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Differentiation and reversal of malignant changes in colon cancer through PPARγ
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PPARγ as a new therapeutic target in inflammatory bowel diseases
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Self-citationIncreased expression of PPARγ in high fat diet-induced liver steatosis in mice
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15-Deoxy-Δ12,14-prostaglandin J2-induced apoptosis does not require PPARγ in breast cancer cells
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Use of Thiazolidinediones and the Risk of Colorectal Cancer in Patients With Diabetes
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Engaging plasticity: Differentiation therapy in solid tumors
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Metadata
- Authors
- Sarraf, P., Mueller, E., Jones, D., King, F. J., DeAngelo, D. J., Partridge, J. B., Holden, S. A., Chen, L. B., Singer, S., Fletcher, C., & Spiegelman, B. M.
- Venue
- Nature Medicine
- Published
- 1998-09
- Volume
- 4
- Issue
- 9
- Pages
- 1046-1052
- ISSN
- 1078-8956 (ISSN-L)
- 1546-170X
- DOI
- 10.1038/2030
- OpenAlex
- W2091321756
- Cited by
- 988
- References
- 4
- FWCI
- 19.36
- Type
- journal article
- Language
- en
- Open access
- closed
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.