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Cited by 172

The DOI matches its Crossref record; 5 of the 5 references it cites were found in a registry or an index.

Regulation of superoxide production in neutrophils: role of calcium influx

Open access
Bréchard, Sabrina, Tschirhart, Eric J·2008·Journal of Leukocyte Biology
DOI 10.1189/jlb.0807553
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Abstract

Upon stimulation, activation of NADPH oxidase complexes in neutrophils produces a burst of superoxide anions contributing to oxidative stress and the development of inflammatory process. Store-operated calcium entry (SOCE), whereby the depletion of intracellular stores induces extracellular calcium influx, is known to be a crucial element of NADPH oxidase regulation. However, the mechanistic basis mediating SOCE is still only partially understood, as is the signal-coupling pathway leading to modulation of store-operated channels. This review emphasizes the role of calcium influx in the control of the NADPH oxidase and summarizes the current knowledge of pathways mediating this extracellular calcium entry in neutrophils. Such investigations into the cross-talk between NADPH oxidase and calcium might allow the identification of novel pharmacological targets with clinical use, particularly in inflammatory diseases.

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Metadata

Authors
Bréchard, S., & Tschirhart, E. J.
Venue
Journal of Leukocyte Biology
Publisher
Oxford University Press (OUP)
Published
2008-06-02
Volume
84
Issue
5
Pages
1223-1237
ISSN
  • 0741-5400 (ISSN-L)
  • 1938-3673
DOI
10.1189/jlb.0807553
OpenAlex
W2169768846
Cited by
172
References
5
FWCI
5.09
Type
journal article
Language
en
Open access
green

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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).

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