The DOI matches its Crossref record; 114 of the 114 references it cites were found in a registry or an index.
Programmed cell death in spinal cord injury pathogenesis and therapy
Open accessAbstract
Spinal cord injury (SCI) always leads to functional deterioration due to a series of processes including cell death. In recent years, programmed cell death (PCD) is considered to be a critical process after SCI, and various forms of PCD were discovered in recent years, including apoptosis, necroptosis, autophagy, ferroptosis, pyroptosis and paraptosis. Unlike necrosis, PCD is known as an active cell death mediated by a cascade of gene expression events, and it is crucial for elimination unnecessary and damaged cells, as well as a defence mechanism. Therefore, it would be meaningful to characterize the roles of PCD to not only enhance our understanding of the pathophysiological processes, but also improve functional recovery after SCI. This review will summarize and explore the most recent advances on how apoptosis, necroptosis, autophagy, ferroptosis, pyroptosis and paraptosis are involved in SCI. This review can help us to understand the various functions of PCD in the pathological processes of SCI, and contribute to our novel understanding of SCI of unknown aetiology in the near future.
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Cites
2 retracted
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RetractedParaptosis triggers mitochondrial pathway-mediated apoptosis in Alzheimer's disease
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RetractedFerroptosis: An Iron-Dependent Form of Nonapoptotic Cell Death
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Ferroptosis: A Regulated Cell Death Nexus Linking Metabolism, Redox Biology, and Disease
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Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury
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Mixed Lineage Kinase Domain-like Protein Mediates Necrosis Signaling Downstream of RIP3 Kinase
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Cited by
Zhao, L., Zhang, H., Li, N., Chen, J., Xu, H., Wang, Y., & Liang, Q. (2023) · Journal of Ethnopharmacology · doi:10.1016/j.jep.2023.116306 · Cited by 963
Spinal cord injury: molecular mechanisms and therapeutic interventions
Hu, X., Xu, W., Ren, Y., Wang, Z., He, X., Huang, R., Ma, B., Zhao, J., Zhu, R., & Cheng, L. (2023) · Signal Transduction and Targeted Therapy · doi:10.1038/s41392-023-01477-6 · Cited by 770
Zhang, M., Han, X., Yan, L., Fu, Y., Kou, H., Shang, C., Wang, J., Liu, H., Jiang, C., Wang, J., et al. (2024) · CNS Neuroscience & Therapeutics · doi:10.1111/cns.14781 · Cited by 32
Metadata
- Authors
- Shi, Z., Yuan, S., Shi, L., Li, J., Ning, G., Kong, X., & Feng, S.
- Venue
- Cell Proliferation
- Publisher
- Wiley
- Published
- 2021-01-27
- Volume
- 54
- Issue
- 3
- Pages
- e12992-e12992
- ISSN
- 0960-7722 (ISSN-L)
- 1365-2184
- OpenAlex
- W3124756354
- Cited by
- 293
- References
- 114
- FWCI
- 25.94
- Type
- journal article
- Language
- en
- Open access
- gold
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- Shi, Z., Yuan, S., Shi, L., Li, J., Ning, G., Kong, X., & Feng, S. (2021). Programmed cell death in spinal cord injury pathogenesis and therapy. Cell Proliferation. https://doi.org/10.1111/cpr.12992
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
