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The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

CPEB2, CPEB3 and CPEB4 are coordinately regulated by miRNAs recognizing conserved binding sites in paralog positions of their 3′-UTRs

Morgan, Marcos, Iaconcig, Alessandra, Muro, Andrés Fernando·2010·Nucleic Acids Research
DOI 10.1093/nar/gkq635PMC PMC2995058
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Crossref · 10.1093/nar/gkq635

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  • miR-92a posttranscriptionally silences Cpeb3.

    “In support of a posttranscriptional silencing of Cpeb3 by miR-92a (39), we observed an induction of neuronal CPEB3 protein expression in miR-92a knockout spinal cords and found a direct interaction of miR-92a with Cpeb3 3′UTR.”

    From MicroRNA-92a–CPEB3 axis protects neurons against inflammatory neurodegeneration (2023) · cites this paper as [39]

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Research works printed but not linked: 39

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  5. Theis, 2003, Two previously undescribed members of the mouse CPEB family of genes and their inducible expression in the principal cell layers of the hippocampus, Proc. Natl Acad. Sci. USA, 100, 9602, 10.1073/pnas.1133424100 · DOI
  6. Huang, 2006, CPEB3 and CPEB4 in neurons: analysis of RNA-binding specificity and translational control of AMPA receptor GluR2 mRNA, EMBO J., 25, 4865, 10.1038/sj.emboj.7601322 · DOI
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  9. Lewis, 2003, Prediction of mammalian microRNA targets, Cell, 115, 787, 10.1016/S0092-8674(03)01018-3 · DOI · Paper page
  10. Schickel, 2008, MicroRNAs: key players in the immune system, differentiation, tumorigenesis and cell death, Oncogene, 27, 5959, 10.1038/onc.2008.274 · DOI
  11. Jiang, 2009, miR2Disease: a manually curated database for microRNA deregulation in human disease, Nucleic Acids Res., 37, D98, 10.1093/nar/gkn714 · DOI
  12. Mendell, 2008, miRiad roles for the miR-17-92 cluster in development and disease, Cell, 133, 217, 10.1016/j.cell.2008.04.001 · DOI
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  14. Friedman, 2009, Most mammalian mRNAs are conserved targets of microRNAs, Genome Res., 19, 92, 10.1101/gr.082701.108 · DOI · Paper page
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  17. Lewis, 2005, Conserved seed pairing, often flanked by adenosines, indicates that thousands of human genes are microRNA targets, Cell, 120, 15, 10.1016/j.cell.2004.12.035 · DOI · Paper page
  18. Schwartz, 2003, MultiPipMaker and supporting tools: Alignments and analysis of multiple genomic DNA sequences, Nucleic Acids Res., 31, 3518, 10.1093/nar/gkg579 · DOI
  19. Livak, 2001, Analysis of relative gene expression data using real-time quantitative PCR and the 2(-Delta Delta C(T)) Method, Methods, 25, 402, 10.1006/meth.2001.1262 · DOI · Paper page
  20. Muro, 2003, Regulated splicing of the fibronectin EDA exon is essential for proper skin wound healing and normal lifespan, J. Cell. Biol., 162, 149, 10.1083/jcb.200212079 · DOI · Paper page
  21. Muro, 2000, Mild spherocytic hereditary elliptocytosis and altered levels of alpha- and gamma-adducins in beta-adducin-deficient mice, Blood, 95, 3978, 10.1182/blood.V95.12.3978 · DOI
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  23. Giraldez, 2006, Zebrafish MiR-430 promotes deadenylation and clearance of maternal mRNAs, Science, 312, 75, 10.1126/science.1122689 · DOI · Paper page
  24. Wakiyama, 2007, Let-7 microRNA-mediated mRNA deadenylation and translational repression in a mammalian cell-free system, Genes Dev., 21, 1857, 10.1101/gad.1566707 · DOI
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  28. Kedde, 2007, RNA-binding protein Dnd1 inhibits microRNA access to target mRNA, Cell, 131, 1273, 10.1016/j.cell.2007.11.034 · DOI
  29. Igea, 2010, Meiosis requires a translational positive loop where CPEB1 ensues its replacement by CPEB4, EMBO J., 29, 2182, 10.1038/emboj.2010.111 · DOI
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Metadata

Authors
Morgan, M., Iaconcig, A., & Muro, A. F.
Venue
Nucleic Acids Research
Publisher
Oxford University Press (OUP)
Published
2010-07-24
Volume
38
Issue
21
Pages
7698-7710
ISSN
  • 1362-4962
  • 0305-1048
DOI
10.1093/nar/gkq635
PMC
PMC2995058
Cited by
29
References
3
Language
en
Licence
creativecommons.org/licenses/by-nc/2.5 (vor, 2298 d)

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