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Cited by 428

The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.

Redox-Dependent Transcriptional Regulation

Open access
Liu, Hongjun, Colavitti, Renata, Rovira, Ilsa I., Finkel, Toren·2005·Circulation Research

Abstract

Reactive oxygen species contribute to the pathogenesis of a number of disparate disorders including tissue inflammation, heart failure, hypertension, and atherosclerosis. In response to oxidative stress, cells activate expression of a number of genes, including those required for the detoxification of reactive molecules as well as for the repair and maintenance of cellular homeostasis. In many cases, these induced genes are regulated by transcription factors whose structure, subcellular localization, or affinity for DNA is directly or indirectly regulated by the level of oxidative stress. This review summarizes the recent progress on how cellular redox status can regulate transcription-factor activity and the implications of this regulation for cardiovascular disease.

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Crossref · 10.1161/01.res.0000188210.72062.10

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Metadata

Authors
Liu, H., Colavitti, R., Rovira, I. I., & Finkel, T.
Venue
Circulation Research
Published
2005-11-11
Volume
97
Issue
10
Pages
967-974
ISSN
  • 0009-7330 (ISSN-L)
  • 1524-4571
OpenAlex
W2097155911
Cited by
428
References
6
FWCI
11.06
Type
journal article
Language
en
Open access
bronze

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CoChat. (2026). Citation check: Redox-Dependent Transcriptional Regulation. https://app.cochat.ai/papers/10.1161/01.res.0000188210.72062.10
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Liu, H., Colavitti, R., Rovira, I. I., & Finkel, T. (2005). Redox-Dependent Transcriptional Regulation. Circulation Research. https://doi.org/10.1161/01.res.0000188210.72062.10

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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.

Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.