Common Structure of Soluble Amyloid Oligomers Implies Common Mechanism of Pathogenesis

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Diffusible, nonfibrillar ligands derived from Aβ 1–42 are potent central nervous system neurotoxins

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Inherent toxicity of aggregates implies a common mechanism for protein misfolding diseases

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Soluble Amyloid β Peptide Concentration as a Predictor of Synaptic Change in Alzheimer's Disease

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Amyloid pores from pathogenic mutations

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Nucleated conformational conversion and the replication of conformational information by a prion determinant.

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Protofibrillar Intermediates of Amyloid β-Protein Induce Acute Electrophysiological Changes and Progressive Neurotoxicity in Cortical Neurons

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Fibrils Formed in Vitro from α-Synuclein and Two Mutant Forms Linked to Parkinson's Disease are Typical Amyloid

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Water-soluble Aβ(N-40, N-42) oligomers in normal and Alzheimer disease brains

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Assembly of Aβ Amyloid Protofibrils: An in Vitro Model for a Possible Early Event in Alzheimer's Disease

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α-Synuclein Oligomers: an Amyloid Pore?

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Bridging the gap: From protein misfolding to protein misfolding diseases

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Transient dynamics of Aβ contribute to toxicity in Alzheimer’s disease

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Dysregulated phosphorylation of Ca2+/calmodulin‐dependent protein kinase II‐α in the hippocampus of subjects with mild cognitive impairment and Alzheimer’s disease

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Sulfated Glycosaminoglycans Accelerate Transthyretin Amyloidogenesis by Quaternary Structural Conversion

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A Fluorescent Pentameric Thiophene Derivative Detects in Vitro-Formed Prefibrillar Protein Aggregates

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Biflavonoids Are Superior to Monoflavonoids in Inhibiting Amyloid-β Toxicity and Fibrillogenesis via Accumulation of Nontoxic Oligomer-like Structures

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The Inhalation Anesthetic Desflurane Induces Caspase Activation and Increases Amyloid β-Protein Levels under Hypoxic Conditions

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Alzheimer’s disease Aβ assemblies mediating rapid disruption of synaptic plasticity and memory

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Activation of Peroxisome Proliferator-Activated Receptor-γ by Rosiglitazone Protects Human Islet Cells against Human Islet Amyloid Polypeptide Toxicity by a Phosphatidylinositol 3′-Kinase-Dependent Pathway

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Passive anti-amyloid immunotherapy in Alzheimer's disease: What are the most promising targets?

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α-Helical Structures Drive Early Stages of Self-Assembly of Amyloidogenic Amyloid Polypeptide Aggregate Formation in Membranes

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The cellular prion protein traps Alzheimer's Aβ in an oligomeric form and disassembles amyloid fibers

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An Account of Amyloid Oligomers: Facts and Figures Obtained from Experiments and Simulations

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Origin of metastable oligomers and their effects on amyloid fibril self-assembly

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Chemical sensing platforms for detecting trace-level Alzheimer's core biomarkers

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Core Structure of Amyloid Fibrils Formed by Residues 106–126 of the Human Prion Protein

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Amyloid‐β oligomers are inefficiently measured by enzyme‐linked immunosorbent assay

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Structural, morphological, and kinetic studies of β-amyloid peptide aggregation on self-assembled monolayers

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Diversity of kinetic pathways in amyloid fibril formation

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Neurodegenerative diseases linked to misfolded proteins and their therapeutic approaches: A review

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α-Helix Mimetics as Modulators of Aβ Self-Assembly

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Rational design of a conformation-specific antibody for the quantification of Aβ oligomers

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Modeling Polyglutamine Pathogenesis in C. elegans

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Molecular Aspects of the Stress Response: Chaperones, Membranes and Networks

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The Contrasting Effect of Macromolecular Crowding on Amyloid Fibril Formation

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An improved method for generating consistent soluble amyloid-beta oligomer preparations for in vitro neurotoxicity studies

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Postsynaptic Receptors for Amyloid-β Oligomers as Mediators of Neuronal Damage in Alzheimer’s Disease

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Distinct Region-Specific α-Synuclein Oligomers in A53T Transgenic Mice: Implications for Neurodegeneration

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Matrix metalloproteinase‐7 disrupts dendritic spines in hippocampal neurons through NMDA receptor activation

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Atorvastatin and pitavastatin improve cognitive function and reduce senile plaque and phosphorylated tau in aged APP mice

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Glutamate and Amyloid β-Protein Rapidly Inhibit Fast Axonal Transport in Cultured Rat Hippocampal Neurons by Different Mechanisms

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Amitriptyline-Mediated Cognitive Enhancement in Aged 3×Tg Alzheimer's Disease Mice Is Associated with Neurogenesis and Neurotrophic Activity

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Amyloid-like ribbons of amelogenins in enamel mineralization

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SERS Detection of Amyloid Oligomers on Metallorganic-Decorated Plasmonic Beads

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Aggregation of Disordered Proteins Associated with Neurodegeneration

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Galantamine inhibits β-amyloid aggregation and cytotoxicity

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Inhibitory effect of green tea extract on β-amyloid-induced PC12 cell death by inhibition of the activation of NF-κB and ERK/p38 MAP kinase pathway through antioxidant mechanisms

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Detecting Morphologically Distinct Oligomeric Forms of α-Synuclein

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Protein folding: Then and now

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Molecular Basis of Orb2 Amyloidogenesis and Blockade of Memory Consolidation

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Cellular Receptors of Amyloid β Oligomers (AβOs) in Alzheimer’s Disease

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Small organic probes as amyloid specific ligands – Past and recent molecular scaffolds

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Protein aggregation and ER stress

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Detection of Polyglutamine Protein Oligomers in Cells by Fluorescence Correlation Spectroscopy

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Hemoglobin promotes A? oligomer formation and localizes in neurons and amyloid deposits

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Amyloid Fibrils Trigger the Release of Neutrophil Extracellular Traps (NETs), Causing Fibril Fragmentation by NET-associated Elastase

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The Hsp104 N-Terminal Domain Enables Disaggregase Plasticity and Potentiation

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TRIM11 protects against tauopathies and is down-regulated in Alzheimer’s disease

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A common landscape for membrane‐active peptides

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Monitoring the Process of HypF Fibrillization and Liposome Permeabilization by Protofibrils

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Why Alzheimer's is a disease of memory: The attack on synapses by Aß oligomers (ADDLs)

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Heavy metals toxicity: effect of cadmium ions on amyloid beta protein 1–42. Possible implications for Alzheimer’s disease

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Interaction of huntingtin fragments with brain membranes – clues to early dysfunction in Huntington's disease

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Calcium-activated Calpain-2 Is a Mediator of Beta Cell Dysfunction and Apoptosis in Type 2 Diabetes

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A β 1-42 Induces Mild Endoplasmic Reticulum Stress in an Aggregation State–Dependent Manner

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Sequestration of Toxic Oligomers by HspB1 as a Cytoprotective Mechanism

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Insights into the molecular basis of the differing susceptibility of varying cell types to the toxicity of amyloid aggregates

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Mechanism of amyloidogenesis: nucleation-dependent fibrillation versus double-concerted fibrillation

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Structure of amyloid oligomers and their mechanisms of toxicities: Targeting amyloid oligomers using novel therapeutic approaches

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Physico-Pathologic Mechanisms Involved in Neurodegeneration: Misfolded Protein-Plasma Membrane Interactions

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Why are Functional Amyloids Non-Toxic in Humans?

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The Role of Macrophage Migration Inhibitory Factor in Alzheimer’s Disease

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Dopamine and the Dopamine Oxidation Product 5,6-Dihydroxylindole Promote Distinct On-Pathway and Off-Pathway Aggregation of α-Synuclein in a pH-Dependent Manner

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Spontaneous Fibril Formation by Polyalanines; Discontinuous Molecular Dynamics Simulations

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Interactions between Amyloid-β and Hemoglobin: Implications for Amyloid Plaque Formation in Alzheimer's Disease

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Role of Amyloid- Glycine 33 in Oligomerization, Toxicity, and Neuronal Plasticity

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Replica Exchange Simulations of the Thermodynamics of Aβ Fibril Growth

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Age-related accumulation of Reelin in amyloid-like deposits

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Amyloid‐β impairs the phagocytosis of dystrophic synapses by astrocytes in Alzheimer's disease

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Polymorphism of Alzheimer's Aβ17-42 (p3) Oligomers: The Importance of the Turn Location and Its Conformation

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Anti-oligomeric Aβ Single-chain Variable Domain Antibody Blocks Aβ-induced Toxicity Against Human Neuroblastoma Cells

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Native Chemical Ligation of Thioamide-Containing Peptides: Development and Application to the Synthesis of Labeled α-Synuclein for Misfolding Studies

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Increased BACE1-AS long noncoding RNA and β-amyloid levels in heart failure

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Tau hyperphosphorylation induces oligomeric insulin accumulation and insulin resistance in neurons

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Misfolding of a Human Islet Amyloid Polypeptide at the Lipid Membrane Populates through β-Sheet Conformers without Involving α-Helical Intermediates

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Elevating the Levels of Calcium Ions Exacerbate Alzheimer’s Disease via Inducing the Production and Aggregation of β-Amyloid Protein and Phosphorylated Tau

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