The DOI matches its Crossref record; 6 of the 6 references it cites were found in a registry or an index.
Helicobacter infection and gastric neoplasia
Open accessAbstract
Chronic gastritis induced by Helicobacter pylori is the strongest known risk factor for adenocarcinoma of the distal stomach, yet only a minority of people who harbour this organism ever develop cancer. H. pylori isolates possess substantial genotypic diversity, which engenders differential host inflammatory responses that influence clinical outcome. H. pylori strains that possess the cag pathogenicity island and secrete a functional cytotoxin induce more severe gastric injury and further augment the risk for developing distal gastric cancer. However, carcinogenesis is also influenced by host genetic diversity, particularly involving immune response genes such as IL‐1ß and TNF‐ α. It is important to gain insight into the pathogenesis of H. pylori ‐induced gastritis and adenocarcinoma, not only to develop more effective treatments for gastric cancer, but also because it might serve as a paradigm for the role of chronic inflammation in the genesis of other malignancies that arise within the gastrointestinal tract. Copyright © 2006 Pathological Society of Great Britain and Ireland. Published by John Wiley & Sons, Ltd.
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Self-citationThe biological functions of IL-17 in different clinical expressions of Helicobacter pylori-infection
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Metadata
- Authors
- Peek, R. M., & Crabtree, J. E.
- Venue
- The Journal of Pathology
- Publisher
- Wiley
- Published
- 2005-12-17
- Volume
- 208
- Issue
- 2
- Pages
- 233-248
- ISSN
- 0022-3417 (ISSN-L)
- 1096-9896
- OpenAlex
- W1999587546
- Cited by
- 550
- References
- 6
- FWCI
- 19.12
- Type
- journal article
- Language
- en
- Open access
- bronze
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Contains information from the Semantic Scholar Academic Graph Datasets, made available under the ODC Attribution License (ODC-BY 1.0).
Semantic Scholar’s data is described in: Kinney, R., et al. (2023). The Semantic Scholar Open Data Platform. arXiv:2301.10140.
