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Dissecting the role of non-coding RNAs in the accumulation of amyloid and tau neuropathologies in Alzheimer’s disease

Patrick, Ellis, Rajagopal, Sathyapriya, Wong, Hon-Kit Andus, McCabe, Cristin, Xu, Jishu, Tang, Anna, Imboywa, Selina H., Schneider, Julie A. +5 more·2017·Molecular Neurodegeneration
DOI 10.1186/s13024-017-0191-yPMC PMC5494142

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  • Other previous studies have shown no differential expression of miR-155 in AD brains.

    “Other previous studies have shown no differential expression of miR-155 in AD brains [48][49][50] .”

    From Identification of a protective microglial state mediated by miR-155 and interferon-γ signaling in a mouse model of Alzheimer’s disease (2023) · cites this paper as [49]

    The Results section on miRNAs associated with pathologic AD identifies two significant miRNAs, but does not report a result for miR-155. Searches for miR-155 and variant wording found no passage establishing whether it was tested or differentially expressed in AD brains. (LLM, conf=0.76)

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Research works printed but not linked: 35

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Authors
Patrick, E., Rajagopal, S., Wong, H.-K. A., McCabe, C., Xu, J., Tang, A., Imboywa, S. H., Schneider, J. A., Pochet, N., Krichevsky, A. M., Chibnik, L. B., Bennett, D. A., & De Jager, P. L.
Venue
Molecular Neurodegeneration
Publisher
Springer Science and Business Media LLC
Published
2017-07-01
Volume
12
Issue
1
ISSN
1750-1326
DOI
10.1186/s13024-017-0191-y
PMC
PMC5494142
Cited by
77
References
6
Language
en
Licence
Funders
National Institute on Aging (R01 AG036042, R01 AG036836, R01 AG17917, RF1 AG15819, R01 AG032990, R01 AG18023, RC2 AG036547, P30 AG10161, U01 AG46152, P50 AG016574, U01 ES017155, K25 AG041906-01)

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