Nelson, Peter T.

Alzheimer's-type neuropathology in the precuneus is not increased relative to other areas of neocortex across a range of cognitive impairment

Nelson, P. T., Abner, E. L., Scheff, S. W., Schmitt, F. A., Kryscio, R. J., Jicha, G. A., Smith, C. D., Patel, E., & Markesbery, W. R. (2009) · Neuroscience Letters

Alzheimer's disease research and treatmentsDementia and Cognitive Impairment Research

Cited by 70

Expression of miR-15/107 Family MicroRNAs in Human Tissues and Cultured Rat Brain Cells

Wang, W.-X., Danaher, R. J., Miller, C. S., Berger, J. R., Nubia, V. G., Wilfred, B. S., Neltner, J. H., Norris, C. M., & Nelson, P. T. (2014) · Genomics, Proteomics & Bioinformatics

MicroRNA in disease regulationPlant Molecular Biology Research

Cited by 67

Individual microRNAs (miRNAs) display distinct mRNA targeting “rules”

Wang, W.-X., Wilfred, B. R., Xie, K., Jennings, M. H., Hu, Y., Stromberg, A. J., & Nelson, P. (2010) · RNA Biology

MicroRNA in disease regulationRNA Research and Splicing

Cited by 61

Specific sequence determinants of miR-15/107 microRNA gene group targets

Nelson, P. T., Wang, W.-X., Mao, G., Wilfred, B. R., Xie, K., Jennings, M. H., Gao, Z., & Wang, X. (2011) · Nucleic Acids Research

MicroRNA in disease regulationRNA Research and Splicing

Cited by 53

The Mitochondria-Associated ER Membranes Are Novel Subcellular Locations Enriched for Inflammatory-Responsive MicroRNAs

Wang, W.-X., Prajapati, P., Nelson, P. T., & Springer, J. E. (2020) · Molecular Neurobiology

MicroRNA in disease regulationMitochondrial Function and Pathology

Cited by 49

“End-Stage” Neurofibrillary Tangle Pathology in Preclinical Alzheimer's Disease: Fact or Fiction?

Abner, E. L., Kryscio, R. J., Schmitt, F. A., SantaCruz, K. S., Jicha, G. A., Lin, Y., Neltner, J. M., Smith, C. D., Van Eldik, L. J., & Nelson, P. T. (2011) · Journal of Alzheimer s Disease

Dementia and Cognitive Impairment ResearchAlzheimer's disease research and treatments

Cited by 49

Dysregulation of the mitogen granulin in human cancer through the miR-15/107 microRNA gene group

Wang, W.-X., Kyprianou, N., Wang, X., & Nelson, P. T. (2010) · Cancer Research

MicroRNA in disease regulationCancer-related molecular mechanisms research

Cited by 48

Thinking Outside the Box: Alzheimer-Type Neuropathology That Does Not Map Directly Onto Current Consensus Recommendations

Nelson, P. T., Kukull, W. A., & Frosch, M. P. (2010) · Journal of Neuropathology & Experimental Neurology

Dementia and Cognitive Impairment ResearchAlzheimer's disease research and treatments

Cited by 37

Hippocampal Sclerosis in Older Patients: Practical Examples and Guidance With a Focus on Cerebral Age-Related TDP-43 With Sclerosis

Cykowski, M. D., Powell, S. Z., Schulz, P. E., Takei, H., Rivera, A. L., Jackson, R. E., Roman, G., Jicha, G. A., & Nelson, P. T. (2017) · Archives of Pathology & Laboratory Medicine

Dementia and Cognitive Impairment ResearchAmyotrophic Lateral Sclerosis Research

Cited by 33

Translating Alzheimer’s disease-associated polymorphisms into functional candidates: a survey of IGAP genes and SNPs

Katsumata, Y., Nelson, P. T., Estus, S., & Fardo, D. W. (2019) · Neurobiology of Aging

Alzheimer's disease research and treatmentsGenetic Associations and Epidemiology

Cited by 30

Amyloid-β predominant Alzheimer’s disease neuropathologic change

Kovacs, G. G., Katsumata, Y., Wu, X., Aung, K. Z., Fardo, D. W., Forrest, S. L., Alzheimer's Disease Genetics Consortium, Bowen, J. D., Crane, P. K., Jarvik, G. P., et al. (2025) · Brain

Alzheimer's disease research and treatmentsDementia and Cognitive Impairment Research

Cited by 19

Apolipoprotein E Proteinopathy Is a Major Dementia-Associated Pathologic Biomarker in Individuals with or without the APOE Epsilon 4 Allele

Gal, J., Katsumata, Y., Zhu, H., Srinivasan, S., Chen, J., Johnson, L. A., Wang, W.-X., Golden, L. R., Wilcock, D. M., Jicha, G. A., et al. (2022) · The American Journal of Pathology

Alzheimer's disease research and treatmentsDementia and Cognitive Impairment Research

Cited by 17

Age-associated B cells (ABCs) develop from a CNS-localized progenitor pool into a pro-inflammatory phenotype after stroke

McAtee, A. M., Kenwood, M., Ujas, T., Colson, M. K., Watkins, J., Winford, E. D., Cotter, K. M., Britsch, D., Betz, D., Turchan-Cholewo, J., et al. (2026) · Journal of Neuroinflammation

Neuroinflammation and Neurodegeneration MechanismsT-cell and B-cell Immunology

Cited by 1