The DOI matches its Crossref record; 92 of the 92 references it cites were found in a registry or an index.
Cellular and molecular mechanisms in kidney fibrosis
Open accessAbstract
Fibrosis is a characteristic feature of all forms of chronic kidney disease. Deposition of pathological matrix in the interstitial space and within the walls of glomerular capillaries as well as the cellular processes resulting in this deposition are increasingly recognized as important factors amplifying kidney injury and accelerating nephron demise. Recent insights into the cellular and molecular mechanisms of fibrogenesis herald the promise of new therapies to slow kidney disease progression. This review focuses on new findings that enhance understanding of cellular and molecular mechanisms of fibrosis, the characteristics of myofibroblasts, their progenitors, and molecular pathways regulating both fibrogenesis and its resolution.
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Self-citationFate Tracing Reveals the Pericyte and Not Epithelial Origin of Myofibroblasts in Kidney Fibrosis
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Cited by
Cardiac Fibrosis: The Fibroblast Awakens
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“While the reasons for these discrepant results are unclear, mosaic transgene expression or expression patterns unfaithful to the endogenous allele, both known properties of random integration transgenics, is one possible explanation (Duffield, 2014; Le et al., 2006).”
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Decoding myofibroblast origins in human kidney fibrosis
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Failed Tubule Recovery, AKI-CKD Transition, and Kidney Disease Progression
Venkatachalam, M. A., Weinberg, J. M., Kriz, W., & Bidani, A. K. (2015) · Journal of the American Society of Nephrology · doi:10.1681/asn.2015010006 · Cited by 744
Metadata
- Authors
- Duffield, J. S.
- Venue
- Journal of Clinical Investigation
- Published
- 2014-06-02
- Volume
- 124
- Issue
- 6
- Pages
- 2299-2306
- ISSN
- 0021-9738 (ISSN-L)
- OpenAlex
- W2044982400
- Cited by
- 629
- References
- 92
- FWCI
- 39.52
- Type
- journal article
- Language
- en
- Open access
- bronze
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
