The DOI matches its Crossref record; 7 of the 7 references it cites were found in a registry or an index.
Oncogenic activities of human papillomaviruses
Open accessAbstract
Infectious etiologies for certain human cancers have long been suggested by epidemiological studies and studies with animals. Important support for this concept came from the discovery by Harald zur Hausen’s group that human cervical carcinoma almost universally contains certain “high-risk” human papillomavirus (HPV) types. Over the years, much has been learned about the carcinogenic activities of high-risk HPVs. These studies have revealed that two viral proteins, E6 and E7, that are consistently expressed in HPV-associated carcinomas, are necessary for induction and maintenance of the transformed phenotype. Hence, HPV-associated tumors are unique amongst human solid tumors in that they are universally caused by exposure to the same, molecularly defined oncogenic agents, and the molecular signal transduction pathways subverted by these viral transforming agents are frequently disrupted in other, non-virus associated human cancers.
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Cites
The human papilloma virus-16 E7-oncoprotein is able to bind to the retinoblastoma gene product
Dyson, N., Howley, P. M., Münger, K., & Harlow, E. (1989) · Science · doi:10.1126/science.2537532 · Cited by 3,017 · cited 1×
“The HPV E7 protein associates with the retinoblastoma tumor suppressor protein pRB (Dyson et al., 1989), which controls S-phase entry through association with E2F transcription factor family members (Figure 4).”
BackgroundWhyte, P., Buchkovich, K. J., Horowitz, J. M., Friend, S. H., Raybuck, M., Weinberg, R. A., & Harlow, E. (1988) · Nature · doi:10.1038/334124a0 · Cited by 1,403
Frisch, S. M., & Screaton, R. A. (2001) · Current Opinion in Cell Biology · doi:10.1016/s0955-0674(00)00251-9 · Cited by 1,249
Cytokinesis failure generating tetraploids promotes tumorigenesis in p53-null cells
Fujiwara, T., Bandi, M., Nitta, M., Ivanova, E. V., Bronson, R. T., & Pellman, D. (2005) · Nature · doi:10.1038/nature04217 · Cited by 1,067
Huibregtse, J. M., Scheffner, M., & Howley, P. M. (1991) · The EMBO Journal · doi:10.1002/j.1460-2075.1991.tb04990.x · Cited by 879
Myers, E. R., McCrory, D. C., Nanda, K., Bastian, L., & Matchar, D. B. (2000) · American Journal of Epidemiology · doi:10.1093/oxfordjournals.aje.a010166 · Cited by 470
Gewin, L., Myers, H., Kiyono, T., & Galloway, D. A. (2004) · Genes & Development · doi:10.1101/gad.1214704 · Cited by 277
Cited by
McLaughlin-Drubin, M. E., Crum, C. P., & Münger, K. (2011) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.1009933108 · Cited by 288
Self-citationHonegger, A., Schilling, D., Bastian, S., Sponagel, J., Kuryshev, V., Sültmann, H., Scheffner, M., Hoppe-Seyler, K., & Hoppe-Seyler, F. (2015) · PLOS Pathogens · doi:10.1371/journal.ppat.1004712 · Cited by 222
Spangle, J. M., & Münger, K. (2010) · Journal of Virology · doi:10.1128/jvi.00974-10 · Cited by 189
Spangle, J. M., & Munger, K. (2013) · PLoS Pathogens · doi:10.1371/journal.ppat.1003237 · Cited by 76
Modulation of microRNA-mRNA Target Pairs by Human Papillomavirus 16 Oncoproteins
Harden, M. E., Prasad, N., Griffiths, A., & Munger, K. (2017) · mBio · doi:10.1128/mbio.02170-16 · Cited by 64
Gao, R., Wu, X., Huang, Z., Wang, B., Li, F., Xu, H., & Ran, L. (2019) · OncoTargets and Therapy · doi:10.2147/ott.s182405 · Cited by 35
McLaughlin-Drubin, M., & Munger, K. (2013) · Viruses · doi:10.3390/v5051231 · Cited by 28
Self-citationYu, Y., & Munger, K. (2013) · Virology · doi:10.1016/j.virol.2013.08.013 · Cited by 15
Yu, Y., & Munger, K. (2012) · Virology · doi:10.1016/j.virol.2012.06.006 · Cited by 12
Metadata
- Authors
- McLaughlin-Drubin, M. E., & Münger, K.
- Venue
- Virus Research
- Publisher
- Elsevier BV
- Published
- 2009-08
- Volume
- 143
- Issue
- 2
- Pages
- 195-208
- ISSN
- 0168-1702 (ISSN-L)
- OpenAlex
- W2077155376
- Cited by
- 321
- References
- 7
- FWCI
- 14.39
- Type
- journal article
- Language
- en
- Open access
- green
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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