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Cited by 806

The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.

Plasminogen-Activator Inhibitor Type 1 and Coronary Artery Disease

Kohler, Hans P., Grant, Peter J.·2000·New England Journal of Medicine

Abstract

The development of coronary artery disease, and specifically myocardial infarction, involves hyperplasia of arterial smooth muscle, the development of fatty streaks, atheroma formation, plaque rupture, and ultimately thrombus formation and vessel occlusion.1 These changes are in part genetically determined, as demonstrated by the fact that the risk of myocardial infarction in persons who have a first-degree relative with myocardial infarction is seven times the risk in persons who do not.2,3 This finding is often used to argue that coronary artery disease has a genetic basis, but the extent to which a shared environment contributes to the risk must also . . .

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Registry

Crossref · 10.1056/nejm200006153422406

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Metadata

Authors
Kohler, H. P., & Grant, P. J.
Venue
New England Journal of Medicine
Published
2000-06-15
Volume
342
Issue
24
Pages
1792-1801
ISSN
  • 0028-4793 (ISSN-L)
  • 1533-4406
OpenAlex
W2117931787
Cited by
806
References
6
FWCI
20.76
Type
journal article
Language
en
Open access
closed

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CoChat. (2026). Citation check: Plasminogen-Activator Inhibitor Type 1 and Coronary Artery Disease. https://app.cochat.ai/papers/10.1056/nejm200006153422406
The paper
Kohler, H. P., & Grant, P. J. (2000). Plasminogen-Activator Inhibitor Type 1 and Coronary Artery Disease. New England Journal of Medicine. https://doi.org/10.1056/nejm200006153422406

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.