The DOI matches its Crossref record; 20 of the 20 references it cites were found in a registry or an index.
Monocytes in Myocardial Infarction
Open accessAbstract
Myocardial infarction (MI) is the leading cause of death in developed countries. Though timely revascularization of the ischemic myocardium and current standard therapy reduce acute mortality after MI, long-term morbidity and mortality remain high. During the first 1 to 2 weeks after MI, tissues in the infarcted myocardium undergo rapid turnover, including digestion of extracellular matrix and fibrosis. Post-MI repair is crucial to survival. Monocytes recruited to the infarcted myocardium remove debris and facilitate the repair process. However, exaggerated inflammation may also impede healing, as demonstrated by the association between elevated white blood cell count and in-hospital mortality after MI. Monocytes produced in the bone marrow and spleen enter the blood after MI and are recruited to the injured myocardium in 2 phases. The first phase is dominated by Ly-6c high monocytes and the second phase by Ly-6c low monocytes. Yet the number of Ly6C low monocytes recruited to the infarct is much lower, and Ly6C high monocytes can differentiate to Ly6C low macrophages in later healing stages. Understanding the signals regulating monocytosis after MI will help design new therapies to facilitate cardiac healing and limit heart failure.
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Cites
Fate Mapping Reveals Origins and Dynamics of Monocytes and Tissue Macrophages under Homeostasis
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Self-citationIdentification of splenic reservoir monocytes and their deployment to inflammatory sites
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Cited by
Kalogeris, T., Baines, C. P., Krenz, M., & Korthuis, R. J. (2016) · Comprehensive Physiology · doi:10.1002/cphy.c160006 · Cited by 718
Ma, Y., Mouton, A. J., & Lindsey, M. L. (2018) · Translational Research · doi:10.1016/j.trsl.2017.10.001 · Cited by 405
Inflammation and fibrosis in murine models of heart failure
Bacmeister, L., Schwarzl, M., Warnke, S., Stoffers, B., Blankenberg, S., Westermann, D., & Lindner, D. (2019) · Basic Research in Cardiology · doi:10.1007/s00395-019-0722-5 · Cited by 382
Tumor-associated macrophages: potential therapeutic strategies and future prospects in cancer
Li, C., Xu, X., Wei, S., Jiang, P., Xue, L., & Wang, J. (2021) · Journal for ImmunoTherapy of Cancer · doi:10.1136/jitc-2020-001341 · Cited by 360
Kologrivova, I., Shtatolkina, M., Suslova, T., & Ryabov, V. (2021) · Frontiers in Immunology · doi:10.3389/fimmu.2021.664457 · Cited by 317
Metadata
- Authors
- Dutta, P., & Nahrendorf, M.
- Venue
- Arteriosclerosis, Thrombosis, and Vascular Biology
- Published
- 2015-05
- Volume
- 35
- Issue
- 5
- Pages
- 1066-1070
- ISSN
- 1079-5642 (ISSN-L)
- 1524-4636
- OpenAlex
- W2081004896
- Cited by
- 208
- References
- 20
- FWCI
- 14.95
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Dutta, P., & Nahrendorf, M. (2015). Monocytes in Myocardial Infarction. Arteriosclerosis, Thrombosis, and Vascular Biology. https://doi.org/10.1161/atvbaha.114.304652
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
