Cited by 1,080

The DOI matches its Crossref record; 3 of the 3 references it cites were found in a registry or an index.

Central Pressure More Strongly Relates to Vascular Disease and Outcome Than Does Brachial Pressure: The Strong Heart Study

Roman, Mary J., Devereux, Richard B., Kizer, Jorge R., Lee, Elisa T., Galloway, James M., Ali, Tauqeer, Umans, Jason G., Howard, Barbara V.·2007·Hypertension

Abstract

Brachial blood pressure is predictive of cardiovascular outcome; however central pressure may better represent the load imposed on the coronary and cerebral arteries and thereby bear a stronger relationship to vascular damage and prognosis. Relations of brachial and central pressures to carotid artery hypertrophy (intimal-medial thickness and vascular mass), extent of atherosclerosis (plaque score), and incident cardiovascular events were examined in the Strong Heart Study. Central pressures were calculated using radial applanation tonometry. Among 3520 participants, central and brachial pulse pressures were more strongly related to vascular hypertrophy and extent of atherosclerosis than were systolic pressures. Central pulse pressure was more strongly related to all 3 arterial measures than was brachial pulse pressure ( r =0.364 versus 0.309 for plaque score; P <0.001 for comparison of Spearman correlation coefficient; r =0.293 versus 0.249 for intimal-medial thickness; P <0.002; r =0.320 versus 0.289 for vascular mass; P <0.05). Among the 2403 participants free of clinical cardiovascular disease at baseline, 319 suffered fatal or nonfatal cardiovascular events during mean follow-up of 4.8±1.3 years. After adjustment for age, gender, current smoking, body mass index, cholesterol:HDL ratio, creatinine, fibrinogen, diabetes, and heart rate, central pulse pressure predicted cardiovascular events more strongly than brachial pulse pressure (hazards ratio=1.15 per 10 mm Hg, χ 2 =13.4, P <0.001 versus hazards ratio=1.10, χ 2 =6.9, P =0.008). In conclusion, noninvasively-determined central pulse pressure is more strongly related to vascular hypertrophy, extent of atherosclerosis, and cardiovascular events than is brachial blood pressure. These findings support prospective examination of use of central blood pressure as a treatment target in future trials.

Overview

Integrity signals

Registry

Crossref · 10.1161/hypertensionaha.107.089078

Cites

3 of 3 references verified0 citation errors · 0 not checkable

Statistics

Recomputing p-values needs the paper’s full text.

Verification

Claims the paper makes

This paper’s full text isn’t openly available, so its claims and self-check weren’t run. The registry and citation checks still apply.

Claims other papers make about this one

Each is checked against this paper’s own text: does it say what the citing paper says it does?

Citations

Cites

Flagged first, then by citations

Research works printed but not linked: 45

  1. 10.1016/S0140-6736(03)14739-3 · DOI · Paper page
  2. 10.1161/01.hyp.0000165020.14745.79 · DOI
  3. 10.7326/0003-4819-138-1-200301070-00006 · DOI
  4. 10.1161/01.hyp.23.3.395 · DOI
  5. Benetos A, Safar M, Rudnichi A, Smulyan H, Richard J-L, Ducimetière P, Guize L. Pulse pressure: a predictor of long-term cardiovascular mortality in a French male population. Hypertension. 1999; 30: 1410–1415.
  6. 10.1161/circ.100.4.354 · DOI
  7. 10.1001/archinte.160.18.2765 · DOI
  8. 10.1016/S0735-1097(00)00687-2 · DOI
  9. 10.1161/hyp.38.4.793 · DOI
  10. Palmieri V, Devereux RB, Holloywood J, Bella JN, Liu JE, Lee ET, Best LG, Howard BV, Roman MJ. Association of pulse pressure with cardiovascular outcome is independent of left ventricular hypertrophy and systolic dysfunction: The Strong Heart Study. J Hypertens. 2006; 19: 601–607.
  11. 10.1161/hyp.32.3.560 · DOI
  12. 10.1161/hyp.36.5.801 · DOI
  13. 10.7326/0003-4819-139-9-200311040-00007 · DOI
  14. 10.1161/circ.103.9.1245 · DOI
  15. 10.1161/hyp.38.4.953 · DOI
  16. 10.1097/00004872-199917080-00004 · DOI
  17. 10.1161/hyp.37.5.1236 · DOI
  18. 10.1161/hyp.39.1.10 · DOI
  19. 10.1161/atvb.21.12.2046 · DOI
  20. 10.1161/circulationaha.104.483628 · DOI
  21. 10.1161/circ.99.18.2434 · DOI
  22. 10.1161/01.hyp.0000159190.71253.c3 · DOI
  23. 10.1161/circulationaha.105.579342 · DOI · Paper page
  24. 10.1161/hyp.38.4.932 · DOI
  25. 10.1161/circulationaha.105.595496 · DOI · Paper page
  26. 10.1152/ajplegacy.1938.125.1.48 · DOI
  27. 10.1161/circ.5.2.257 · DOI
  28. Nichols WW O’Rourke MF eds. McDonald’s Blood Flow in Arteries: Theoretical Experimental and Clinical Principles. Fifth Edition. Oxford: Hodder Arnold; 2005: 193–213 339–386.
  29. 10.1093/oxfordjournals.aje.a115757 · DOI
  30. North KE, Howard BV, Welty TK, Best LG, Lee ET, Yeh JL, Fabsitz RR, Roman MJ, MacCluer JW. Genetic and environmental contributions to cardiovascular disease risk in American Indians: The Strong Heart Family Study. Am J Epidemiol. 2002; 157: 303–314.
  31. 10.2337/diacare.26.11.3160 · DOI · Paper page
  32. 10.1161/circ.99.18.2389 · DOI
  33. 10.1093/oxfordjournals.aje.a009406 · DOI
  34. 10.1016/j.echo.2006.04.020 · DOI
  35. 10.1016/0735-1097(96)00225-2 · DOI
  36. Kelly R, Hayward C, Ganis J, Daley J, Avolio A, O’Rourke M. Noninvasive registration of the arterial pressure waveform using high-fidelity applanation tonometry. J Vasc Med Biol. 1989; 1: 142–149.
  37. 10.1161/01.HYP.27.2.168 · DOI
  38. 10.1016/0021-9290(80)90191-8 · DOI
  39. 10.1161/circ.80.1.2610739 · DOI
  40. 10.1161/circ.100.13.1387 · DOI
  41. 10.1161/hyp.38.4.927 · DOI
  42. 10.1097/01.hjh.0000125448.28861.fc · DOI
  43. 10.1161/hyp.39.3.735 · DOI
  44. 10.1161/01.hyp.0000209340.33592.50 · DOI
  45. 10.1093/eurheartj/ehl254 · DOI · Paper page

Cited by

Sorted by citations

Metadata

Authors
Roman, M. J., Devereux, R. B., Kizer, J. R., Lee, E. T., Galloway, J. M., Ali, T., Umans, J. G., & Howard, B. V.
Affiliations
From the Division of Cardiology (M.J.R., R.B.D., J.R.K.), Weill Medical College of Cornell University, New York; the Center for American Indian Health Research (E.T.L., T.A.), University of Oklahoma Health Sciences Center, Oklahoma City; the University of Arizona (J.M.G.), Tucson; and Medstar Research Institute (J.G.U., B.V.H.), Washington, DC.
Venue
Hypertension
Published
2007-07
Volume
50
Issue
1
Pages
197-203
ISSN
  • 0194-911X
  • 1524-4563
Cited by
1,080
References
3
Language
en

About these checks

Registry, retraction, Cites and statistics results are computed, not judged, with one exception: a printed reference that no index lists is read once by a model before it counts as a citation error. Claims are extracted by a model and kept only when their quote appears verbatim in the text; each cited source is then checked, with a model’s help, for whether it says what the claim relies on. Those verdicts can be wrong — read the claim’s quote and the cited source before relying on one.

Statistics and arithmetic are recomputed, not judged.

Confidence bands are shown only for fields calibrated on our annotated test set. None is calibrated yet, so no band is shown.

What counts as a claim ›How we measure confidence ›How we check this ›

Are you an author, or do you see a problem with this page? Request a review