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Cited by 4,877

The DOI matches its Crossref record; 106 of the 106 references it cites were found in a registry or an index.

The Pathogenesis of Atherosclerosis — An Update

Ross, Russell·1986·New England Journal of Medicine

Abstract

CARDIOVASCULAR disease remains the chief cause of death in the United States and Western Europe, and atherosclerosis, the principal cause of myocardial and cerebral infarction, accounts for the majority of these deaths.1 This review, like its predecessor,2 will not attempt to cover all literature on atherosclerosis. In a previous review of the pathogenesis of atherosclerosis,2 Glomset and I discussed various hypotheses of atherogenesis2 3 4 5 6 7 and emphasized the importance of intimal smooth-muscle proliferation as the key event in the development of the advanced lesions of atherosclerosis. The response-to-injury hypothesis of atherogenesis2 3 4 5 6 proposes that "injury" to the endothelium is the initiating event in . . .

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Metadata

Authors
Ross, R.
Venue
New England Journal of Medicine
Published
1986-02-20
Volume
314
Issue
8
Pages
488-500
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W2061755142
Cited by
4,877
References
106
FWCI
90.17
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: The Pathogenesis of Atherosclerosis — An Update. https://app.cochat.ai/papers/10.1056/nejm198602203140806
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Ross, R. (1986). The Pathogenesis of Atherosclerosis — An Update. New England Journal of Medicine. https://doi.org/10.1056/nejm198602203140806

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.