The DOI matches its Crossref record; 25 of the 25 references it cites were found in a registry or an index.
Mechanisms underlying hypoxia-induced neuronal apoptosis
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Citations
Cites
Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell death
Oltval, Z. N., Milliman, C. L., & Korsmeyer, S. J. (1993) · Cell · doi:10.1016/0092-8674(93)90509-o · Cited by 6,003
Death Receptors: Signaling and Modulation
Ashkenazi, A., & Dixit, V. M. (1998) · Science · doi:10.1126/science.281.5381.1305 · Cited by 5,866
Mdm2 promotes the rapid degradation of p53
Haupt, Y., Maya, R., Kazaz, A., & Oren, M. (1997) · Nature · doi:10.1038/387296a0 · Cited by 4,506
Tumor suppressor p53 is a direct transcriptional activator of the human bax gene
Toshiyuki, M., & Reed, J. C. (1995) · Cell · doi:10.1016/0092-8674(95)90412-3 · Cited by 4,063
Nagata, S., & Golstein, P. (1995) · Science · doi:10.1126/science.7533326 · Cited by 3,979
Regulation of p53 stability by Mdm2
Kubbutat, M. H. G., Jones, S. N., & Vousden, K. H. (1997) · Nature · doi:10.1038/387299a0 · Cited by 3,312 · cited 2×
“The low concentrations of p53 appear to be the consequence of mdm2-regulated ubiquitin-mediated proteolysis (Haupt et al., 1997; Honda et al., 1997; Kubbutat et al., 1997; Midgley and Lane, 1997).”
Momand, J., Zambetti, G. P., Olson, D. C., George, D., & Levine, A. J. (1992) · Cell · doi:10.1016/0092-8674(92)90644-r · Cited by 3,113
Kastan, M. B., Zhan, Q., El-Deiry, W. S., Carrier, F., Jacks, T., Walsh, W. V., Plunkett, B. S., Vogelstein, B., & Fornace, A. J. (1992) · Cell · doi:10.1016/0092-8674(92)90593-2 · Cited by 3,047 · cited 1×
“GADD45 binding to PCNA is also important for stimulating DNA excision repair (Kastan et al., 1992; Smith et al., 1993; Hall et al., 1995).”
Identification and characterization of a new member of the TNF family that induces apoptosis
Wiley, S. R., Schooley, K., Smolak, P. J., Din, W. S., Huang, C.-P., Nicholl, J. K., Sutherland, G. R., Smith, T. D., Rauch, C., Smith, C. A., et al. (1995) · Immunity · doi:10.1016/1074-7613(95)90057-8 · Cited by 2,900
DNA damage-induced phosphorylation of p53 alleviates inhibition by MDM2
Shieh, S.-Y., Ikeda, M., Taya, Y., & Prives, C. (1997) · Cell · doi:10.1016/s0092-8674(00)80416-x · Cited by 2,100
Cited by
(2001) · PubMed Central · Cited by 289
Cortical and retinal defects caused by dosage-dependent reductions in VEGF-A paracrine signaling
Haigh, J. J., Morelli, P. I., Gerhardt, H., Haigh, K., Tsien, J., Damert, A., Miquerol, L., Muhlner, U., Klein, R., Ferrara, N., et al. (2003) · Developmental Biology · doi:10.1016/s0012-1606(03)00356-7 · Cited by 272
From oxygen to erythropoietin: Relevance of hypoxia for retinal development, health and disease
Caprara, C., & Grimm, C. (2012) · Progress in Retinal and Eye Research · doi:10.1016/j.preteyeres.2011.11.003 · Cited by 176
Ferguson, K. L., Vanderluit, J. L., Hébert, J. M., McIntosh, W. C., Tibbo, E., MacLaurin, J. G., Park, D. S., Wallace, V. A., Vooijs, M., McConnell, S. K., et al. (2002) · The EMBO Journal · doi:10.1093/emboj/cdf338 · Cited by 156
Beck, T. N., Nicolas, E., Kopp, M. C., & Golemis, E. A. (2014) · Oncoscience · doi:10.18632/oncoscience.64 · Cited by 50
Metadata
- Authors
- Banasiak, K. J., Xia, Y., & Haddad, G. G.
- Venue
- Progress in Neurobiology
- Publisher
- Elsevier BV
- Published
- 2000-10
- Volume
- 62
- Issue
- 3
- Pages
- 215-249
- ISSN
- 0301-0082 (ISSN-L)
- OpenAlex
- W1994682771
- Cited by
- 303
- References
- 25
- FWCI
- 5.75
- Type
- journal article
- Language
- en
- Open access
- closed
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- CoChat. (2026). Citation check: Mechanisms underlying hypoxia-induced neuronal apoptosis. https://app.cochat.ai/papers/10.1016/s0301-0082(00)00011-3
- The paper
- Banasiak, K. J., Xia, Y., & Haddad, G. G. (2000). Mechanisms underlying hypoxia-induced neuronal apoptosis. Progress in Neurobiology. https://doi.org/10.1016/s0301-0082(00)00011-3
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
