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Role of inhibitory autophosphorylation of calcium/calmodulin-dependent kinase II (αCAMKII) in persistent (>24h) hippocampal LTP and in LTD facilitated by novel object-place learning and recognition in mice

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Overexpression of hippocampal Ca2+/calmodulin‐dependent protein kinase II improves spatial memory

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Evidence of Selective Learning Deficits on Tests of Pavlovian and Instrumental Conditioning in α-CaMKIIT286A Mutant Mice

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Enriching the environment of αCaMKIIT286A mutant mice reveals that LTD occurs in memory processing but must be subsequently reversed by LTPPreprint

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