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Alteration of Lysosome Fusion and Low-grade Inflammation Mediated by Super-low-dose Endotoxin

Baker, Bianca, Geng, Shuo, Chen, Keqiang, Diao, Na, Yuan, Ruoxi, Xu, Xiguang, Dougherty, Sean, Stephenson, Caroline, Xiong, Huabao, Chu, Hong Wei, Li, Liwu·2015·Journal of Biological Chemistry
DOI 10.1074/jbc.m114.611442
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  1. Andreasen, 2011, Type 2 diabetes is associated with altered NF-κB DNA binding activity, JNK phosphorylation, and AMPK phosphorylation in skeletal muscle after LPS, PLoS ONE, 6, e23999, 10.1371/journal.pone.0023999 · DOI
  2. Kim, 2000, Regional difference in susceptibility to lipopolysaccharide-induced neurotoxicity in the rat brain: role of microglia, J. Neurosci, 20, 6309, 10.1523/JNEUROSCI.20-16-06309.2000 · DOI
  3. Maitra, 2009, An innate immunity signaling process suppresses macrophage ABCA1 expression through IRAK-1-mediated downregulation of retinoic acid receptor α and NFATc2, Mol. Cell. Biol, 29, 5989, 10.1128/MCB.00541-09 · DOI
  4. Shoelson, 2006, Inflammation and insulin resistance, J. Clin. Invest, 116, 1793, 10.1172/JCI29069 · DOI · Paper page
  5. Chen, 2012, Autophagy dysregulation in amyotrophic lateral sclerosis, Brain Pathol, 22, 110, 10.1111/j.1750-3639.2011.00546.x · DOI
  6. Jiang, 2014, Autophagy and human diseases, Cell Res, 24, 69, 10.1038/cr.2013.161 · DOI
  7. Wolfe, 2013, Autophagy failure in Alzheimer's disease and the role of defective lysosomal acidification, Eur. J. Neurosci, 37, 1949, 10.1111/ejn.12169 · DOI
  8. Yang, 2010, Dysregulation of autophagy and Parkinson's disease: the MEF2D link, Apoptosis, 15, 1410, 10.1007/s10495-010-0475-y · DOI
  9. Lassenius, 2011, Bacterial endotoxin activity in human serum is associated with dyslipidemia, insulin resistance, obesity, and chronic inflammation, Diabetes Care, 34, 1809, 10.2337/dc10-2197 · DOI
  10. Chang, 2011, Metabolic endotoxemia: a novel concept in chronic disease pathology, J. Med. Sci, 31, 191
  11. Wiesner, 2010, Low doses of lipopolysaccharide and minimally oxidized low-density lipoprotein cooperatively activate macrophages via nuclear factor κB and activator protein-1: possible mechanism for acceleration of atherosclerosis by subclinical endotoxemia, Circ. Res, 107, 56, 10.1161/CIRCRESAHA.110.218420 · DOI
  12. Maitra, 2012, Molecular mechanisms responsible for the selective and low-grade induction of proinflammatory mediators in murine macrophages by lipopolysaccharide, J. Immunol, 189, 1014, 10.4049/jimmunol.1200857 · DOI
  13. Parkinson-Lawrence, 2010, Lysosomal storage disease: revealing lysosomal function and physiology, Physiology, 25, 102, 10.1152/physiol.00041.2009 · DOI
  14. Cannizzo, 2012, Age-related oxidative stress compromises endosomal proteostasis, Cell Rep, 2, 136, 10.1016/j.celrep.2012.06.005 · DOI · Paper page
  15. Mayer, 2013, Rescue of dysfunctional autophagy attenuates hyperinflammatory responses from cystic fibrosis cells, J. Immunol, 190, 1227, 10.4049/jimmunol.1201404 · DOI
  16. Wang, 2005, CpG-independent synergistic induction of β-chemokines and a dendritic cell phenotype by orthophosphorothioate oligodeoxynucleotides and granulocyte-macrophage colony-stimulating factor in elutriated human primary monocytes, J. Immunol, 174, 6113, 10.4049/jimmunol.174.10.6113 · DOI
  17. Miura, 2011, Ehrlichia chaffeensis induces monocyte inflammatory responses through MyD88, ERK, and NF-κB but not through TRIF, interleukin-1 receptor 1 (IL-1R1)/IL-18R1, or Toll-like receptors, Infect. Immun, 79, 4947, 10.1128/IAI.05640-11 · DOI
  18. Mao, 2011, AMPK activates autophagy by phosphorylating ULK1, Circ. Res, 108, 787, 10.1161/RES.0b013e3182194c29 · DOI
  19. Wong, 2013, The ULK1 complex: sensing nutrient signals for autophagy activation, Autophagy, 9, 124, 10.4161/auto.23323 · DOI
  20. Devereaux, 2013, Regulation of mammalian autophagy by class II and III PI 3-kinases through PI3P synthesis, PLoS ONE, 8, e76405, 10.1371/journal.pone.0076405 · DOI · Paper page
  21. Jaber, 2012, Class III PI3K Vps34 plays an essential role in autophagy and in heart and liver function, Proc. Natl. Acad. Sci. U.S.A, 109, 2003, 10.1073/pnas.1112848109 · DOI
  22. Juhász, 2008, The class III PI(3)K Vps34 promotes autophagy and endocytosis but not TOR signaling in Drosophila, J. Cell Biol, 181, 655, 10.1083/jcb.200712051 · DOI
  23. Noda, 2010, Regulation of membrane biogenesis in autophagy via PI3P dynamics, Semin. Cell Dev. Biol, 21, 671, 10.1016/j.semcdb.2010.04.002 · DOI
  24. Furuya, 2010, Negative regulation of Vps34 by Cdk-mediated phosphorylation, Mol. Cell, 38, 500, 10.1016/j.molcel.2010.05.009 · DOI
  25. Kim, 2013, Differential regulation of distinct Vps34 complexes by AMPK in nutrient stress and autophagy, Cell, 152, 290, 10.1016/j.cell.2012.12.016 · DOI
  26. Funderburk, 2010, The beclin 1-VPS34 complex–at the crossroads of autophagy and beyond, Trends Cell Biol, 20, 355, 10.1016/j.tcb.2010.03.002 · DOI
  27. Li, 2004, Characterization of Tollip protein upon lipopolysaccharide challenge, Mol. Immunol, 41, 85, 10.1016/j.molimm.2004.03.009 · DOI · Paper page
  28. Piao, 2009, Endotoxin tolerance dysregulates MyD88- and Toll/IL-1R domain-containing adapter inducing IFN-β-dependent pathways and increases expression of negative regulators of TLR signaling, J. Leukoc. Biol, 86, 863, 10.1189/jlb.0309189 · DOI
  29. Zhang, 2002, Negative regulation of Toll-like receptor-mediated signaling by Tollip, J. Biol. Chem, 277, 7059, 10.1074/jbc.M109537200 · DOI
  30. Puri, 2012, Dysfunctions in endosomal-lysosomal and autophagy pathways underlie neuropathology in a mouse model for Lafora disease, Hum. Mol. Genet, 21, 175, 10.1093/hmg/ddr452 · DOI
  31. Katoh, 2004, Tollip and Tom1 form a complex and recruit ubiquitin-conjugated proteins onto early endosomes, J. Biol. Chem, 279, 24435, 10.1074/jbc.M400059200 · DOI
  32. Katoh, 2006, Recruitment of clathrin onto endosomes by the Tom1-Tollip complex, Biochem. Biophys. Res. Commun, 341, 143, 10.1016/j.bbrc.2005.12.156 · DOI
  33. Mitra, 2013, Ubiquitin interacts with the Tollip C2 and CUE domains and inhibits binding of Tollip to phosphoinositides, J. Biol. Chem, 288, 25780, 10.1074/jbc.M113.484170 · DOI · Paper page
  34. Lu, 2014, Autophagic clearance of polyQ proteins mediated by ubiquitin-Atg8 adaptors of the conserved CUET protein family, Cell, 158, 549, 10.1016/j.cell.2014.05.048 · DOI · Paper page
  35. Emanuel, 2014, Induction of lysosomal biogenesis in atherosclerotic macrophages can rescue lipid-induced lysosomal dysfunction and downstream sequelae, Arterioscler. Thromb. Vasc. Biol, 34, 1942, 10.1161/ATVBAHA.114.303342 · DOI
  36. Madsen, 2013, M2-like macrophages are responsible for collagen degradation through a mannose receptor-mediated pathway, J. Cell Biol, 202, 951, 10.1083/jcb.201301081 · DOI
  37. Morris, 2012, Molecular mechanisms and pathological consequences of endotoxin tolerance and priming, Arch. Immunol. Ther. Exp, 60, 13, 10.1007/s00005-011-0155-9 · DOI
  38. Capelluto, 2012, Tollip: a multitasking protein in innate immunity and protein trafficking, Microbes Infect, 14, 140, 10.1016/j.micinf.2011.08.018 · DOI
  39. Shah, 2012, Human TOLLIP regulates TLR2 and TLR4 signaling and its polymorphisms are associated with susceptibility to tuberculosis, J. Immunol, 189, 1737, 10.4049/jimmunol.1103541 · DOI
  40. Montoya-Buelna, 2013, Role of Toll-interacting protein gene polymorphisms in leprosy Mexican patients, BioMed Res. Int, 2013, 459169, 10.1155/2013/459169 · DOI
  41. Noth, 2013, Genetic variants associated with idiopathic pulmonary fibrosis susceptibility and mortality: a genome-wide association study, Lancet Respir. Med, 1, 309, 10.1016/S2213-2600(13)70045-6 · DOI
  42. Song, 2011, Variants in the Toll-interacting protein gene are associated with susceptibility to sepsis in the Chinese Han population, Crit. Care, 15, R12, 10.1186/cc9413 · DOI
  43. Schimming, 2007, Association of Toll-interacting protein gene polymorphisms with atopic dermatitis, BMC Dermatol, 7, 3, 10.1186/1471-5945-7-3 · DOI

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Metadata

Authors
Baker, B., Geng, S., Chen, K., Diao, N., Yuan, R., Xu, X., Dougherty, S., Stephenson, C., Xiong, H., Chu, H. W., & Li, L.
Venue
Journal of Biological Chemistry
Publisher
Elsevier BV
Published
2015-03
Volume
290
Issue
10
Pages
6670-6678
ISSN
0021-9258
DOI
10.1074/jbc.m114.611442
Cited by
47
References
2
Language
en
Licence
Funders
National Institutes of Health (R01HL115835, AI106287)

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