The DOI matches its Crossref record; 64 of the 64 references it cites were found in a registry or an index.
Accelerated Atherosclerosis in Autoimmune Rheumatic Diseases
Open accessAbstract
Enhanced and premature atherosclerosis is a feature of some AIRDs and a possible feature of others because of inflammation and more specific immune mechanisms (Table 3). RA, SLE, and APS carry an increased risk for CVD. RA and SLE are characterized by an increased risk of coronary artery disease and prevalence of typical risk factors for these diseases and an increased extent of subclinical atherosclerosis. However, other factors attributed to disease activity, inflammation, and therapeutic interventions are also implicated in the higher prevalence of atherosclerosis and its complications in both diseases. APS is a prothrombotic statecharacterized by thrombosis of any vessel; however, the association of aPL with CVDs in the general population, the findings of enhanced subclinical atherosclerosis in APS patients, and the proatherogenic effect of aPL in animal models support a possible proatherogenic (in addition to prothrombotic) role of these autoantibodies. In these 3 conditions, premature atherosclerosis can be detected in some patients in its preclinical stage. Physicians thus should attempt to minimize the presence of conventional CVD risk factors in their patients and treat their patients as belonging to a group having a high risk for CVD. In SSc and PSV, although there is a high prevalence of macrovascular disease, there are few data supporting enhanced atherosclerosis in these conditions. In addition, no data yet exist to support enhanced atherosclerosis in SS. Future research is needed to determine whether these AIRDs are also associated with accelerated atherosclerosis and its manifestations.
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Cites
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Cited by
Tyndall, A. J., Bannert, B., Vonk, M., Airò, P., Cozzi, F., Carreira, P. E., Bancel, D. F., Allanore, Y., Müller-Ladner, U., Distler, O., et al. (2010) · Annals of the Rheumatic Diseases · doi:10.1136/ard.2009.114264 · Cited by 1,443
Pablos, J. L., Galindo, M., Carmona, L., Lledó, A., Retuerto, M., Blanco, R., Gonzalez-Gay, M. A., Martinez-Lopez, D., Castrejón, I., Alvaro-Gracia, J. M., et al. (2020) · Annals of the Rheumatic Diseases · doi:10.1136/annrheumdis-2020-218296 · Cited by 238
Chronic Inflammatory Diseases and Endothelial Dysfunction
Castellon, X., & Bogdanova, V. (2016) · Aging and disease · doi:10.14336/ad.2015.0803 · Cited by 191
Nazir, S., Jankowski, V., Bender, G., Zewinger, S., Rye, K.-A., & van der Vorst, E. P. C. (2020) · Advanced Drug Delivery Reviews · doi:10.1016/j.addr.2020.10.006 · Cited by 140
Chen, P.-C., Tseng, T.-C., Hsieh, J.-Y., & Lin, H.-W. (2011) · Stroke · doi:10.1161/strokeaha.110.612655 · Cited by 23
Metadata
- Authors
- Shoenfeld, Y., Gerli, R., Doria, A., Matsuura, E., Cerinic, M. M., Ronda, N., Jara, L. J., Abu-Shakra, M., Meroni, P. L., & Sherer, Y.
- Venue
- Circulation
- Published
- 2005-11-22
- Volume
- 112
- Issue
- 21
- Pages
- 3337-3347
- ISSN
- 0009-7322 (ISSN-L)
- 1524-4539
- OpenAlex
- W2039093757
- Cited by
- 555
- References
- 64
- FWCI
- 17.28
- Type
- journal article
- Language
- en
- Open access
- bronze
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- Shoenfeld, Y., Gerli, R., Doria, A., Matsuura, E., Cerinic, M. M., Ronda, N., Jara, L. J., Abu-Shakra, M., Meroni, P. L., & Sherer, Y. (2005). Accelerated Atherosclerosis in Autoimmune Rheumatic Diseases. Circulation. https://doi.org/10.1161/circulationaha.104.507996
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
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