Cited by 175Parkinson's Disease Mechanisms and TreatmentsNuclear Receptors and SignalingMitochondrial Function and Pathology

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Mitochondrial dysfunction in Parkinson's disease

Schapira, A H V·2007·Cell Death & Differentiation

DOI 10.1038/sj.cdd.4402160

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Pramanik, S., Debnath, B., Chakraborty, A. J., Islam, A., Mullick, S., Chaudhary, P., Nath, R., Chellappan, D. K., Mondal, M., & Ashique, S. (2026) · Molecular Neurobiology · doi:10.1007/s12035-026-06008-2
SUMOylation modulates α-synuclein toxicity and fibril formation
Krumova, P. (2010) · eDiss (Georg-August-Universität Göttingen) · doi:10.53846/goediss-3286

Authors: Schapira, A. H. V.
Affiliations: The Royal Free Hospital (GB)
Corresponding author: Schapira, A H V
ORCID: Schapira, A H V
Venue: Cell Death & Differentiation
Publisher: Springer Science and Business Media LLC
Published: 2007-04-27
Volume: 14
Issue: 7
Pages: 1261-1266
ISSN: 1350-9047 (ISSN-L)
1476-5403
DOI: 10.1038/sj.cdd.4402160
OpenAlex: W1990162995
Cited by: 175
References: 95
Citations by year: 2026: 5 · 2025: 7 · 2024: 7 · 2023: 7 · 2022: 13 · 2021: 12 · 2020: 10 · 2019: 8 · 2018: 9 · 2017: 9 · 2016: 3 · 2015: 13 · 2014: 7 · 2013: 14 · 2012: 6
FWCI: 1.46
Type: journal article
Language: en
Open access: closed
Licence: www.springer.com/tdm (tdm)
Locations: Cell Death and Differentiation (publishedVersion)
PubMed (publishedVersion)
UCL Discovery (University College London) (submittedVersion)
Topics: Health Sciences › Medicine › Clinical Neurology › Parkinson's Disease Mechanisms and Treatments
Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Nuclear Receptors and Signaling
Life Sciences › Biochemistry, Genetics and Molecular Biology › Molecular Biology › Mitochondrial Function and Pathology
Keywords: Parkinson's disease, mitochondrial dysfunction, dopaminergic neuron loss, substantia nigra, PINK1, DJ-1, OMI, PARK genes, familial Parkinson's disease, sporadic Parkinson's disease, oxidative damage, neuroinflammation, proteasomal dysfunction, mitochondrial respiratory chain, genetic mutations
MeSH terms: Protein Deglycase DJ-1, PTEN-Induced Putative Kinase, Animals, DNA, Mitochondrial, Humans, Mitochondria, Mutation, Parkinson Disease, Protein Kinases, Oncogene Proteins, Mitochondrial Diseases, Electron Transport Complex I, Ubiquitin-Protein Ligases, Intracellular Signaling Peptides and Proteins

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