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Cited by 310

The DOI matches its Crossref record; 130 of the 130 references it cites were found in a registry or an index.

Causes and Mechanisms of Intrauterine Hypoxia and Its Impact on the Fetal Cardiovascular System: A Review

Open access
Hutter, Damian, Kingdom, John, Jaeggi, Edgar·2010·International Journal of Pediatrics

Abstract

Until today the role of oxygen in the development of the fetus remains controversially discussed. It is still believed that lack of oxygen in utero might be responsible for some of the known congenital cardiovascular malformations. Over the last two decades detailed research has given us new insights and a better understanding of embryogenesis and fetal growth. But most importantly it has repeatedly demonstrated that oxygen only plays a minor role in the early intrauterine development. After organogenesis has taken place hypoxia becomes more important during the second and third trimester of pregnancy when fetal growth occurs. This review will briefly adress causes and mechanisms leading to intrauterine hypoxia and their impact on the fetal cardiovascular system.

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Crossref · 10.1155/2010/401323

Cites

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Metadata

Authors
Hutter, D., Kingdom, J., & Jaeggi, E.
Venue
International Journal of Pediatrics
Publisher
Wiley
Published
2010
Volume
2010
Pages
1-9
ISSN
  • 1687-9740 (ISSN-L)
  • 1687-9759
OpenAlex
W2021704352
Cited by
310
References
130
FWCI
1.68
Type
journal article
Language
en
Open access
gold

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CoChat. (2026). Citation check: Causes and Mechanisms of Intrauterine Hypoxia and Its Impact on the Fetal Cardiovascular System: A Review. https://app.cochat.ai/papers/10.1155/2010/401323
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Hutter, D., Kingdom, J., & Jaeggi, E. (2010). Causes and Mechanisms of Intrauterine Hypoxia and Its Impact on the Fetal Cardiovascular System: A Review. International Journal of Pediatrics. https://doi.org/10.1155/2010/401323

Data sources

Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.