The DOI matches its Crossref record; 2 of the 2 references it cites were found in a registry or an index.
Baroreflex sensitivity, clinical correlates, and cardiovascular mortality among patients with a first myocardial infarction. A prospective study.
Open accessAbstract
Experimental studies have shown that among dogs with a healed myocardial infarction, depressed baroreflex sensitivity (BRS) identifies a subgroup at higher risk for sudden death. We have examined the relation among BRS, several clinical cardiovascular variables, and subsequent mortality in 78 patients below the age of 65 years who have had a first myocardial infarction. BRS was assessed by calculating the regression line relating phenylephrine-induced increases in systolic blood pressure to the attendant changes in the RR interval. A reduced BRS primarily reflects an impairment in the vagal efferent component of the baroreceptor reflexes. The BRS of the entire population was 7.8 +/- 4.9 msec/mm Hg. BRS was lower among patients with an inferior myocardial infarction (6.1 +/- 3.3 vs. 8.9 +/- 5.8 msec/mm Hg, p = 0.03), with a three- versus a one-vessel disease (4.8 +/- 2.7 vs. 7.1 +/- 3.1 msec/mm Hg, p = 0.04), and with episodes of ventricular tachycardia (5.1 +/- 3.0 vs. 8.3 +/- 5.1, p = 0.03). There was no correlation between BRS and left ventricular ejection fraction or with mean pulmonary capillary wedge pressure at peak exercise, but a correlation (r = 0.35, p less than 0.001) was present with exercise tolerance. During the 24 months mean follow-up period, there were six cardiovascular deaths (7.6%), and four were sudden.(ABSTRACT TRUNCATED AT 250 WORDS)
Overview
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Integrity signals
Registry
Cites
Statistics
Verification
Claims the paper makes
The claims and statistics of this paper haven’t been checked yet.
Run the check — free, no account needed. It reads the open-access text and checks the sources it cites.
Self-check
Percentages, sums and reported differences re-computed from the paper’s own numbers.
The self-check needs the paper’s full text.
Citations
Cites
Kleiger, R. E., Miller, J. P., Bigger, J. T., & Moss, A. J. (1987) · The American Journal of Cardiology · doi:10.1016/0002-9149(87)90795-8 · Cited by 3,995
Sudden cardiac death: The major challenge confronting contemporary cardiology
Lown, B. (1979) · The American Journal of Cardiology · doi:10.1016/s0002-9149(79)80021-1 · Cited by 451
Cited by
Rovere, M. T. L., Bigger, J. T., Marcus, F. I., Mortara, A., & Schwartz, P. J. (1998) · The Lancet · doi:10.1016/s0140-6736(97)11144-8 · Cited by 3,120
Self-citationThe Relationship of Depression to Cardiovascular Disease: Epidemiology, Biology, and Treatment
Musselman, D. L., Evans, D. L., & Nemeroff, C. B. (1998) · Archives of General Psychiatry · doi:10.1001/archpsyc.55.7.580 · Cited by 1,532
Goldberger, J. J., Cain, M. E., Hohnloser, S. H., Kadish, A. H., Knight, B. P., Lauer, M. S., Maron, B. J., Page, R. L., Passman, R. S., Siscovick, D., et al. (2008) · Circulation · doi:10.1161/circulationaha.107.189375 · Cited by 1,450
Role of the Autonomic Nervous System in Modulating Cardiac Arrhythmias
Shen, M. J., & Zipes, D. P. (2014) · Circulation Research · doi:10.1161/circresaha.113.302549 · Cited by 867
Heart Rate Variability ? A Historical Perspective
Billman, G. E. (2011) · Frontiers in Physiology · doi:10.3389/fphys.2011.00086 · Cited by 786
“(La Rovere et al., 1988, 1998; Malik et al., 1989; Mazzuero et al., 1992; Huikuri et al., 1996; Task Force of the European Society of Cardiology and the North American Society of Pacing and Electrophysiology, 1996; Bigger, 1997; Hohnloser et al., 1997; Lanza et al., 1998).”
BackgroundExercise is the real polypill.
Fiuza-Luces, C., Garatachea, N., Berger, N. A., & Lucia, A. (2013) · Physiology · doi:10.1152/physiol.00019.2013 · Cited by 791
La Rovere, M. T., Pinna, G. D., Hohnloser, S. H., Marcus, F. I., Mortara, A., Nohara, R., Bigger, J. T., Camm, A. J., & Schwartz, P. J. (2001) · Circulation · doi:10.1161/01.cir.103.16.2072 · Cited by 697
Self-citationVanoli, E., De Ferrari, G. M., Stramba-Badiale, M., Hull, S. S., Foreman, R. D., & Schwartz, P. J. (1991) · Circulation Research · doi:10.1161/01.res.68.5.1471 · Cited by 681
Self-citationClinical neurocardiology defining the value of neuroscience-based cardiovascular therapeutics
Shivkumar, K., Ajijola, O. A., Anand, I., Armour, J. A., Chen, P., Esler, M., De Ferrari, G. M., Fishbein, M. C., Goldberger, J. J., Harper, R. M., et al. (2016) · The Journal of Physiology · doi:10.1113/jp271870 · Cited by 308
Self-citationDepressed Baroreflex Sensitivity in Patients With Obstructive Sleep Apnea
Carlson, J. T., Hedner, J. A., Sellgren, J., Elam, M., & Wallin, B. G. (1996) · American journal of respiratory and critical care medicine · doi:10.1164/ajrccm.154.5.8912770 · Cited by 239
CPAP and measures of cardiovascular risk in males with OSAS
Kohler, M., Pepperell, J. C. T., Casadei, B., Craig, S., Crosthwaite, N., Stradling, J. R., & Davies, R. J. O. (2008) · European Respiratory Journal · doi:10.1183/09031936.00026608 · Cited by 197
Aerobic exercise conditioning: a nonpharmacological antiarrhythmic intervention
Billman, G. E. (2002) · Journal of Applied Physiology · doi:10.1152/japplphysiol.00874.2001 · Cited by 185
Schwartz, P. J., & De Ferrari, G. M. (2011) · Heart Failure Reviews · doi:10.1007/s10741-010-9179-1 · Cited by 158
Self-citationMalfatto, G., Facchini, M., Bragato, R., Branzi, G., Sala, L., & Leonetti, G. (1996) · European Heart Journal · doi:10.1093/oxfordjournals.eurheartj.a014905 · Cited by 139
Billman, G. E. (2006) · Pharmacology & Therapeutics · doi:10.1016/j.pharmthera.2006.01.002 · Cited by 131
Radaelli, A., Raco, R., Perfetti, P., Viola, A., Azzellino, A., Signorini, M. G., & Ferrari, A. U. (2004) · Journal of Hypertension · doi:10.1097/01.hjh.0000125446.28861.51 · Cited by 88
Autonomic Control of the Aging Heart
Kaye, D. M., & Esler, M. D. (2008) · NeuroMolecular Medicine · doi:10.1007/s12017-008-8034-1 · Cited by 92
Matsutani, D., Sakamoto, M., Iuchi, H., Minato, S., Suzuki, H., Kayama, Y., Takeda, N., Horiuchi, R., & Utsunomiya, K. (2018) · Cardiovascular Diabetology · doi:10.1186/s12933-018-0683-2 · Cited by 52
The autonomic nervous system and cardiac arrhythmias
Esler, M. (1992) · Clinical Autonomic Research · doi:10.1007/bf01819669 · Cited by 48
Metadata
- Authors
- La Rovere, M. T., Specchia, G., Mortara, A., & Schwartz, P. J.
- Venue
- Circulation
- Published
- 1988-10
- Volume
- 78
- Issue
- 4
- Pages
- 816-824
- ISSN
- 0009-7322 (ISSN-L)
- 1524-4539
- OpenAlex
- W2139470316
- Cited by
- 631
- References
- 2
- FWCI
- 14.16
- Type
- journal article
- Language
- en
- Open access
- bronze
About these checks
Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.
Statistics and arithmetic are recomputed, not judged.
Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.
What counts as a claim ›How we measure confidence ›How we check this ›
Are you an author, or do you see a problem with this page? Request a review
Data sources
Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
