. (2007) · Cell · doi:10.1016/j.cell.2007.03.044 · Cited by 1,449 · cited 1×
“Satellite stem cells can also give rise to committed progenitor cells through asymmetric cell division and by this means control the overall satellite cell pool ([36], Figure 3).”
Self-citationA Pax3/Pax7-dependent population of skeletal muscle progenitor cells
Relaix, F., Rocancourt, D., Mansouri, A., & Buckingham, M. (2005) · Nature · doi:10.1038/nature03594 · Cited by 1,167
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Pax7-expressing satellite cells are indispensable for adult skeletal muscle regeneration
Sambasivan, R., Yao, R., Kissenpfennig, A., Van Wittenberghe, L., Paldi, A., Gayraud-Morel, B., Guenou, H., Malissen, B., Tajbakhsh, S., & Galy, A. (2011) · Development · doi:10.1242/dev.067587 · Cited by 939
Aberrant repair and fibrosis development in skeletal muscle
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“Fibrosis is often associated with muscular dystrophies, which is thought to contribute to disease pathology [51].”
Cited by
Sarcopenia: Aging-Related Loss of Muscle Mass and Function
Larsson, L., Degens, H., Li, M., Salviati, L., Lee, Y. I., Thompson, W., Kirkland, J. L., & Sandri, M. (2019) · Physiological Reviews · doi:10.1152/physrev.00061.2017 · Cited by 1,797
Pax7 is critical for the normal function of satellite cells in adult skeletal muscle
von Maltzahn, J., Jones, A. E., Parks, R. J., & Rudnicki, M. A. (2013) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.1307680110 · Cited by 602
Self-citationIntrinsic and extrinsic mechanisms regulating satellite cell function
Dumont, N. A., Wang, Y. X., & Rudnicki, M. A. (2015) · Development · doi:10.1242/dev.114223 · Cited by 491
Self-citationCellular dynamics in the muscle satellite cell niche
Bentzinger, C. F., Wang, Y. X., Dumont, N. A., & Rudnicki, M. A. (2013) · The EMBO Reports · doi:10.1038/embor.2013.182 · Cited by 398
Self-citationSkeletal muscle fibrosis: an overview
Mahdy, M. A. A. (2019) · Cell and Tissue Research · doi:10.1007/s00441-018-2955-2 · Cited by 363
Metadata
- Authors
- von Maltzahn, J., Chang, N. C., Bentzinger, C. F., & Rudnicki, M. A.
- Venue
- Trends in Cell Biology
- Publisher
- Elsevier BV
- Published
- 2012-11
- Volume
- 22
- Issue
- 11
- Pages
- 602-609
- ISSN
- 0962-8924 (ISSN-L)
- OpenAlex
- W1974547730
- Cited by
- 393
- References
- 63
- FWCI
- 1.77
- Type
- journal article
- Language
- en
- Open access
- green
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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