The DOI matches its Crossref record; 28 of the 28 references it cites were found in a registry or an index.
Cellular senescence as a tumor-suppressor mechanism
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In the citing paper
“Although little is known about how cells choose between apoptotic and senescence responses, there is scant doubt that both responses are crucial for suppressing cancer (Campisi, 2001; Green and Evan, 2002).”
From Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors (2005) · cites this paper as Campisi, 2001
In the citing paper
“These pathways are governed by the gatekeeper tumor suppressor proteins p53 and pRB (Bringold and Serrano, 2000; Lundberg et al., 2000; Campisi, 2001).”
From Senescent cells, tumor suppression, and organismal aging: good citizens, bad neighbors (2005) · cites this paper as Campisi, 2001
In the citing paper
“Under standard conditions of tissue culture, normal somatic cells achieve a limited number of population doublings before entering a growth-arrested state known as replicative senescence (6,7).”
From Glycolytic Enzymes Can Modulate Cellular Life Span (2005) · cites this paper as [7]
Citations
Cites
Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a
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“7 Serrano, M. et al. (1997) Oncogenic ras provokes premature cell”
BackgroundAtmospheric carbon dioxide concentrations over the past 60 million years
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Senescence of human fibroblasts induced by oncogenic Raf
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Microarray analysis of replicative senescence
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Opposing effects of Ets and Id proteins on p16INK4a expression during cellular senescence
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Replicative Senescence: Implications for in Vivo Aging and Tumor Suppression
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PML is induced by oncogenic ras and promotes premature senescence
Ferbeyre, G., de Stanchina, E., Querido, E., Baptiste, N., Prives, C., & Lowe, S. W. (2000) · Genes & Development · doi:10.1101/gad.14.16.2015 · Cited by 536
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Nicotinamide extends replicative lifespan of human cells
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Emerging roles of RB family: New defense mechanisms against tumor progression
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Immortalization of Cementoblast Progenitor Cells With Bmi-1 and TERT
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Metadata
- Authors
- Campisi, J.
- Affiliations
- Lawrence Berkeley National Laboratory (US)
- Corresponding author
- Campisi, Judith
- ORCID
- Campisi, Judith
- Venue
- Trends in Cell Biology
- Publisher
- Elsevier BV
- Published
- 2001-11
- Volume
- 11
- Pages
- S27-S31
- ISSN
- 0962-8924 (ISSN-L)
- OpenAlex
- W4232925121
- Cited by
- 390
- References
- 28
- Citations by year
- 2026: 7 · 2025: 7 · 2024: 13 · 2023: 12 · 2022: 16 · 2021: 12 · 2020: 10 · 2019: 12 · 2018: 16 · 2017: 16 · 2016: 19 · 2015: 21 · 2014: 16 · 2013: 17 · 2012: 14
- FWCI
- 2.68
- Type
- journal article
- Language
- en
- Open access
- closed
- Locations
- Trends in Cell Biology (publishedVersion)
- Topics
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Cell Biology › Telomeres, Telomerase, and Senescence
- Life Sciences › Biochemistry, Genetics and Molecular Biology › Aging › Genetics, Aging, and Longevity in Model Organisms
- Health Sciences › Medicine › Oncology › Cancer-related Molecular Pathways
- Keywords
- cellular senescence, tumor suppression, cancer prevention, oncogenic transformation, growth arrest, tissue renewal
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