The DOI matches its Crossref record; 94 of the 94 references it cites were found in a registry or an index.
Autophagy and inflammation
Open accessAbstract
Autophagy is a homeostatic mechanism involved in the disposal of damaged organelles, denatured proteins as well as invaded pathogens through a lysosomal degradation pathway. Recently, increasing evidences have demonstrated its role in both innate and adaptive immunity, and thereby influence the pathogenesis of inflammatory diseases. The detection of autophagy machinery facilitated the measurement of autophagy during physiological and pathophysiological processes. Autophagy plays critical roles in inflammation through influencing the development, homeostasis and survival of inflammatory cells, including macrophages, neutrophils and lymphocytes; effecting the transcription, processing and secretion of a number of cytokines, as well as being regulated by cytokines. Recently, autophagy‐dependent mechanisms have been studied in the pathogenesis of several inflammatory diseases, including infectious diseases, Crohn's disease, cystic fibrosis, pulmonary hypertension, chronic obstructive pulmonary diseases and so on. These studies suggested that modulation of autophagy might lead to therapeutic interventions for diseases associated with inflammation. Here we highlight recent advances in investigating the roles of autophagy in inflammation as well as inflammatory diseases.
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Cites
Repertoires of Autophagy in the Pathogenesis of Ocular Diseases
Li, Y.-J., Jiang, Q., Cao, G.-F., Yao, J., & Yan, B. (2015) · Cellular Physiology and Biochemistry · doi:10.1159/000373980 · Cited by 16,304
Autophagosome formation: core machinery and adaptations
Xie, Z., & Klionsky, D. J. (2007) · Nature Cell Biology · doi:10.1038/ncb1007-1102 · Cited by 2,164
Isolation and characterization of autophagy‐defective mutants of Saccharomyces cerevisiae
Tsukada, M., & Ohsumi, Y. (1993) · FEBS Letters · doi:10.1016/0014-5793(93)80398-e · Cited by 1,772
Autophagy-Dependent Viral Recognition by Plasmacytoid Dendritic Cells
Lee, H. K., Lund, J. M., Ramanathan, B., Mizushima, N., & Iwasaki, A. (2007) · Science · doi:10.1126/science.1136880 · Cited by 864
Autophagy Controls IL-1β Secretion by Targeting Pro-IL-1β for Degradation
Harris, J., Hartman, M., Roche, C., Zeng, S. G., O'Shea, A., Sharp, F. A., Lambe, E. M., Creagh, E. M., Golenbock, D. T., Tschopp, J., et al. (2011) · Journal of Biological Chemistry · doi:10.1074/jbc.m110.202911 · Cited by 794
Autophagy, Inflammation, and Immunity: A Troika Governing Cancer and Its Treatment
Zhong, Z., Sanchez-Lopez, E., & Karin, M. (2016) · Cell · doi:10.1016/j.cell.2016.05.051 · Cited by 576
The Atg5–Atg12 conjugate associates with innate antiviral immune responses
Jounai, N., Takeshita, F., Kobiyama, K., Sawano, A., Miyawaki, A., Xin, K.-Q., Ishii, K. J., Kawai, T., Akira, S., Suzuki, K., et al. (2007) · Proceedings of the National Academy of Sciences · doi:10.1073/pnas.0704014104 · Cited by 565
Isolation of autophagocytosis mutants of Saccharomyces cerevisiae
Thumm, M., Egner, R., Koch, B., Schlumpberger, M., Straub, M., Veenhuis, M., & Wolf, D. H. (1994) · FEBS Letters · doi:10.1016/0014-5793(94)00672-5 · Cited by 560
Luciani, A., Villella, V. R., Esposito, S., Brunetti-Pierri, N., Medina, D., Settembre, C., Gavina, M., Pulze, L., Giardino, I., Pettoello-Mantovani, M., et al. (2010) · Nature Cell Biology · doi:10.1038/ncb2090 · Cited by 467
Zhao, Z., Fux, B., Goodwin, M., Dunay, I. R., Strong, D., Miller, B. C., Cadwell, K., Delgado, M. A., Ponpuak, M., Green, K. G., et al. (2008) · Cell Host & Microbe · doi:10.1016/j.chom.2008.10.003 · Cited by 419
Cited by
New insights into the interplay between autophagy, gut microbiota and inflammatory responses in IBD
Larabi, A., Barnich, N., & Nguyen, H. T. T. (2020) · Autophagy · doi:10.1080/15548627.2019.1635384 · Cited by 835
Role of inflammatory cells in airway remodeling in COPD
Wang, Y., Xu, J., Meng, Y., Adcock, I. M., & Yao, X. (2018) · International Journal of Chronic Obstructive Pulmonary Disease · doi:10.2147/copd.s176122 · Cited by 373
Autophagy and inflammation in ischemic stroke
Liu, K.-Y., Mo, Y., & Sun, Y.-Y. (2020) · Neural Regeneration Research · doi:10.4103/1673-5374.274331 · Cited by 322
Gornowicz, A., Szymanowska, A., Mojzych, M., Bielawski, K., & Bielawska, A. (2020) · International Journal of Molecular Sciences · doi:10.3390/ijms21155221 · Cited by 298
Therapeutic Potential of Volatile Terpenes and Terpenoids from Forests for Inflammatory Diseases
Kim, T., Song, B., Cho, K. S., & Lee, I.-S. (2020) · International Journal of Molecular Sciences · doi:10.3390/ijms21062187 · Cited by 281
Metadata
- Authors
- Qian, M., Fang, X., & Wang, X.
- Venue
- Clinical and Translational Medicine
- Publisher
- Wiley
- Published
- 2017-07-26
- Volume
- 6
- Issue
- 1
- Pages
- 24-24
- ISSN
- 2001-1326 (ISSN-L)
- OpenAlex
- W2947088163
- Cited by
- 316
- References
- 94
- FWCI
- 18.92
- Type
- journal article
- Language
- en
- Open access
- gold
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Bibliographic data: Semantic Scholar, OpenAlex, Crossref.
Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
