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Cited by 385

The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.

Motor Neuron–Derived Retinoid Signaling Specifies the Subtype Identity of Spinal Motor Neurons

Open access
Sockanathan, Shanthini, Jessell, Thomas M·1998·Cell

Abstract

The diversification of neuronal cell types in the vertebrate central nervous system depends on inductive signals provided by local organizing cell groups of both neural and nonneural origin. The influence of signals provided by postmitotic neurons on the fate of neurons born at subsequent development stages, however, remains unclear. We provide evidence that a retinoid-mediated signal provided by one subset of early-born spinal motor neurons imposes a local variation in the number of motor neurons generated at different axial levels and also specifies the identity of a later-born subset of motor neurons. Thus, in the vertebrate central nervous system the distinct fates of late-born neurons may be acquired in response to signals provided by early-born neurons.

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Crossref · 10.1016/s0092-8674(00)81591-3

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Metadata

Authors
Sockanathan, S., & Jessell, T. M.
Venue
Cell
Publisher
Elsevier BV
Published
1998-08
Volume
94
Issue
4
Pages
503-514
ISSN
0092-8674 (ISSN-L)
OpenAlex
W2010364898
Cited by
385
References
2
FWCI
8.90
Type
journal article
Language
en
Open access
hybrid

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CoChat. (2026). Citation check: Motor Neuron–Derived Retinoid Signaling Specifies the Subtype Identity of Spinal Motor Neurons. https://app.cochat.ai/papers/10.1016/s0092-8674(00)81591-3
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Sockanathan, S., & Jessell, T. M. (1998). Motor Neuron–Derived Retinoid Signaling Specifies the Subtype Identity of Spinal Motor Neurons. Cell. https://doi.org/10.1016/s0092-8674(00)81591-3

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.