The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.
Death by Design: Programmed Cell Death in Cardiovascular Biology and Disease
Abstract
Abstract Programmed cell death (apoptosis) is recognized, increasingly, as a contributing cause of cardiac myocyte loss with ischemia/reperfusion injury, myocardial infarction, and long-standing heart failure. Although the exact mechanisms initiating apoptosis in these in vivo settings remain unproven, insights into the molecular circuitry controlling apoptosis more widely suggest the potential to protect mammalian ventricular muscle from apoptosis through one or more of these pathways, by pharmacological means or, conceivably, gene transfer.
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Citations
Cites
Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours
Donehower, L. A., Harvey, M., Slagle, B. L., McArthur, M. J., Montgomery, C. A., Butel, J. S., & Bradley, A. (1992) · Nature · doi:10.1038/356215a0 · Cited by 4,649
Tumor suppressor p53 is a direct transcriptional activator of the human bax gene
Toshiyuki, M., & Reed, J. C. (1995) · Cell · doi:10.1016/0092-8674(95)90412-3 · Cited by 4,063
Elevated Circulating Levels of Tumor Necrosis Factor in Severe Chronic Heart Failure
Levine, B., Kalman, J., Mayer, L., Fillit, H. M., & Packer, M. (1990) · New England Journal of Medicine · doi:10.1056/nejm199007263230405 · Cited by 2,582
Liu, Z.-G., Hsu, H., Goeddel, D. V., & Karin, M. (1996) · Cell · doi:10.1016/s0092-8674(00)81375-6 · Cited by 1,937
Sandstrom, P. A., & Buttke, T. M. (1993) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.90.10.4708 · Cited by 186
Cited by
Myocyte proliferation in end-stage cardiac failure in humans
Kajstura, J., Leri, A., Finato, N., Di Loreto, C., Beltrami, C. A., & Anversa, P. (1998) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.95.15.8801 · Cited by 567
Catecholamines, Cardiac β-Adrenergic Receptors, and Heart Failure
Lefkowitz, R. J., Rockman, H. A., & Koch, W. J. (2000) · Circulation · doi:10.1161/01.cir.101.14.1634 · Cited by 291
Shang, L., Ananthakrishnan, R., Li, Q., Quadri, N., Abdillahi, M., Zhu, Z., Qu, W., Rosario, R., Touré, F., Yan, S. F., et al. (2010) · PLoS ONE · doi:10.1371/journal.pone.0010092 · Cited by 93
Li, J., Li, C., Zhang, D., Shi, D., Qi, M., Feng, J., Yuan, T., Xu, X., Liang, D., Xu, L., et al. (2014) · Nature Communications · doi:10.1038/ncomms6177 · Cited by 46
Metadata
- Authors
- MacLellan, W. R., & Schneider, M. D.
- Venue
- Circulation Research
- Published
- 1997-08
- Volume
- 81
- Issue
- 2
- Pages
- 137-144
- ISSN
- 0009-7330 (ISSN-L)
- 1524-4571
- OpenAlex
- W2157201513
- Cited by
- 358
- References
- 5
- FWCI
- 11.59
- Type
- journal article
- Language
- en
- Open access
- closed
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- MacLellan, W. R., & Schneider, M. D. (1997). Death by Design: Programmed Cell Death in Cardiovascular Biology and Disease. Circulation Research. https://doi.org/10.1161/01.res.81.2.137
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
